Michael Bauer
Sourced from institutional research profiles (UAMS TRI or ARA).
Assitstant Professor
Also affiliated: Western University (1981–2023); Novartis (Switzerland) (2023); Strong Memorial Hospital (2009); University of Arkansas Medical Center (2023); Frauenklinik Heidelberg (1986); Universitäts Frauenklinik (1986); Universitätsklinik für Strahlentherapie (1984); Drees & Sommer (Germany) (2013); Winthrop Rockefeller Foundation (2019–2023); Miami Children's Hospital (2024); University Hospital Ulm (2011); Bassett Healthcare Network (2005); Bassett Medical Center (1999); Cytoskeleton (United States) (2023); University of Rochester (1992–2009); Ludwig-Maximilians-Universität München (1988)
Faculty Researcher
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Michael Bauer's research focuses on the molecular mechanisms and evolutionary pathways of multiple myeloma. His work investigates the progression of the disease from precursor stages to relapse-refractory states, utilizing multi-omics approaches to understand alterations in the immune microenvironment and genomic landscape. Bauer's publications have explored the role of chromothripsis as a pathogenic driver and the impact of structural variants on clinical outcomes in multiple myeloma.
His research group also investigates the relationship between environmental factors and health outcomes, as evidenced by a publication linking low-level environmental heavy metals to obesity in postmenopausal women. Additionally, Bauer has examined genome-wide DNA methylation signatures predictive of doxorubicin-induced cardiotoxicity in breast cancer patients. His scholarship metrics include an h-index of 25, 397 total publications, and 3,381 total citations, designating him as a highly cited researcher.
Metrics
- h-index: 25
- Publications: 397
- Citations: 3,412
Selected Publications
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Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation (2026)
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Folinic Acid Improves Healing of Diabetic Foot Ulcers (2026)
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Proteostasis sustains T cell differentiation potential and tumor-infiltrating lymphocyte function (2026)
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How Does Age at Diagnosis Influence Multiple Myeloma Survival? Empirical Evidence (2025)
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Start Time End Time Integration (STETI): Method for Including Recent Data to Analyze Trends in Kidney Cancer Survival (2025)
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EZH2 loss during metabolic stress drives restoration of MHC class I machinery in melanoma (2025)
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Multi-Omics Reveal Immune Microenvironment Alterations in Multiple Myeloma and Its Precursor Stages (2024)
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Multi-omics reveal immune microenvironment alterations in multiple myeloma and its precursor stages (2024)
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RNA sequencing of formalin fixed paraffin-embedded heart tissue provides transcriptomic information about chemotherapy-induced cardiotoxicity (2024)
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Increased Utilization of Low-Dose CT for Lung Cancer Screening at an Arkansas Community Oncology Clinic (2023)
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1211 Defining the role of PCK2 in T cell metabolic plasticity in Glioblastoma (2023)
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High NEK2 expression in myeloid progenitors suppresses T cell immunity in multiple myeloma (2023)
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EDNRA-Expressing Mesenchymal Cells Are Expanded in Myeloma Interstitial Bone Marrow and Associated with Disease Progression (2023)
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Association of DNA methylation signatures with cognitive performance among smokers and ex-smokers (2023)
Federal Grants 1
Grants & Funding
As listed on this researcher's institutional profile. Federal awards with verified records are shown above.
- Center for Studies of Host Response to Cancer Therapy NIH
Collaboration Network
Top Collaborators
- The molecular make up of smoldering myeloma highlights the evolutionary pathways leading to multiple myeloma
- The spatio-temporal evolution of multiple myeloma from baseline to relapse-refractory states
- Chromothripsis as a pathogenic driver of multiple myeloma
- Multi-omics reveal immune microenvironment alterations in multiple myeloma and its precursor stages
- Structural variants shape the genomic landscape and clinical outcome of multiple myeloma
Showing 5 of 13 shared publications
- The molecular make up of smoldering myeloma highlights the evolutionary pathways leading to multiple myeloma
- The spatio-temporal evolution of multiple myeloma from baseline to relapse-refractory states
- Characterizing the role of the immune microenvironment in multiple myeloma progression at a single-cell level
- Multi-omics reveal immune microenvironment alterations in multiple myeloma and its precursor stages
- High NEK2 expression in myeloid progenitors suppresses T cell immunity in multiple myeloma
Showing 5 of 11 shared publications
- The molecular make up of smoldering myeloma highlights the evolutionary pathways leading to multiple myeloma
- The spatio-temporal evolution of multiple myeloma from baseline to relapse-refractory states
- Characterizing the role of the immune microenvironment in multiple myeloma progression at a single-cell level
- Multi-omics reveal immune microenvironment alterations in multiple myeloma and its precursor stages
- High NEK2 expression in myeloid progenitors suppresses T cell immunity in multiple myeloma
Showing 5 of 11 shared publications
- The molecular make up of smoldering myeloma highlights the evolutionary pathways leading to multiple myeloma
- The spatio-temporal evolution of multiple myeloma from baseline to relapse-refractory states
- Characterizing the role of the immune microenvironment in multiple myeloma progression at a single-cell level
- Chromothripsis as a pathogenic driver of multiple myeloma
- Structural variants shape the genomic landscape and clinical outcome of multiple myeloma
Showing 5 of 11 shared publications
- The molecular make up of smoldering myeloma highlights the evolutionary pathways leading to multiple myeloma
- The spatio-temporal evolution of multiple myeloma from baseline to relapse-refractory states
- Characterizing the role of the immune microenvironment in multiple myeloma progression at a single-cell level
- Chromothripsis as a pathogenic driver of multiple myeloma
- Structural variants shape the genomic landscape and clinical outcome of multiple myeloma
Showing 5 of 11 shared publications
- The molecular make up of smoldering myeloma highlights the evolutionary pathways leading to multiple myeloma
- The spatio-temporal evolution of multiple myeloma from baseline to relapse-refractory states
- Characterizing the role of the immune microenvironment in multiple myeloma progression at a single-cell level
- Multi-omics reveal immune microenvironment alterations in multiple myeloma and its precursor stages
- High NEK2 expression in myeloid progenitors suppresses T cell immunity in multiple myeloma
Showing 5 of 10 shared publications
- The molecular make up of smoldering myeloma highlights the evolutionary pathways leading to multiple myeloma
- The spatio-temporal evolution of multiple myeloma from baseline to relapse-refractory states
- Characterizing the role of the immune microenvironment in multiple myeloma progression at a single-cell level
- Multi-omics reveal immune microenvironment alterations in multiple myeloma and its precursor stages
- High NEK2 expression in myeloid progenitors suppresses T cell immunity in multiple myeloma
Showing 5 of 10 shared publications
- The molecular make up of smoldering myeloma highlights the evolutionary pathways leading to multiple myeloma
- The spatio-temporal evolution of multiple myeloma from baseline to relapse-refractory states
- Chromothripsis as a pathogenic driver of multiple myeloma
- Structural variants shape the genomic landscape and clinical outcome of multiple myeloma
- Insights into high-risk multiple myeloma from an analysis of the role of PHF19 in cancer
Showing 5 of 10 shared publications
- The molecular make up of smoldering myeloma highlights the evolutionary pathways leading to multiple myeloma
- Chromothripsis as a pathogenic driver of multiple myeloma
- Structural variants shape the genomic landscape and clinical outcome of multiple myeloma
- Insights into high-risk multiple myeloma from an analysis of the role of PHF19 in cancer
- High‐risk transcriptional profiles in multiple myeloma are an acquired feature that can occur in any subtype and more frequently with each subsequent relapse
Showing 5 of 8 shared publications
- The molecular make up of smoldering myeloma highlights the evolutionary pathways leading to multiple myeloma
- Structural variants shape the genomic landscape and clinical outcome of multiple myeloma
- FiNGS: high quality somatic mutations using filters for next generation sequencing
- High‐risk transcriptional profiles in multiple myeloma are an acquired feature that can occur in any subtype and more frequently with each subsequent relapse
- Plasma cells expression from smouldering myeloma to myeloma reveals the importance of the PRC2 complex, cell cycle progression, and the divergent evolutionary pathways within the different molecular subgroups
Showing 5 of 6 shared publications
- The molecular make up of smoldering myeloma highlights the evolutionary pathways leading to multiple myeloma
- The spatio-temporal evolution of multiple myeloma from baseline to relapse-refractory states
- Chromothripsis as a pathogenic driver of multiple myeloma
- Structural variants shape the genomic landscape and clinical outcome of multiple myeloma
- Multiomic Mapping of Copy Number and Structural Variation on Chromosome 1 (Chr1) Highlights Multiple Recurrent Disease Drivers
Showing 5 of 6 shared publications
- The molecular make up of smoldering myeloma highlights the evolutionary pathways leading to multiple myeloma
- The spatio-temporal evolution of multiple myeloma from baseline to relapse-refractory states
- Chromothripsis as a pathogenic driver of multiple myeloma
- Structural variants shape the genomic landscape and clinical outcome of multiple myeloma
- Multiomic Mapping of Copy Number and Structural Variation on Chromosome 1 (Chr1) Highlights Multiple Recurrent Disease Drivers
Showing 5 of 6 shared publications
- The molecular make up of smoldering myeloma highlights the evolutionary pathways leading to multiple myeloma
- The spatio-temporal evolution of multiple myeloma from baseline to relapse-refractory states
- High‐risk transcriptional profiles in multiple myeloma are an acquired feature that can occur in any subtype and more frequently with each subsequent relapse
- Plasma cells expression from smouldering myeloma to myeloma reveals the importance of the PRC2 complex, cell cycle progression, and the divergent evolutionary pathways within the different molecular subgroups
- The Spatio-Temporal Evolution of Multiple Myeloma from Baseline to Relapse-Refractory States
- Chromothripsis as a pathogenic driver of multiple myeloma
- Structural variants shape the genomic landscape and clinical outcome of multiple myeloma
- Insights into high-risk multiple myeloma from an analysis of the role of PHF19 in cancer
- Hispanic or Latin American Ancestry Is Associated with a Similar Genomic Profile and a Trend Toward Inferior Outcomes in Newly Diagnosed Multiple Myeloma As Compared to Non-Hispanic White Patients in the Multiple Myeloma Research Foundation (MMRF) CoMMpassstudy
- Multiomic Mapping of Copy Number and Structural Variation on Chromosome 1 (Chr1) Highlights Multiple Recurrent Disease Drivers
- Chromothripsis as a pathogenic driver of multiple myeloma
- Insights into high-risk multiple myeloma from an analysis of the role of PHF19 in cancer
- Plasma cells expression from smouldering myeloma to myeloma reveals the importance of the PRC2 complex, cell cycle progression, and the divergent evolutionary pathways within the different molecular subgroups
- Hispanic or Latin American Ancestry Is Associated with a Similar Genomic Profile and a Trend Toward Inferior Outcomes in Newly Diagnosed Multiple Myeloma As Compared to Non-Hispanic White Patients in the Multiple Myeloma Research Foundation (MMRF) CoMMpassstudy
- Multiomic Mapping of Copy Number and Structural Variation on Chromosome 1 (Chr1) Highlights Multiple Recurrent Disease Drivers
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