Jeffrey L. Osborn
Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.
Vice President of Research and Professor
Also affiliated: University of Southern California (1990); University of Iowa (1981–1983); University of Kentucky (2006–2022); Medical College of Wisconsin (1984–2001); Dow Chemical (United States) (1976); Iowa City VA Medical Center (1981–1983); Michigan State University (1976–2019)
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Jeffrey L. Osborn's research focuses on understanding the mechanisms of hypertension and renal function, particularly in animal models. His work has investigated the role of the sympathetic nervous system and various receptors in regulating blood pressure and kidney function. This includes studies on the antinatriuretic and renin secretion responses to renal nerve stimulation in dogs, examining the mediation of these responses by adrenoceptors and beta-1 receptors.
Osborn has also published on the long-term consequences of renal ischemic injury, demonstrating permanent damage to peritubular capillaries and its influence on kidney function. His research includes the development and utilization of animal models for studying hypertension, contributing to a broader understanding of cardiovascular health. His scholarship metrics include an h-index of 24, with 145 total publications and over 3,100 citations.
Metrics
- h-index: 24
- Publications: 142
- Citations: 3,023
Positions
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Vice President of Research and Professor 2022–presentArkansas Colleges of Health Education Pathophysiology ORCID
Selected Publications
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Human soluble prorenin receptor expressed in mouse renal collecting duct shows sex-specific effect on cardiorenal function (2024)
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Renal-Derived Human sPRR Does Not Increase Blood Pressure in High Fat Diet Mice (2024)
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Expression of human sPRR in renal collecting duct cells increases blood pressure in female mice independently of its circulating levels (2023)
Collaboration Network
Top Collaborators
- Human soluble prorenin receptor expressed in mouse renal collecting duct shows sex-specific effect on cardiorenal function
- Expression of human sPRR in renal collecting duct cells increases blood pressure in female mice independently of its circulating levels
- Renal-Derived Human sPRR Does Not Increase Blood Pressure in High Fat Diet Mice
- Human soluble prorenin receptor expressed in mouse renal collecting duct shows sex-specific effect on cardiorenal function
- Expression of human sPRR in renal collecting duct cells increases blood pressure in female mice independently of its circulating levels
- Renal-Derived Human sPRR Does Not Increase Blood Pressure in High Fat Diet Mice
- Human soluble prorenin receptor expressed in mouse renal collecting duct shows sex-specific effect on cardiorenal function
- Expression of human sPRR in renal collecting duct cells increases blood pressure in female mice independently of its circulating levels
- Renal-Derived Human sPRR Does Not Increase Blood Pressure in High Fat Diet Mice
- Human soluble prorenin receptor expressed in mouse renal collecting duct shows sex-specific effect on cardiorenal function
- Expression of human sPRR in renal collecting duct cells increases blood pressure in female mice independently of its circulating levels
- Human soluble prorenin receptor expressed in mouse renal collecting duct shows sex-specific effect on cardiorenal function
- Renal-Derived Human sPRR Does Not Increase Blood Pressure in High Fat Diet Mice
- Expression of human sPRR in renal collecting duct cells increases blood pressure in female mice independently of its circulating levels
- Renal-Derived Human sPRR Does Not Increase Blood Pressure in High Fat Diet Mice
- Human soluble prorenin receptor expressed in mouse renal collecting duct shows sex-specific effect on cardiorenal function
- Human soluble prorenin receptor expressed in mouse renal collecting duct shows sex-specific effect on cardiorenal function
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