Teresita Bellido Institution-verified
Sourced from institutional research profiles (UAMS TRI or ARA).
◆ ARA Academy
Federal Grant PI
High Impact
Musculoskeletal Health and Disease Research - UAMS Creativity Hub Leadership
faculty
75 h-index
318 pubs
22,966 cited
Research Areas
Biography and Research Information
OverviewAI-generated summary
Teresita Bellido's research focuses on bone and musculoskeletal health, with a particular emphasis on the role of osteocytes in bone diseases. Her work investigates the signaling functions of osteocytes and their contribution to conditions such as diabetes-induced bone disease and multiple myeloma. She has received federal funding from the NIH for studies on glucocorticoid-induced atrophy in bone and muscle, and for research into the contribution of osteocytes to the musculoskeletal effects of multiple myeloma. Bellido also leads a research group and is an active member of the ARA Academy, recognized as a high-impact researcher with a significant publication record and citation count.
Her recent publications explore diverse areas within musculoskeletal research. These include the function of osteocytes as signaling cells, the use of decellularized matrices to accelerate bone repair, and the targeting of Notch inhibitors to reduce tumor growth and bone destruction in multiple myeloma. Bellido also investigates the reversal of diabetic bone signatures with anabolic therapies and the specific signaling pathways involved in diabetes-induced bone disease. Her work extends to the clinical applications of bisphosphonates for osteoporosis. Bellido collaborates with several researchers at the University of Arkansas for Medical Sciences, including Jesús Delgado‐Calle, Hayley M. Sabol, Amy Y. Sato, and Nisreen Akel.
Metrics
- h-index: 75
- Publications: 318
- Citations: 22,966
Selected Publications
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Region- and Compartment-Specific Elevation of Bone Mass in Mice Following Tsc1 Deletion in 8-kb Dmp1-Cre-Expressing Cells (2026)
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The development of a collagen-nanoscale hydroxyapatite three-dimensional (3D) in vitro culture system for reproducing osteocyte differentiation and tissue mineralization (2025)
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A novel CCL3-HMGB1 signaling axis regulating osteocyte RANKL expression in multiple myeloma (2024)
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Generation of BT-Amide, a Bone-Targeted Pyk2 Inhibitor, Effective <i>via</i> Oral Administration, for the Prevention of Glucocorticoid-Induced Bone Loss (2024)
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Pharmacologic or genetic interference with atrogene signaling protects against glucocorticoid-induced musculoskeletal and cardiac disease (2024)
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Sclerostin antibody corrects periodontal disease in type 2 diabetic mice (2024)
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Abaloparatide is more potent than teriparatide in restoring bone mass and strength in type 1 diabetic male mice (2024)
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Proceedings of the 2023 Santa Fe Bone Symposium: Progress and Controversies in the Management of Patients with Skeletal Diseases (2023)
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OR29-04 Vitamin D Signaling Prevents Glucocorticoid-Induced Musculoskeletal Tissue Loss And Cardiac Dysfunction By Targeting The Atrogene Pathway (2023)
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THU346 Repairing Skeletal Deterioration In Diabetes With Bone Anabolic Therapies (2023)
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Loss of Nmp4 enhances bone gain from sclerostin antibody administration (2023)
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Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice (2023)
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Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice (2023)
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The <scp>LRP5</scp> high‐bone‐mass mutation causes alveolar bone accrual with minor craniofacial alteration (2023)
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Reversal of the diabetic bone signature with anabolic therapies in mice (2023)
ARA Academy 2018 ARA Scholar
Dr. Bellido is an internationally recognized leader in bone research. She holds major grants from the NIH and Veterans Administration and has served as president of the American Society for Bone and Mineral Research (2020). Her research focuses on signal transduction in bone and muscle, with emphasis on osteocyte biology in health and disease and hormonal action mechanisms in the musculoskeletal system.
Policy Impact
Holds major NIH and VA grants; former president of the American Society for Bone and Mineral Research, bringing national leadership and federal funding to UAMS.
Growth Areas
['Population Health Innovations & Clinical Research']
Federal Grants 4 $805,856 total
NIH/National Institute of Arthritis and Musculoskeletal and Skin Diseases
Contact PI
Jul 2020 - Jun 2024
NIH/National Cancer Institute
Co-PI
Mar 2017 - Jun 2028
Contribution of osteocytes to the musculoskeletal effects of Multiple Myeloma
NSF
PI
Jul 2023 - Jun 2024
Grants & Funding
Collaboration Network
Top Collaborators
Meloney Cregor
Indiana University School of Medicine
28 shared publications
- Targeting Notch Inhibitors to the Myeloma Bone Marrow Niche Decreases Tumor Growth and Bone Destruction without Gut Toxicity
- Reversal of the diabetic bone signature with anabolic therapies in mice
- Notch3 signaling between myeloma cells and osteocytes in the tumor niche promotes tumor growth and bone destruction
- Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice
- Abaloparatide is more potent than teriparatide in restoring bone mass and strength in type 1 diabetic male mice
Showing 5 of 28 shared publications
Jesús Delgado‐Calle
Winthrop Rockefeller Foundation
24 shared publications
In database
- The osteocyte as a signaling cell
- Targeting Notch Inhibitors to the Myeloma Bone Marrow Niche Decreases Tumor Growth and Bone Destruction without Gut Toxicity
- Notch3 signaling between myeloma cells and osteocytes in the tumor niche promotes tumor growth and bone destruction
- A novel CCL3-HMGB1 signaling axis regulating osteocyte RANKL expression in multiple myeloma
- Wnt/β-catenin Signaling Controls Maxillofacial Hyperostosis
Showing 5 of 24 shared publications
Judith L. Anderson
18 shared publications
- Supplementary figure legends from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Supplementary figure 3 from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Supplementary figure 2 from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Supplementary methods from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Data from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
Showing 5 of 18 shared publications
Lilian I. Plotkin
Indiana University School of Medicine
16 shared publications
- Osteocyte-Derived CaMKK2 Regulates Osteoclasts and Bone Mass in a Sex-Dependent Manner through Secreted Calpastatin
- Loss of Nmp4 enhances bone gain from sclerostin antibody administration
- Osteocyte-Derived CaMKK2 Regulates Osteoclasts and Bone Mass in a Sex-Dependent Manner Through Secreted Calpastatin
- Author response for "Conditional Loss of <i>Nmp4</i> in Mesenchymal Stem Progenitor Cells Enhances <scp>PTH‐Induced</scp> Bone Formation"
- Supplementary figure legends from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
Showing 5 of 16 shared publications
G. David Roodman
Indiana University – Purdue University Indianapolis (US)
12 shared publications
- Notch3 signaling between myeloma cells and osteocytes in the tumor niche promotes tumor growth and bone destruction
- A novel CCL3-HMGB1 signaling axis regulating osteocyte RANKL expression in multiple myeloma
- Supplementary figure legends from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Supplementary methods from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Data from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
Showing 5 of 12 shared publications
Masahiro Hiasa
12 shared publications
- Supplementary figure legends from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Supplementary figure 3 from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Supplementary figure 2 from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Supplementary methods from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Data from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
Showing 5 of 12 shared publications
John M. Chirgwin
12 shared publications
- Supplementary figure legends from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Supplementary figure 3 from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Supplementary figure 2 from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Supplementary methods from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Data from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
Showing 5 of 12 shared publications
Nadia Carlesso
12 shared publications
- Supplementary figure legends from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Supplementary figure 3 from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Supplementary figure 2 from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Supplementary methods from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Data from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
Showing 5 of 12 shared publications
Toshiyuki Yoneda
12 shared publications
- Supplementary figure legends from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Supplementary figure 3 from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Supplementary figure 2 from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Supplementary methods from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Data from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
Showing 5 of 12 shared publications
Hayley M. Sabol
University of Arkansas for Medical Sciences
10 shared publications
In database
- Targeting Notch Inhibitors to the Myeloma Bone Marrow Niche Decreases Tumor Growth and Bone Destruction without Gut Toxicity
- Notch3 signaling between myeloma cells and osteocytes in the tumor niche promotes tumor growth and bone destruction
- A novel CCL3-HMGB1 signaling axis regulating osteocyte RANKL expression in multiple myeloma
- Abstract 5672: Notch3 signaling between myeloma cells and osteocytes in the tumor niche promotes tumor growth and bone destruction
- Data from Targeting Notch Inhibitors to the Myeloma Bone Marrow Niche Decreases Tumor Growth and Bone Destruction without Gut Toxicity
Showing 5 of 10 shared publications
G. David Roodman
10 shared publications
- Targeting Notch Inhibitors to the Myeloma Bone Marrow Niche Decreases Tumor Growth and Bone Destruction without Gut Toxicity
- Abstract 5672: Notch3 signaling between myeloma cells and osteocytes in the tumor niche promotes tumor growth and bone destruction
- Supplementary figure 3 from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Supplementary figure 2 from Bidirectional Notch Signaling and Osteocyte-Derived Factors in the Bone Marrow Microenvironment Promote Tumor Cell Proliferation and Bone Destruction in Multiple Myeloma
- Data from Targeting Notch Inhibitors to the Myeloma Bone Marrow Niche Decreases Tumor Growth and Bone Destruction without Gut Toxicity
Showing 5 of 10 shared publications
Tânia Amorim
9 shared publications
- Targeting Notch Inhibitors to the Myeloma Bone Marrow Niche Decreases Tumor Growth and Bone Destruction without Gut Toxicity
- Notch3 signaling between myeloma cells and osteocytes in the tumor niche promotes tumor growth and bone destruction
- Abstract 5672: Notch3 signaling between myeloma cells and osteocytes in the tumor niche promotes tumor growth and bone destruction
- Data from Targeting Notch Inhibitors to the Myeloma Bone Marrow Niche Decreases Tumor Growth and Bone Destruction without Gut Toxicity
- Supplementary Table 1 from Targeting Notch Inhibitors to the Myeloma Bone Marrow Niche Decreases Tumor Growth and Bone Destruction without Gut Toxicity
Showing 5 of 9 shared publications
Kevin McAndrews
Indiana University School of Medicine
9 shared publications
- Targeting Notch Inhibitors to the Myeloma Bone Marrow Niche Decreases Tumor Growth and Bone Destruction without Gut Toxicity
- Abaloparatide is more potent than teriparatide in restoring bone mass and strength in type 1 diabetic male mice
- Pharmacologic or genetic interference with atrogene signaling protects against glucocorticoid-induced musculoskeletal and cardiac disease
- Data from Targeting Notch Inhibitors to the Myeloma Bone Marrow Niche Decreases Tumor Growth and Bone Destruction without Gut Toxicity
- Supplementary Table 1 from Targeting Notch Inhibitors to the Myeloma Bone Marrow Niche Decreases Tumor Growth and Bone Destruction without Gut Toxicity
Showing 5 of 9 shared publications
Noriyoshi Kurihara
9 shared publications
- Targeting Notch Inhibitors to the Myeloma Bone Marrow Niche Decreases Tumor Growth and Bone Destruction without Gut Toxicity
- Notch3 signaling between myeloma cells and osteocytes in the tumor niche promotes tumor growth and bone destruction
- Abstract 5672: Notch3 signaling between myeloma cells and osteocytes in the tumor niche promotes tumor growth and bone destruction
- Data from Targeting Notch Inhibitors to the Myeloma Bone Marrow Niche Decreases Tumor Growth and Bone Destruction without Gut Toxicity
- Supplementary Table 1 from Targeting Notch Inhibitors to the Myeloma Bone Marrow Niche Decreases Tumor Growth and Bone Destruction without Gut Toxicity
Showing 5 of 9 shared publications
Amy Y. Sato
University of Arkansas for Medical Sciences
9 shared publications
In database
- Reversal of the diabetic bone signature with anabolic therapies in mice
- Myogenic tissue nanotransfection improves muscle torque recovery following volumetric muscle loss
- Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice
- Abaloparatide is more potent than teriparatide in restoring bone mass and strength in type 1 diabetic male mice
- Pharmacologic or genetic interference with atrogene signaling protects against glucocorticoid-induced musculoskeletal and cardiac disease
Showing 5 of 9 shared publications
Similar Researchers
Based on overlapping research topics
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