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Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-10-05
A
Amy Y. Sato
Assistant Professor
Also affiliated: Central Arkansas Veterans Healthcare System (2022–2024); Richard L. Roudebush VA Medical Center (2024); John L. McClellan Memorial Veterans Hospital (2023–2024); Indiana University Indianapolis (2014–2024); Indiana University School of Medicine (2014–2024); Indiana University – Purdue University Indianapolis (2016)
Physiology & Cell Biology, College of Medicine
14 h-index
33 pubs
1,213 cited
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Amy Y. Sato investigates the mechanisms underlying bone and muscle health, with a particular focus on the effects of glucocorticoids. Her research has examined how these hormones contribute to bone and muscle atrophy and explored protective strategies against glucocorticoid-induced bone loss. Sato's work also delves into the role of specific signaling pathways and proteins, such as sclerostin and PTHrP, in maintaining bone homeostasis and strength. She has studied how these factors interact with cellular processes like endoplasmic reticulum stress and mechanotransduction to influence bone remodeling and prevent osteoporosis. Her collaborations include extensive work with researchers at the University of Arkansas for Medical Sciences, including Teresita Bellido, Nisreen Akel, Gaston Troncoso, and Betiana Perez. Sato's scholarly output is reflected in her h-index of 14 and over 1,200 citations across 39 publications.
Metrics
- h-index: 14
- Publications: 33
- Citations: 1,213
Positions
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Assistant Professor publications 2020–2026University of Arkansas for Medical Sciences Physiology & Cell Biology, College of Medicine Institutional directory
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University of Arkansas for Medical Sciences publications 2020–2026ORCID
Selected Publications
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Osteocytic Lipocalin-2 regulates bone formation locally through iron-dependent ferroptosis and Wnt suppression (2026)
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Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength (2025)
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Lipocalin-2 Regulates Osteocyte Ferroptosis and Osteocyte-Osteoblast Crosstalk via Wnt Signaling to Control Bone Formation (2025)
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Generation of BT-Amide, a Bone-Targeted Pyk2 Inhibitor, Effective via Oral Administration, for the Prevention of Glucocorticoid-Induced Bone Loss (2024)
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Pharmacologic or genetic interference with atrogene signaling protects against glucocorticoid-induced musculoskeletal and cardiac disease (2024)
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CRISPR activation of Tfeb , a master regulator of autophagy and lysosomal biogenesis, in osteoblast lineage cells increases bone mass and strength (2024)
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Editorial: Pathophysiology of bone and mineral metabolism (2024)
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Abaloparatide is more potent than teriparatide in restoring bone mass and strength in type 1 diabetic male mice (2024)
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OR29-04 Vitamin D Signaling Prevents Glucocorticoid-Induced Musculoskeletal Tissue Loss And Cardiac Dysfunction By Targeting The Atrogene Pathway (2023)
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THU346 Repairing Skeletal Deterioration In Diabetes With Bone Anabolic Therapies (2023)
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Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice (2023)
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Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice (2023)
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Reversal of the diabetic bone signature with anabolic therapies in mice (2023)
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Myogenic tissue nanotransfection improves muscle torque recovery following volumetric muscle loss (2022)
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Skeletal Protection and Promotion of Microbiome Diversity by Dietary Boosting of the Endogenous Antioxidant Response (2020)
Grants & Funding
As listed on this researcher's institutional profile.
- TRI KL2 Scholars award - Sato UAMS Internal Research Awards Principal Investigator
- Center for Musculoskeletal Disease Research (CMDR) NIH/Nat. Inst. of General Medical Sciences Principal Investigator
- Sato ASBMR 2025 Young Investigator Travel Award for Allison Abney American Society for Bone and Mineral Research Principal Investigator
- TRI KL2 Scholars award - Ashby UAMS College of Medicine Principal Investigator
- ASBMR 2025 Young Investigator Travel Grant for Sudip Panday (Sato-Mentor) American Society for Bone and Mineral Research Principal Investigator
- Glucocorticoid-induced Atrophy in Bone and Muscle NIH Co-Investigator
Collaboration Network
Top Collaborators
Teresita Bellido
Central Arkansas Veterans Healthcare System
10 shared publications
In database
- Reversal of the diabetic bone signature with anabolic therapies in mice
- Skeletal Protection and Promotion of Microbiome Diversity by Dietary Boosting of the Endogenous Antioxidant Response
- Myogenic tissue nanotransfection improves muscle torque recovery following volumetric muscle loss
- Abaloparatide is more potent than teriparatide in restoring bone mass and strength in type 1 diabetic male mice
- Generation of BT-Amide, a Bone-Targeted Pyk2 Inhibitor, Effective via Oral Administration, for the Prevention of Glucocorticoid-Induced Bone Loss
Showing 5 of 10 shared publications
Meloney Cregor
Indiana University School of Medicine
8 shared publications
- Reversal of the diabetic bone signature with anabolic therapies in mice
- Skeletal Protection and Promotion of Microbiome Diversity by Dietary Boosting of the Endogenous Antioxidant Response
- Abaloparatide is more potent than teriparatide in restoring bone mass and strength in type 1 diabetic male mice
- Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice
- Pharmacologic or genetic interference with atrogene signaling protects against glucocorticoid-induced musculoskeletal and cardiac disease
Showing 5 of 8 shared publications
Silvia Marino
Central Arkansas Veterans Healthcare System (US)
5 shared publications
- Reversal of the diabetic bone signature with anabolic therapies in mice
- Abaloparatide is more potent than teriparatide in restoring bone mass and strength in type 1 diabetic male mice
- Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice
- Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice
- THU346 Repairing Skeletal Deterioration In Diabetes With Bone Anabolic Therapies
Nisreen Akel
University of Arkansas for Medical Sciences
5 shared publications
In database
- Reversal of the diabetic bone signature with anabolic therapies in mice
- Generation of BT-Amide, a Bone-Targeted Pyk2 Inhibitor, Effective via Oral Administration, for the Prevention of Glucocorticoid-Induced Bone Loss
- Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice
- Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice
- THU346 Repairing Skeletal Deterioration In Diabetes With Bone Anabolic Therapies
Intawat Nookaew
Institute for Musculoskeletal Health
5 shared publications
In database
- Reversal of the diabetic bone signature with anabolic therapies in mice
- Elevation of master autophagy regulator Tfeb in osteoblast lineage cells increases bone mass and strength
- Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice
- Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice
- THU346 Repairing Skeletal Deterioration In Diabetes With Bone Anabolic Therapies
Shenyang Li
University of Arkansas for Medical Sciences (US)
4 shared publications
- Reversal of the diabetic bone signature with anabolic therapies in mice
- Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice
- Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice
- THU346 Repairing Skeletal Deterioration In Diabetes With Bone Anabolic Therapies
Betiana Perez
University of Arkansas for Medical Sciences
4 shared publications
In database
- Reversal of the diabetic bone signature with anabolic therapies in mice
- Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice
- Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice
- THU346 Repairing Skeletal Deterioration In Diabetes With Bone Anabolic Therapies
Gaston Troncoso
University of Arkansas for Medical Sciences
4 shared publications
In database
- Reversal of the diabetic bone signature with anabolic therapies in mice
- Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice
- Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice
- THU346 Repairing Skeletal Deterioration In Diabetes With Bone Anabolic Therapies
Jomeeka Meeks
University of Arkansas for Medical Sciences
4 shared publications
In database
- Reversal of the diabetic bone signature with anabolic therapies in mice
- Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice
- Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice
- THU346 Repairing Skeletal Deterioration In Diabetes With Bone Anabolic Therapies
Kevin McAndrews
Indiana University School of Medicine
3 shared publications
- Skeletal Protection and Promotion of Microbiome Diversity by Dietary Boosting of the Endogenous Antioxidant Response
- Abaloparatide is more potent than teriparatide in restoring bone mass and strength in type 1 diabetic male mice
- Pharmacologic or genetic interference with atrogene signaling protects against glucocorticoid-induced musculoskeletal and cardiac disease
Meghan Jones
University of Arkansas for Medical Sciences (US)
3 shared publications
- Reversal of the diabetic bone signature with anabolic therapies in mice
- Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice
- Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice
Scott Stewart
University of Arkansas for Medical Sciences
2 shared publications
In database
- Reversal of the diabetic bone signature with anabolic therapies in mice
- Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice
Scott A. Stuart
University of Arkansas for Medical Sciences
2 shared publications
In database
- Author Correction: Reversal of the diabetic bone signature with anabolic therapies in mice
- THU346 Repairing Skeletal Deterioration In Diabetes With Bone Anabolic Therapies
Bhawana Adhikari
University of Arkansas for Medical Sciences
2 shared publications
In database
- Pharmacologic or genetic interference with atrogene signaling protects against glucocorticoid-induced musculoskeletal and cardiac disease
- OR29-04 Vitamin D Signaling Prevents Glucocorticoid-Induced Musculoskeletal Tissue Loss And Cardiac Dysfunction By Targeting The Atrogene Pathway
Marjan Boerma
University of Arkansas for Medical Sciences
2 shared publications
In database
- Pharmacologic or genetic interference with atrogene signaling protects against glucocorticoid-induced musculoskeletal and cardiac disease
- OR29-04 Vitamin D Signaling Prevents Glucocorticoid-Induced Musculoskeletal Tissue Loss And Cardiac Dysfunction By Targeting The Atrogene Pathway
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