Biography and Research Information
OverviewAI-generated summary
Hayley M. Theriot's research focuses on understanding host responses to infectious agents and the impact of genetic conditions on disease severity. Her work has investigated the role of specific immune molecules, such as Interleukin-17, in contributing to lung inflammation during pneumonic plague, a severe bacterial infection. Theriot has also examined how SARS-CoV-2 infection affects individuals with Down syndrome, finding that this genetic condition can lead to heightened disease severity and mortality in mouse models. Her collaborative network includes researchers from the University of Arkansas for Medical Sciences, with whom she has co-authored multiple publications. Theriot's scholarly output to date includes two publications, with a total of eight citations and an h-index of 2.
Metrics
- h-index: 2
- Publications: 2
- Citations: 8
Selected Publications
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SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome (2024)
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Pulmonary Expression of Interleukin-17 Contributes to Neutrophil Infiltration into the Lungs during Pneumonic Plague (2023)
Collaboration Network
Top Collaborators
Priyangi A. Malaviarachchi
University of Arkansas for Medical Sciences
2 shared publications
In database
- Pulmonary Expression of Interleukin-17 Contributes to Neutrophil Infiltration into the Lungs during Pneumonic Plague
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
Roger D. Pechous
University of Arkansas for Medical Sciences (US)
2 shared publications
- Pulmonary Expression of Interleukin-17 Contributes to Neutrophil Infiltration into the Lungs during Pneumonic Plague
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
Madeleine G. Scott
University of Arkansas for Medical Sciences
1 shared publication
In database
- Pulmonary Expression of Interleukin-17 Contributes to Neutrophil Infiltration into the Lungs during Pneumonic Plague
Kenneth T. Appell
University of Arkansas for Medical Sciences
1 shared publication
In database
- Pulmonary Expression of Interleukin-17 Contributes to Neutrophil Infiltration into the Lungs during Pneumonic Plague
Srijon K. Banerjee
University of Pittsburgh
1 shared publication
In database
- Pulmonary Expression of Interleukin-17 Contributes to Neutrophil Infiltration into the Lungs during Pneumonic Plague
Zhuo Xing
Cancer Genetics (United States) (US)
1 shared publication
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
A. R. Douglas
Cancer Genetics (United States) (US)
1 shared publication
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
Samantha D. Crane
University of Arkansas for Medical Sciences
1 shared publication
In database
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
Zijing Zhang
University of Arkansas for Medical Sciences
1 shared publication
In database
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
Alireza Ghaffarieh
University of Arkansas for Medical Sciences (US)
1 shared publication
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
Lu Huang
University of Arkansas for Medical Sciences
1 shared publication
In database
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
Yichao Yu
Cancer Genetics (United States) (US)
1 shared publication
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
Xuming Zhang
University of Arkansas for Medical Sciences (US)
1 shared publication
- SARS-CoV-2 Infection Causes Heightened Disease Severity and Mortality in a Mouse Model of Down Syndrome
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