Match tier
Listed
Presence
Current · Arkansas
Last published
2025
Sources
OpenAlex · ORCID
Refreshed
2026-08-18
M
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Madeleine G. Scott's research focuses on the molecular mechanisms of bacterial pathogenesis, particularly concerning the bacterium *Yersinia pestis*. Her work investigates how this pathogen regulates the expression of virulence factors and interacts with host immune responses. Scott has studied the role of specific bacterial proteins, such as BipA, in regulating pesticin production and the type VI secretion system, which is crucial for bacterial competition and virulence. Additionally, her research has explored the regulation of plasminogen activator protease by the PhoP/PhoQ two-component system in *Yersinia pestis*. Her publications also examine the host's immune response, specifically the contribution of Interleukin-17 to neutrophil infiltration in the lungs during pneumonic plague in animal models.
Metrics
- h-index: 1
- Publications: 3
- Citations: 9
Selected Publications
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<i>Yersinia pestis</i> plasminogen activator protease is regulated by the PhoP/PhoQ two-component system (2025)
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<i>Yersinia pestis</i> BipA is a novel regulator of pesticin and a type 6 secretion system (2025)
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Pulmonary Expression of Interleukin-17 Contributes to Neutrophil Infiltration into the Lungs during Pneumonic Plague (2023)
Collaboration Network
Top Collaborators
Kenneth T. Appell
University of Arkansas for Medical Sciences
3 shared publications
In database
- Pulmonary Expression of Interleukin-17 Contributes to Neutrophil Infiltration into the Lungs during Pneumonic Plague
- <i>Yersinia pestis</i> BipA is a novel regulator of pesticin and a type 6 secretion system
- <i>Yersinia pestis</i> plasminogen activator protease is regulated by the PhoP/PhoQ two-component system
Roger D. Pechous
East Carolina University (US)
3 shared publications
- Pulmonary Expression of Interleukin-17 Contributes to Neutrophil Infiltration into the Lungs during Pneumonic Plague
- <i>Yersinia pestis</i> BipA is a novel regulator of pesticin and a type 6 secretion system
- <i>Yersinia pestis</i> plasminogen activator protease is regulated by the PhoP/PhoQ two-component system
Wanfeng Guo
University of Arkansas for Medical Sciences
2 shared publications
In database
- <i>Yersinia pestis</i> BipA is a novel regulator of pesticin and a type 6 secretion system
- <i>Yersinia pestis</i> plasminogen activator protease is regulated by the PhoP/PhoQ two-component system
Jon S. Blevins
University of Arkansas for Medical Sciences
2 shared publications
In database
- <i>Yersinia pestis</i> BipA is a novel regulator of pesticin and a type 6 secretion system
- <i>Yersinia pestis</i> plasminogen activator protease is regulated by the PhoP/PhoQ two-component system
Hayley M. Theriot
University of Arkansas for Medical Sciences
1 shared publication
In database
- Pulmonary Expression of Interleukin-17 Contributes to Neutrophil Infiltration into the Lungs during Pneumonic Plague
Priyangi A. Malaviarachchi
University of Arkansas for Medical Sciences
1 shared publication
In database
- Pulmonary Expression of Interleukin-17 Contributes to Neutrophil Infiltration into the Lungs during Pneumonic Plague
Srijon K. Banerjee
University of Pittsburgh
1 shared publication
In database
- Pulmonary Expression of Interleukin-17 Contributes to Neutrophil Infiltration into the Lungs during Pneumonic Plague
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