Match tier
Listed
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-08-15
J
Joseph C. Bryant
Researcher
Also affiliated: Mississippi State University (2016)
Unknown Researcher
4 h-index
9 pubs
92 cited
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Joseph C. Bryant's research focuses on the molecular mechanisms governing bacterial cell division and cell wall synthesis in *Escherichia coli*. His work investigates how gene expression and metabolic pathways impact bacterial growth and structure. Recent publications explore the role of specific proteins, such as the DigH glycosyl hydrolase, in daughter cell separation and how dysregulation of nucleotidyltransferases can lead to defects in division and surface glycan production by altering metabolite levels.
Bryant has collaborated with researchers at the University of Arkansas for Medical Sciences, including Matthew A. Jorgenson, Intawat Nookaew, Alongkorn Kurilung, and Emily J. Robbs, on multiple publications. His scholarship metrics include an h-index of 4, with 9 total publications and 88 citations. His recent activity indicates ongoing contributions to the field of bacterial genetics and cell biology.
Metrics
- h-index: 4
- Publications: 9
- Citations: 92
Selected Publications
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Dysregulation of a nucleotidyltransferase induces division and surface glycan defects in <i>Escherichia coli</i> by altering related metabolite levels (2026)
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The DigH glycosyl hydrolase is conditionally required for daughter cell separation in <i>Escherichia coli</i> (2025)
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Engineering Escherichia coli for increased Und-P availability leads to material improvements in glycan expression technology (2024)
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A genetic screen to identify factors affected by undecaprenyl phosphate recycling uncovers novel connections to morphogenesis in <i>Escherichia coli</i> (2020)
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A genetic screen to identify factors affected by undecaprenyl phosphate recycling uncovers novel connections to morphogenesis in <i>Escherichia coli</i> (2020)
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Simultaneously inhibiting undecaprenyl phosphate production and peptidoglycan synthases promotes rapid lysis in <i>Escherichia coli</i> (2019)
Collaboration Network
Top Collaborators
Matthew A. Jorgenson
University of Arkansas for Medical Sciences
3 shared publications
In database
- Engineering Escherichia coli for increased Und-P availability leads to material improvements in glycan expression technology
- The DigH glycosyl hydrolase is conditionally required for daughter cell separation in <i>Escherichia coli</i>
- Dysregulation of a nucleotidyltransferase induces division and surface glycan defects in <i>Escherichia coli</i> by altering related metabolite levels
Emily J. Kay
London School of Hygiene & Tropical Medicine (GB)
1 shared publication
- Engineering Escherichia coli for increased Und-P availability leads to material improvements in glycan expression technology
Manoj K. Dooda
University of North Carolina at Charlotte (US)
1 shared publication
- Engineering Escherichia coli for increased Und-P availability leads to material improvements in glycan expression technology
Amanda J. Reid
University of North Carolina at Charlotte (US)
1 shared publication
- Engineering Escherichia coli for increased Und-P availability leads to material improvements in glycan expression technology
Brendan W. Wren
London School of Hygiene & Tropical Medicine (GB)
1 shared publication
- Engineering Escherichia coli for increased Und-P availability leads to material improvements in glycan expression technology
Jerry M. Troutman
University of North Carolina at Charlotte (US)
1 shared publication
- Engineering Escherichia coli for increased Und-P availability leads to material improvements in glycan expression technology
Emily J. Robbs
University of Arkansas for Medical Sciences
1 shared publication
In database
- The DigH glycosyl hydrolase is conditionally required for daughter cell separation in <i>Escherichia coli</i>
Alongkorn Kurilung
University of Arkansas for Medical Sciences
1 shared publication
In database
- The DigH glycosyl hydrolase is conditionally required for daughter cell separation in <i>Escherichia coli</i>
Brittney A. Dinkel
Buena Vista University (US)
1 shared publication
- The DigH glycosyl hydrolase is conditionally required for daughter cell separation in <i>Escherichia coli</i>
Intawat Nookaew
University of Arkansas for Medical Sciences
1 shared publication
In database
- The DigH glycosyl hydrolase is conditionally required for daughter cell separation in <i>Escherichia coli</i>
Maggie Zheng
New York University (US)
1 shared publication
- Dysregulation of a nucleotidyltransferase induces division and surface glycan defects in <i>Escherichia coli</i> by altering related metabolite levels
Andrea Koid
New York University (US)
1 shared publication
- Dysregulation of a nucleotidyltransferase induces division and surface glycan defects in <i>Escherichia coli</i> by altering related metabolite levels
Mia Sheshova
New York University (US)
1 shared publication
- Dysregulation of a nucleotidyltransferase induces division and surface glycan defects in <i>Escherichia coli</i> by altering related metabolite levels
Hanee Kim
New York University (US)
1 shared publication
- Dysregulation of a nucleotidyltransferase induces division and surface glycan defects in <i>Escherichia coli</i> by altering related metabolite levels
Tania J. Lupoli
New York University (US)
1 shared publication
- Dysregulation of a nucleotidyltransferase induces division and surface glycan defects in <i>Escherichia coli</i> by altering related metabolite levels
Similar Researchers
Based on overlapping research topics
Matthew A. Jorgenson
University of Arkansas for Medical Sciences
Bacterial Genetics and Biotechnology
Microbial infections and disease research
Host-Pathogen Interactions
Emily J. Robbs
University of Arkansas for Medical Sciences
Bacterial Genetics and Biotechnology
Microbial infections and disease research
Host-Pathogen Interactions
K. Shankar
University of Arkansas for Medical Sciences
Bacterial Genetics and Biotechnology
Microbial infections and disease research
Host-Pathogen Interactions
John Harris
University of Arkansas for Medical Sciences
Bacterial Genetics and Biotechnology
Microbial infections and disease research
Host-Pathogen Interactions
Kathy D. Eisenach
University of Arkansas for Medical Sciences
Bacterial Genetics and Biotechnology
Microbial infections and disease research
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Janet C. Bryant
University of Arkansas for Medical Sciences
Microbial infections and disease research
Host-Pathogen Interactions
Cellular transport and secretion