Eva Allen
Researcher
Also affiliated: Argonne National Laboratory (2026); Medical University of South Carolina (2023–2025); MUSC Hollings Cancer Center (2023–2025); Winthrop Rockefeller Foundation (2026)
Unknown Researcher
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Eva Allen's research focuses on the mechanisms underlying radiation-induced damage and repair in epithelial cells, particularly in the context of head and neck cancers. Her work investigates the role of specific proteins, such as Sirtuin 2 (SIRT2), in mitigating radiation-induced oral mucositis by promoting DNA double-strand break repair through homologous recombination. Allen also explores strategies for optimizing chimeric antigen receptors (CARs) to minimize pro-inflammatory programs in human regulatory T cells, a relevant area for cancer immunotherapy. Her research involves animal models and in vitro studies using epithelial stem cells. Allen collaborates with researchers at the University of Arkansas for Medical Sciences, including Ratan Sadhukhan and Forouh Kalantari, on these investigations. She has authored nine publications and her work has been cited 40 times, with an h-index of 3.
Metrics
- h-index: 3
- Publications: 9
- Citations: 43
Selected Publications
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SIRT2 mitigates radiation-induced oral mucositis by promoting homologous recombination-mediated DNA double-strand break repair in epithelial stem cells (2026)
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Sirt2 Mitigates Radiation-Induced Oral Mucositis by Promoting Homologous Recombination-Mediated DNA Double-Strand Break Repair in Epithelial Stem Cells (2025)
Collaboration Network
Top Collaborators
- Sirt2 Mitigates Radiation-Induced Oral Mucositis by Promoting Homologous Recombination-Mediated DNA Double-Strand Break Repair in Epithelial Stem Cells
- SIRT2 mitigates radiation-induced oral mucositis by promoting homologous recombination-mediated DNA double-strand break repair in epithelial stem cells
- Sirt2 Mitigates Radiation-Induced Oral Mucositis by Promoting Homologous Recombination-Mediated DNA Double-Strand Break Repair in Epithelial Stem Cells
- Sirt2 Mitigates Radiation-Induced Oral Mucositis by Promoting Homologous Recombination-Mediated DNA Double-Strand Break Repair in Epithelial Stem Cells
- Sirt2 Mitigates Radiation-Induced Oral Mucositis by Promoting Homologous Recombination-Mediated DNA Double-Strand Break Repair in Epithelial Stem Cells
- Sirt2 Mitigates Radiation-Induced Oral Mucositis by Promoting Homologous Recombination-Mediated DNA Double-Strand Break Repair in Epithelial Stem Cells
- SIRT2 mitigates radiation-induced oral mucositis by promoting homologous recombination-mediated DNA double-strand break repair in epithelial stem cells
- SIRT2 mitigates radiation-induced oral mucositis by promoting homologous recombination-mediated DNA double-strand break repair in epithelial stem cells
- SIRT2 mitigates radiation-induced oral mucositis by promoting homologous recombination-mediated DNA double-strand break repair in epithelial stem cells
- SIRT2 mitigates radiation-induced oral mucositis by promoting homologous recombination-mediated DNA double-strand break repair in epithelial stem cells
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