Biography and Research Information
OverviewAI-generated summary
Bonnie Robinson's research investigates the molecular and cellular mechanisms underlying neurodevelopment and neurotoxicity. Her work has explored how environmental factors, such as inorganic arsenic, can alter neuronal development pathways. For instance, her 2022 publication in zebrafish demonstrated that inorganic arsenic specifically affects dopaminergic and motor neuron development through the Sonic hedgehog pathway, while not impacting serotonergic neurons. Further research in 2023 utilized gene expression analyses to identify potential mechanisms of inorganic arsenic-induced apoptosis in zebrafish, contributing to a deeper understanding of its toxicological effects.
In addition to environmental toxicology, Robinson's research interests extend to neuropharmacology and the identification of biomarkers for neurotoxicity. Her 2021 publication on the antidepressant actions of ketamine explored the potential role of L-type calcium channels. Concurrently, another 2021 publication focused on a proteomics approach to identify circulating biomarkers of central nervous system toxicity in rodent models. These studies collectively highlight her engagement with diverse aspects of neuroscience and toxicology, aiming to elucidate disease mechanisms and identify novel therapeutic targets or diagnostic indicators. Robinson has a scholarly impact indicated by an h-index of 20 and over 1,800 citations across 43 publications.
Metrics
- h-index: 21
- Publications: 43
- Citations: 1,901
Selected Publications
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Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish (2023)
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Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity (2021)
Collaboration Network
Top Collaborators
- Inorganic arsenic alters the development of dopaminergic neurons but not serotonergic neurons and induces motor neuron development via Sonic hedgehog pathway in zebrafish
- Antidepressant Actions of Ketamine: Potential Role of L-Type Calcium Channels
- Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish
- Inorganic arsenic alters the development of dopaminergic neurons but not serotonergic neurons and induces motor neuron development via Sonic hedgehog pathway in zebrafish
- Antidepressant Actions of Ketamine: Potential Role of L-Type Calcium Channels
- Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish
- Inorganic arsenic alters the development of dopaminergic neurons but not serotonergic neurons and induces motor neuron development via Sonic hedgehog pathway in zebrafish
- Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish
- Inorganic arsenic alters the development of dopaminergic neurons but not serotonergic neurons and induces motor neuron development via Sonic hedgehog pathway in zebrafish
- Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish
- Inorganic arsenic alters the development of dopaminergic neurons but not serotonergic neurons and induces motor neuron development via Sonic hedgehog pathway in zebrafish
- Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish
- Inorganic arsenic alters the development of dopaminergic neurons but not serotonergic neurons and induces motor neuron development via Sonic hedgehog pathway in zebrafish
- Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
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