Camila Silva
Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.
Research Biologist
Also affiliated: Rutgers, The State University of New Jersey (2022); Universidade Estadual de Maringá (2019–2021); United States Food and Drug Administration (2015–2026); Universidade de Ribeirão Preto (2012–2013); Universidade Federal Rural do Rio de Janeiro (2013); Pontificia Universidad Católica de Chile (2015–2019); Universidade de São Paulo (2007–2013); Portuguese Army (2006–2008); Universidade Federal do Rio Grande do Norte (2015–2016); Spanish National Cancer Research Centre (2024); Instituto Chico Mendes de Conservação da Biodiversidade (2020); Clinics Hospital of Ribeirão Preto (2013); Hospital Bandeirantes (2007); Fundação Oswaldo Cruz (2015); University of Glasgow (2025); Universidade Estadual Paulista (Unesp) (2009–2023)
Faculty Researcher
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Camila Silva's research focuses on understanding the toxicological effects of environmental contaminants and biological mechanisms underlying disease. Her work has investigated the impact of inorganic arsenic on neuronal development in zebrafish, revealing alterations in dopaminergic and motor neurons and identifying the Sonic hedgehog pathway as a potential mediator. Silva has also explored the role of the thyroid axis and stress response in temperature-induced sex reversal in males. Her recent publications include studies on the temporal dynamics of SARS-CoV-2 in wastewater, the metabolic role of the gut microbiome, and the signaling pathways involved in liver zonation. Silva has authored 54 publications with 396 citations and an h-index of 13. She collaborates with researchers at the National Center for Toxicological Research, including Volodymyr Tryndyak, Luísa Camacho, Nathan C. Twaddle, and Jyotshna Kanungo.
Metrics
- h-index: 13
- Publications: 54
- Citations: 398
Selected Publications
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N-Acetylcysteine Prevents Arsenic-Induced Apoptosis but Not Supernumerary Motor Neuron Development in Zebrafish Embryos: Assessment of Protein Carbonylation and the p53 Pathway (2026)
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Wastewater dataset on the SARS-CoV-2 sublineages circulating in Central Arkansas, USA, post-COVID-19 pandemic (2025)
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Acetyl L‐Carnitine Protects Zebrafish Embryos From Verapamil and Inorganic Arsenic‐Induced Cardiotoxicity and Developmental Toxicity With No Effect on Supernumerary Motor Neuron Development (2025)
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Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish (2023)
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Temporal dynamics of SARS-CoV-2 genome and detection of variants of concern in wastewater influent from two metropolitan areas in Arkansas (2022)
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A robust biostatistical method leverages informative but uncertainly determined qPCR data for biomarker detection, early diagnosis, and treatment (2022)
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Reproducibility Challenges for Biomarker Detection with Uncertain But Informative Experimental Data (2020)
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Data on the effect of heat and other technical variables on the detection of microRNAs in human serum (2019)
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Identification of whole blood mRNA and microRNA biomarkers of tissue damage and immune function resulting from amphetamine exposure or heat stroke in adult male rats (2019)
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Effects of a 28-day dietary co-exposure to melamine and cyanuric acid on the levels of serum microRNAs in male and female Fisher 344 rats (2016)
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Antioxidant Treatment and Alcoholism (2015)
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Human Respiratory Coronaviruses Detected In Patients with InfluenzaLike Illness in Arkansas, USA (2014)
Collaboration Network
Top Collaborators
- Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish
- Acetyl L‐Carnitine Protects Zebrafish Embryos From Verapamil and Inorganic Arsenic‐Induced Cardiotoxicity and Developmental Toxicity With No Effect on Supernumerary Motor Neuron Development
- Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish
- Acetyl L‐Carnitine Protects Zebrafish Embryos From Verapamil and Inorganic Arsenic‐Induced Cardiotoxicity and Developmental Toxicity With No Effect on Supernumerary Motor Neuron Development
- Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish
- Acetyl L‐Carnitine Protects Zebrafish Embryos From Verapamil and Inorganic Arsenic‐Induced Cardiotoxicity and Developmental Toxicity With No Effect on Supernumerary Motor Neuron Development
- A robust biostatistical method leverages informative but uncertainly determined qPCR data for biomarker detection, early diagnosis, and treatment
- A robust biostatistical method leverages informative but uncertainly determined qPCR data for biomarker detection, early diagnosis, and treatment
- A robust biostatistical method leverages informative but uncertainly determined qPCR data for biomarker detection, early diagnosis, and treatment
- A robust biostatistical method leverages informative but uncertainly determined qPCR data for biomarker detection, early diagnosis, and treatment
- Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish
- Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish
- Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish
- Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish
- Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish
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