Match tier
Institution-verified
Presence
Current · Arkansas
Last published
2026
Sources
Institutional record
Refreshed
2026-08-15
H
Heba Allam
Sourced from institutional research profiles (UAMS TRI or ARA).
Assistant Professor
Also affiliated: University of Denver (2009); Cairo University (2017); Hamad General Hospital (2016); University of Oklahoma Health Sciences Center (2011); Hamad Medical Corporation (2016); Menoufia University (2013–2024); University of Oklahoma (2013)
Faculty Researcher
7 h-index
26 pubs
318 cited
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Heba Allam's research investigates molecular mechanisms underlying cellular responses to DNA damage and therapeutic interventions. Her work has focused on the role of specific proteins, such as SIRT2 and GSK3B, in DNA repair pathways and their influence on cancer treatment outcomes, particularly in the context of radiation mucositis and PARP1 inhibition. Allam has published on these topics, contributing to a better understanding of how these pathways affect tumor response and cellular resilience. Her scholarship metrics include an h-index of 7, with 26 total publications and 313 total citations. Allam collaborates with researchers at the University of Arkansas for Medical Sciences, including Mousumi Patra, Ratan Sadhukhan, Forouh Kalantari, and Brian Koss, with whom she shares multiple publications.
Metrics
- h-index: 7
- Publications: 26
- Citations: 318
Selected Publications
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Abstract B040: 53BP1-T336 Phosphorylation by GSK3β regulates DNA damage response and radioresistance in GBM (2026)
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SIRT2 mitigates radiation-induced oral mucositis by promoting homologous recombination-mediated DNA double-strand break repair in epithelial stem cells (2026)
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GSK3B directs DNA repair choice and determines tumor response to PARP1 inhibition independent of BRCA1 (2025)
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Sirt2 Mitigates Radiation-Induced Oral Mucositis by Promoting Homologous Recombination-Mediated DNA Double-Strand Break Repair in Epithelial Stem Cells (2025)
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376 Inhibition of GSK3β-mediated 53BP1 T334 phosphorylation in T cells enhances infiltration and cytotoxicity against head and neck squamous cell carcinoma (2024)
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Abstract PR011: Novel role of glycogen synthase kinase-3β in determining cancer cell response to PARPi through regulation of 53BP1 function (2024)
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Novel Role of Glycogen Synthase Kinase-3β in Determining Cancer Cell Response to Genotoxic Stress through Regulation of 53BP1 Function (2023)
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Glycomic Analysis of Membrane Glycoproteins with Bisecting Glycosylation from Ovarian Cancer Tissues Reveals Novel Structures and Functions (2014)
Grants & Funding
As listed on this researcher's institutional profile.
- The novel role of Sirtuin 2 in regulation of transcription-associated DNA damage repair NIH
- Glycomics Laboratory for the Early Detection of Epithelial Ovarian Cancer NIH
- Novel functions of Pyruvate kinase M2 in DNA double-strand break repair NIH
Collaboration Network
Top Collaborators
Mousumi Patra
University of Arkansas for Medical Sciences
5 shared publications
In database
- Novel Role of Glycogen Synthase Kinase-3β in Determining Cancer Cell Response to Genotoxic Stress through Regulation of 53BP1 Function
- Abstract PR011: Novel role of glycogen synthase kinase-3β in determining cancer cell response to PARPi through regulation of 53BP1 function
- 376 Inhibition of GSK3β-mediated 53BP1 T334 phosphorylation in T cells enhances infiltration and cytotoxicity against head and neck squamous cell carcinoma
- Sirt2 Mitigates Radiation-Induced Oral Mucositis by Promoting Homologous Recombination-Mediated DNA Double-Strand Break Repair in Epithelial Stem Cells
- GSK3B directs DNA repair choice and determines tumor response to PARP1 inhibition independent of BRCA1
Fen Xia
University of Arkansas for Medical Sciences
4 shared publications
In database
- Abstract PR011: Novel role of glycogen synthase kinase-3β in determining cancer cell response to PARPi through regulation of 53BP1 function
- 376 Inhibition of GSK3β-mediated 53BP1 T334 phosphorylation in T cells enhances infiltration and cytotoxicity against head and neck squamous cell carcinoma
- Sirt2 Mitigates Radiation-Induced Oral Mucositis by Promoting Homologous Recombination-Mediated DNA Double-Strand Break Repair in Epithelial Stem Cells
- GSK3B directs DNA repair choice and determines tumor response to PARP1 inhibition independent of BRCA1
Ratan Sadhukhan
Winthrop Rockefeller Foundation
4 shared publications
In database
- 376 Inhibition of GSK3β-mediated 53BP1 T334 phosphorylation in T cells enhances infiltration and cytotoxicity against head and neck squamous cell carcinoma
- Sirt2 Mitigates Radiation-Induced Oral Mucositis by Promoting Homologous Recombination-Mediated DNA Double-Strand Break Repair in Epithelial Stem Cells
- GSK3B directs DNA repair choice and determines tumor response to PARP1 inhibition independent of BRCA1
- SIRT2 mitigates radiation-induced oral mucositis by promoting homologous recombination-mediated DNA double-strand break repair in epithelial stem cells
Scarlett Acklin
Duke University (US)
2 shared publications
- Novel Role of Glycogen Synthase Kinase-3β in Determining Cancer Cell Response to Genotoxic Stress through Regulation of 53BP1 Function
- Abstract PR011: Novel role of glycogen synthase kinase-3β in determining cancer cell response to PARPi through regulation of 53BP1 function
Eva Allen
Winthrop Rockefeller Foundation
2 shared publications
In database
- Sirt2 Mitigates Radiation-Induced Oral Mucositis by Promoting Homologous Recombination-Mediated DNA Double-Strand Break Repair in Epithelial Stem Cells
- SIRT2 mitigates radiation-induced oral mucositis by promoting homologous recombination-mediated DNA double-strand break repair in epithelial stem cells
R. Kiss
University of Arkansas for Medical Sciences (US)
1 shared publication
- Novel Role of Glycogen Synthase Kinase-3β in Determining Cancer Cell Response to Genotoxic Stress through Regulation of 53BP1 Function
Jacqueline Y. K. Leung
University of Arkansas for Medical Sciences (US)
1 shared publication
- Novel Role of Glycogen Synthase Kinase-3β in Determining Cancer Cell Response to Genotoxic Stress through Regulation of 53BP1 Function
Xia Fang
University of Arkansas for Medical Sciences (US)
1 shared publication
- Novel Role of Glycogen Synthase Kinase-3β in Determining Cancer Cell Response to Genotoxic Stress through Regulation of 53BP1 Function
Kirk L. West
University of Arkansas for Medical Sciences
1 shared publication
In database
- Abstract PR011: Novel role of glycogen synthase kinase-3β in determining cancer cell response to PARPi through regulation of 53BP1 function
Justin Leung
1 shared publication
- Abstract PR011: Novel role of glycogen synthase kinase-3β in determining cancer cell response to PARPi through regulation of 53BP1 function
Jessica L Kelliher
University of Arkansas for Medical Sciences
1 shared publication
In database
- 376 Inhibition of GSK3β-mediated 53BP1 T334 phosphorylation in T cells enhances infiltration and cytotoxicity against head and neck squamous cell carcinoma
Yong‐Chen Lu
University of Arkansas for Medical Sciences
1 shared publication
In database
- 376 Inhibition of GSK3β-mediated 53BP1 T334 phosphorylation in T cells enhances infiltration and cytotoxicity against head and neck squamous cell carcinoma
Brian Koss
University of Arkansas for Medical Sciences
1 shared publication
In database
- 376 Inhibition of GSK3β-mediated 53BP1 T334 phosphorylation in T cells enhances infiltration and cytotoxicity against head and neck squamous cell carcinoma
Forouh Kalantari
University of Arkansas for Medical Sciences
1 shared publication
In database
- Sirt2 Mitigates Radiation-Induced Oral Mucositis by Promoting Homologous Recombination-Mediated DNA Double-Strand Break Repair in Epithelial Stem Cells
Scarlett Acklin-Wehnert
Duke Medical Center (US)
1 shared publication
- GSK3B directs DNA repair choice and determines tumor response to PARP1 inhibition independent of BRCA1
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