Biography and Research Information
OverviewAI-generated summary
Eva Allen's research focuses on the molecular mechanisms underlying cellular responses to therapeutic interventions and disease. Her work investigates the signaling pathways activated by chimeric antigen receptors (CARs) in human regulatory T cells, aiming to optimize these receptors to mitigate pro-inflammatory responses. Allen also studies the role of Sirtuin 2 (SIRT2) in mitigating radiation-induced oral mucositis. This research explores SIRT2's function in promoting DNA double-strand break repair within epithelial stem cells, highlighting a potential therapeutic target for managing side effects of radiation therapy. Allen has published five papers and has a citation count of 25, with an h-index of 2. She collaborates with several researchers at the University of Arkansas for Medical Sciences, including Ratan Sadhukhan and Forouh Kalantari.
Metrics
- h-index: 3
- Publications: 9
- Citations: 33
Selected Publications
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SIRT2 mitigates radiation-induced oral mucositis by promoting homologous recombination-mediated DNA double-strand break repair in epithelial stem cells (2026)
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Sirt2 Mitigates Radiation-Induced Oral Mucositis by Promoting Homologous Recombination-Mediated DNA Double-Strand Break Repair in Epithelial Stem Cells (2025)
Collaboration Network
Top Collaborators
Russell W Cochrane
Medical University of South Carolina (US)
3 shared publications
- High-affinity chimeric antigen receptor signaling induces an inflammatory program in human regulatory T cells
- High affinity chimeric antigen receptor signaling induces an inflammatory program in human regulatory T cells
- Optimizing chimeric antigen receptors to minimize pro-inflammatory programs in human regulatory T cells 3265
Rob A. Robino
Medical University of South Carolina (US)
3 shared publications
- High-affinity chimeric antigen receptor signaling induces an inflammatory program in human regulatory T cells
- High affinity chimeric antigen receptor signaling induces an inflammatory program in human regulatory T cells
- Optimizing chimeric antigen receptors to minimize pro-inflammatory programs in human regulatory T cells 3265
Bryan Granger
Medical University of South Carolina (US)
3 shared publications
- High-affinity chimeric antigen receptor signaling induces an inflammatory program in human regulatory T cells
- High affinity chimeric antigen receptor signaling induces an inflammatory program in human regulatory T cells
- Optimizing chimeric antigen receptors to minimize pro-inflammatory programs in human regulatory T cells 3265
Silvia G. Vaena
Medical University of South Carolina (US)
3 shared publications
- High-affinity chimeric antigen receptor signaling induces an inflammatory program in human regulatory T cells
- High affinity chimeric antigen receptor signaling induces an inflammatory program in human regulatory T cells
- Optimizing chimeric antigen receptors to minimize pro-inflammatory programs in human regulatory T cells 3265
Martin J. Romeo
Medical University of South Carolina (US)
3 shared publications
- High-affinity chimeric antigen receptor signaling induces an inflammatory program in human regulatory T cells
- High affinity chimeric antigen receptor signaling induces an inflammatory program in human regulatory T cells
- Optimizing chimeric antigen receptors to minimize pro-inflammatory programs in human regulatory T cells 3265
Stefano Berto
Medical University of South Carolina (US)
3 shared publications
- High-affinity chimeric antigen receptor signaling induces an inflammatory program in human regulatory T cells
- High affinity chimeric antigen receptor signaling induces an inflammatory program in human regulatory T cells
- Optimizing chimeric antigen receptors to minimize pro-inflammatory programs in human regulatory T cells 3265
Aguirre A. de Cubas
Medical University of South Carolina (US)
2 shared publications
- High-affinity chimeric antigen receptor signaling induces an inflammatory program in human regulatory T cells
- High affinity chimeric antigen receptor signaling induces an inflammatory program in human regulatory T cells
Leonardo M. R. Ferreira
Medical University of South Carolina (US)
2 shared publications
- High-affinity chimeric antigen receptor signaling induces an inflammatory program in human regulatory T cells
- High affinity chimeric antigen receptor signaling induces an inflammatory program in human regulatory T cells
Ratan Sadhukhan
University of Arkansas for Medical Sciences
2 shared publications
In database
- Sirt2 Mitigates Radiation-Induced Oral Mucositis by Promoting Homologous Recombination-Mediated DNA Double-Strand Break Repair in Epithelial Stem Cells
- SIRT2 mitigates radiation-induced oral mucositis by promoting homologous recombination-mediated DNA double-strand break repair in epithelial stem cells
Mousumi Patra
University of Arkansas for Medical Sciences
1 shared publication
In database
- Sirt2 Mitigates Radiation-Induced Oral Mucositis by Promoting Homologous Recombination-Mediated DNA Double-Strand Break Repair in Epithelial Stem Cells
Forouh Kalantari
University of Arkansas for Medical Sciences
1 shared publication
In database
- Sirt2 Mitigates Radiation-Induced Oral Mucositis by Promoting Homologous Recombination-Mediated DNA Double-Strand Break Repair in Epithelial Stem Cells
Heba Allam
University of Arkansas for Medical Sciences
1 shared publication
In database
- Sirt2 Mitigates Radiation-Induced Oral Mucositis by Promoting Homologous Recombination-Mediated DNA Double-Strand Break Repair in Epithelial Stem Cells
Fen Xia
University of Arkansas for Medical Sciences
1 shared publication
In database
- Sirt2 Mitigates Radiation-Induced Oral Mucositis by Promoting Homologous Recombination-Mediated DNA Double-Strand Break Repair in Epithelial Stem Cells
Aguirre De Cubas
Medical University of South Carolina (US)
1 shared publication
- Optimizing chimeric antigen receptors to minimize pro-inflammatory programs in human regulatory T cells 3265
Leonardo Ferreira
MUSC Hollings Cancer Center (US)
1 shared publication
- Optimizing chimeric antigen receptors to minimize pro-inflammatory programs in human regulatory T cells 3265
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