Matthew Bell Institution-verified
Sourced from institutional research profiles (UAMS TRI or ARA).
Researcher
faculty
Research Areas
Biography and Research Information
OverviewAI-generated summary
Matthew Bell's research focuses on the role of mononuclear myeloid cells in inflammatory conditions, particularly inflammatory bowel disease (IBD) and Crohn's disease. He investigates therapeutic strategies targeting these cells to modulate inflammatory mediators and pathways. His work includes the study of BET protein inhibitors and their effects on specific inflammatory pathways, as well as the exploration of carboxylesterase-1 assisted targeting of HDAC inhibitors.
His recent publications also touch upon the epigenetic regulation of dendritic cell function and potential applications in immunotherapy. Bell has contributed to studies on specific treatment syndromes, including Trofinetide-Induced Enterocolitis Syndrome in children with Rett Syndrome. His research network includes collaborators from the University of Arkansas for Medical Sciences, such as Stephen G. Jones and John Alberty.
Metrics
- h-index: 9
- Publications: 17
- Citations: 801
Selected Publications
-
Trofinetide-Induced Enterocolitis Syndrome in a Child with Rett Syndrome (2025)
Collaboration Network
Top Collaborators
- Carboxylesterase-1 Assisted Targeting of HDAC Inhibitors to Mononuclear Myeloid Cells in Inflammatory Bowel Disease
- A BET Protein Inhibitor Targeting Mononuclear Myeloid Cells Affects Specific Inflammatory Mediators and Pathways in Crohn’s Disease
- The Epigenetic Reader Protein SP140 Regulates Dendritic Cell Activation, Maturation and Tolerogenic Potential
- Mo1130: CHARACTERISING MONONUCLEAR MYELOID CELLS AND PROFILING THE EFFECTS OF BET INHIBITORS IN BIOLOGICS NON-RESPONDING CROHN'S DISEASE PATIENTS
- Carboxylesterase-1 Assisted Targeting of HDAC Inhibitors to Mononuclear Myeloid Cells in Inflammatory Bowel Disease
- A BET Protein Inhibitor Targeting Mononuclear Myeloid Cells Affects Specific Inflammatory Mediators and Pathways in Crohn’s Disease
- Mo1130: CHARACTERISING MONONUCLEAR MYELOID CELLS AND PROFILING THE EFFECTS OF BET INHIBITORS IN BIOLOGICS NON-RESPONDING CROHN'S DISEASE PATIENTS
- Carboxylesterase-1 Assisted Targeting of HDAC Inhibitors to Mononuclear Myeloid Cells in Inflammatory Bowel Disease
- A BET Protein Inhibitor Targeting Mononuclear Myeloid Cells Affects Specific Inflammatory Mediators and Pathways in Crohn’s Disease
- Mo1130: CHARACTERISING MONONUCLEAR MYELOID CELLS AND PROFILING THE EFFECTS OF BET INHIBITORS IN BIOLOGICS NON-RESPONDING CROHN'S DISEASE PATIENTS
- Carboxylesterase-1 Assisted Targeting of HDAC Inhibitors to Mononuclear Myeloid Cells in Inflammatory Bowel Disease
- A BET Protein Inhibitor Targeting Mononuclear Myeloid Cells Affects Specific Inflammatory Mediators and Pathways in Crohn’s Disease
- Mo1130: CHARACTERISING MONONUCLEAR MYELOID CELLS AND PROFILING THE EFFECTS OF BET INHIBITORS IN BIOLOGICS NON-RESPONDING CROHN'S DISEASE PATIENTS
- Carboxylesterase-1 Assisted Targeting of HDAC Inhibitors to Mononuclear Myeloid Cells in Inflammatory Bowel Disease
- A BET Protein Inhibitor Targeting Mononuclear Myeloid Cells Affects Specific Inflammatory Mediators and Pathways in Crohn’s Disease
- The Epigenetic Reader Protein SP140 Regulates Dendritic Cell Activation, Maturation and Tolerogenic Potential
- Carboxylesterase-1 Assisted Targeting of HDAC Inhibitors to Mononuclear Myeloid Cells in Inflammatory Bowel Disease
- A BET Protein Inhibitor Targeting Mononuclear Myeloid Cells Affects Specific Inflammatory Mediators and Pathways in Crohn’s Disease
- Mo1130: CHARACTERISING MONONUCLEAR MYELOID CELLS AND PROFILING THE EFFECTS OF BET INHIBITORS IN BIOLOGICS NON-RESPONDING CROHN'S DISEASE PATIENTS
- Carboxylesterase-1 Assisted Targeting of HDAC Inhibitors to Mononuclear Myeloid Cells in Inflammatory Bowel Disease
- A BET Protein Inhibitor Targeting Mononuclear Myeloid Cells Affects Specific Inflammatory Mediators and Pathways in Crohn’s Disease
- Carboxylesterase-1 Assisted Targeting of HDAC Inhibitors to Mononuclear Myeloid Cells in Inflammatory Bowel Disease
- The Epigenetic Reader Protein SP140 Regulates Dendritic Cell Activation, Maturation and Tolerogenic Potential
- Carboxylesterase-1 Assisted Targeting of HDAC Inhibitors to Mononuclear Myeloid Cells in Inflammatory Bowel Disease
- A BET Protein Inhibitor Targeting Mononuclear Myeloid Cells Affects Specific Inflammatory Mediators and Pathways in Crohn’s Disease
- Carboxylesterase-1 Assisted Targeting of HDAC Inhibitors to Mononuclear Myeloid Cells in Inflammatory Bowel Disease
- Mo1130: CHARACTERISING MONONUCLEAR MYELOID CELLS AND PROFILING THE EFFECTS OF BET INHIBITORS IN BIOLOGICS NON-RESPONDING CROHN'S DISEASE PATIENTS
- Carboxylesterase-1 Assisted Targeting of HDAC Inhibitors to Mononuclear Myeloid Cells in Inflammatory Bowel Disease
- A BET Protein Inhibitor Targeting Mononuclear Myeloid Cells Affects Specific Inflammatory Mediators and Pathways in Crohn’s Disease
- Carboxylesterase-1 Assisted Targeting of HDAC Inhibitors to Mononuclear Myeloid Cells in Inflammatory Bowel Disease
- A BET Protein Inhibitor Targeting Mononuclear Myeloid Cells Affects Specific Inflammatory Mediators and Pathways in Crohn’s Disease
- A BET Protein Inhibitor Targeting Mononuclear Myeloid Cells Affects Specific Inflammatory Mediators and Pathways in Crohn’s Disease
- Mo1130: CHARACTERISING MONONUCLEAR MYELOID CELLS AND PROFILING THE EFFECTS OF BET INHIBITORS IN BIOLOGICS NON-RESPONDING CROHN'S DISEASE PATIENTS
- A BET Protein Inhibitor Targeting Mononuclear Myeloid Cells Affects Specific Inflammatory Mediators and Pathways in Crohn’s Disease
- Mo1130: CHARACTERISING MONONUCLEAR MYELOID CELLS AND PROFILING THE EFFECTS OF BET INHIBITORS IN BIOLOGICS NON-RESPONDING CROHN'S DISEASE PATIENTS
- A BET Protein Inhibitor Targeting Mononuclear Myeloid Cells Affects Specific Inflammatory Mediators and Pathways in Crohn’s Disease
- Mo1130: CHARACTERISING MONONUCLEAR MYELOID CELLS AND PROFILING THE EFFECTS OF BET INHIBITORS IN BIOLOGICS NON-RESPONDING CROHN'S DISEASE PATIENTS
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