Craig C. Porter
Professor
Also affiliated: University of Iowa (1993–2008); Arkansas Children's Hospital (2015); Children's National (1985); University of Nottingham (2011–2017); University of South Carolina (2014); George Washington University (1985); Medical College of Wisconsin (2012); Charles University (2023); University of Exeter (2023); University of Arkansas Medical Center (2025); Shriners Hospitals for Children - Galveston (2012–2020); University of Iowa Stead Family Children’s Hospital (2008); Thomayer University Hospital (2023); Shriners Hospitals for Children (2014–2017); Arkansas Children's Nutrition Center (2015–2026); University Hospital Kralovske Vinohrady (2023); Arkansas Children's Research Institute (2021–2026); University of Oklahoma Health Sciences Center (2021); The University of Texas Medical Branch at Galveston (2012–2023); University of Oklahoma (2021)
Peds Pediatrics, College of Medicine
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Craig C. Porter's research focuses on the metabolic stress response, particularly in the context of severe physiological stress such as burn trauma. His work investigates the role of brown adipose tissue in improving whole-body glucose homeostasis and insulin sensitivity in humans. Porter has studied the browning of subcutaneous white adipose tissue following severe adrenergic stress and examined the activation of brown adipose tissue and its systemic effects on lipid metabolism.
His research also extends to mitochondrial function in skeletal muscle, exploring how resistance exercise training alters these processes. Porter has compared the function of mitochondria in human and mouse brown adipose tissue, noting their comparability in UCP1 function. He has a publication record of over 200 articles and a citation count of nearly 6,000, with an h-index of 36, indicating a significant impact in his field. Porter leads a research group and collaborates with multiple colleagues at the University of Arkansas for Medical Sciences.
Metrics
- h-index: 36
- Publications: 172
- Citations: 5,867
Positions
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USDA-ARS Arkansas Children's Nutrition Center 2019–presentORCID
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Associate Profesor 2019–presentUniversity of Arkansas for Medical Sciences Pediatrics ORCID
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Arkansas Childrens Hospital Research Institute 2019–presentORCID
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Professor publications 2015–2026University of Arkansas for Medical Sciences Peds Pediatrics, College of Medicine Institutional directory
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Associate Professor 2018–2019University of Texas Medical Branch Department of Surgery ORCID
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Director 2016–2019Shriners Hospitals for Children Galveston Metabolism Unit ORCID
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Assistant Professor 2011–2018University of Texas Medical Branch Department of Surgery ORCID
Selected Publications
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Muscle glycogen metabolism is rapidly dysregulated in critical illness, which may have implications for muscle ATP resynthesis and ICU acquired weakness. (2026)
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C-955-06. Housing Temperature Alters the Pathophysiological and Proteomic Responses to Burn Injury in Mice (2026)
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C-955-10. Wound Excision Drives Hypermetabolism and Cachexia in Mice with Scald Burn Injuries (2026)
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566. Burn Injury Alters Peripheral Blood Mononuclear Cell Bioenergetics (2026)
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Inborn cardiorespiratory fitness and exercise training modulate brown adipose tissue function and plasticity in early life (2026)
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Carnitine deficiency alters fuel metabolism and voluntary wheel running in mice (2026)
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Characterizing Human Milk RNA Degradation Over Time to Optimize Storage of Human Milk for RNA Quality (2026)
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Novel murine partial tracheal occlusion model with altered respiratory dynamics (2025)
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Mitochondrial respiratory function in human platelets: Influence of sample preparation, assay buffer, and instrumental platform (2025)
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Shared and distinct adaptations to early‐life exercise training based on inborn fitness (2025)
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Determination of Burn Size in Mouse Models of Burn Injury: A Scoping Review of Studies Leveraging Scald and Contact Burns (2025)
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Impact of the metabolic disease status in obesity and surgical weight loss on human adipose tissue bioenergetics (2025)
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Shared and distinct adaptations to early-life exercise training based on inborn fitness (2024)
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HOUSING TEMPERATURE ALTERS BURN-INDUCED HYPERMETABOLISM IN MICE (2024)
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Maternal hemoglobin A1c and left ventricular hypertrophy in infants of mothers with pre-gestational diabetes (2024)
Grants & Funding
As listed on this researcher's institutional profile.
- ACRI GF034110 Porter The Role of the Mitochondrion in the Metabolic Stress Response to Burn Trauma NIH/Nat. Inst. of General Medical Sciences Principal Investigator
Collaboration Network
Top Collaborators
- A modest change in housing temperature alters whole body energy expenditure and adipocyte thermogenic capacity in mice
- Parental cardiorespiratory fitness influences early life energetics and metabolic health
- Protonophore treatment augments energy expenditure in mice housed at thermoneutrality
- Shared and distinct adaptations to early‐life exercise training based on inborn fitness
- Housing Temperature Alters Energy Expenditure and Adipose Tissue Bioenergetics in Mice
Showing 5 of 11 shared publications
- Parental cardiorespiratory fitness influences early life energetics and metabolic health
- Shared and distinct adaptations to early‐life exercise training based on inborn fitness
- Predictors of Platelet Mitochondria Respiration in Children – The Arkansas Active Kids Study
- Shared and distinct adaptations to early-life exercise training based on inborn fitness
- Mitochondrial respiratory function in human platelets: Influence of sample preparation, assay buffer, and instrumental platform
Showing 5 of 9 shared publications
- A modest change in housing temperature alters whole body energy expenditure and adipocyte thermogenic capacity in mice
- Parental cardiorespiratory fitness influences early life energetics and metabolic health
- Protonophore treatment augments energy expenditure in mice housed at thermoneutrality
- Shared and distinct adaptations to early‐life exercise training based on inborn fitness
- Maternal hemoglobin A1c and left ventricular hypertrophy in infants of mothers with pre-gestational diabetes
Showing 5 of 9 shared publications
- Shared and distinct adaptations to early‐life exercise training based on inborn fitness
- Determination of Burn Size in Mouse Models of Burn Injury: A Scoping Review of Studies Leveraging Scald and Contact Burns
- HOUSING TEMPERATURE ALTERS BURN-INDUCED HYPERMETABOLISM IN MICE
- Shared and distinct adaptations to early-life exercise training based on inborn fitness
- Inborn cardiorespiratory fitness and exercise training modulate brown adipose tissue function and plasticity in early life
Showing 5 of 8 shared publications
- Rehabilitative Exercise Training for Burn Injury
- Current problems in burn hypermetabolism
- The impact of catecholamines on skeletal muscle following massive burns: Friend or foe?
- Liver-Specific Nonviral Gene Delivery of Fibroblast Growth Factor 21 Protein Expression in Mice Regulates Body Mass and White/Brown Fat Respiration
- Skeletal Muscle Bioenergetics in Critical Limb Ischemia and Diabetes
Showing 5 of 7 shared publications
- A modest change in housing temperature alters whole body energy expenditure and adipocyte thermogenic capacity in mice
- Parental cardiorespiratory fitness influences early life energetics and metabolic health
- Protonophore treatment augments energy expenditure in mice housed at thermoneutrality
- Shared and distinct adaptations to early‐life exercise training based on inborn fitness
- Shared and distinct adaptations to early-life exercise training based on inborn fitness
Showing 5 of 7 shared publications
- Determination of Burn Size in Mouse Models of Burn Injury: A Scoping Review of Studies Leveraging Scald and Contact Burns
- HOUSING TEMPERATURE ALTERS BURN-INDUCED HYPERMETABOLISM IN MICE
- Carnitine deficiency alters fuel metabolism and voluntary wheel running in mice
- Inborn cardiorespiratory fitness and exercise training modulate brown adipose tissue function and plasticity in early life
- 566. Burn Injury Alters Peripheral Blood Mononuclear Cell Bioenergetics
Showing 5 of 7 shared publications
- Current problems in burn hypermetabolism
- Supervised Resistance Training on Functional Capacity, Muscle Strength and Vascular Function in Peripheral Artery Disease: An Updated Systematic Review and Meta-Analysis
- The impact of catecholamines on skeletal muscle following massive burns: Friend or foe?
- Skeletal Muscle Bioenergetics in Critical Limb Ischemia and Diabetes
- In-Brief
- Supervised Resistance Training on Functional Capacity, Muscle Strength and Vascular Function in Peripheral Artery Disease: An Updated Systematic Review and Meta-Analysis
- Liver-Specific Nonviral Gene Delivery of Fibroblast Growth Factor 21 Protein Expression in Mice Regulates Body Mass and White/Brown Fat Respiration
- Skeletal Muscle Bioenergetics in Critical Limb Ischemia and Diabetes
- SAT-652 Increased Fibroblast Growth Factor 21 Protein Expression via in Vivo Delivery of a Liver-Specific Expression Plasmid
- The Effect of Lipolysis Inhibitor Acipimox on Brown Adipose Tissue Bioenergetics and Uncoupling Protein Abundance in Severely Burned Rats
- Parental cardiorespiratory fitness influences early life energetics and metabolic health
- Shared and distinct adaptations to early‐life exercise training based on inborn fitness
- Shared and distinct adaptations to early-life exercise training based on inborn fitness
- Inborn cardiorespiratory fitness and exercise training modulate brown adipose tissue function and plasticity in early life
- HOUSING TEMPERATURE ALTERS BURN-INDUCED HYPERMETABOLISM IN MICE
- 566. Burn Injury Alters Peripheral Blood Mononuclear Cell Bioenergetics
- C-955-10. Wound Excision Drives Hypermetabolism and Cachexia in Mice with Scald Burn Injuries
- C-955-06. Housing Temperature Alters the Pathophysiological and Proteomic Responses to Burn Injury in Mice
- Determination of Burn Size in Mouse Models of Burn Injury: A Scoping Review of Studies Leveraging Scald and Contact Burns
- 566. Burn Injury Alters Peripheral Blood Mononuclear Cell Bioenergetics
- C-955-10. Wound Excision Drives Hypermetabolism and Cachexia in Mice with Scald Burn Injuries
- C-955-06. Housing Temperature Alters the Pathophysiological and Proteomic Responses to Burn Injury in Mice
- Mortality among infants of diabetic mothers with hypertrophic cardiomyopathy
- Maternal hemoglobin A1c and left ventricular hypertrophy in infants of mothers with pre-gestational diabetes
- Carnitine deficiency alters fuel metabolism and voluntary wheel running in mice
- Parental cardiorespiratory fitness influences early life energetics and metabolic health
- Shared and distinct adaptations to early‐life exercise training based on inborn fitness
- Shared and distinct adaptations to early-life exercise training based on inborn fitness
- Current problems in burn hypermetabolism
- In-Brief
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