Biography and Research Information
OverviewAI-generated summary
Tim Wen's research focuses on understanding the molecular mechanisms that regulate gene expression, specifically examining the role of protein complexes in cellular processes. His recent publication investigates the function of FAM60A, identifying it as a regulator of RNA splicing that operates independently of the established Sin3/HDAC complex. This work contributes to the broader understanding of how gene expression is controlled at the post-transcriptional level. Wen collaborates with researchers at the University of Arkansas for Medical Sciences, including Preeti Nagar, Sayem Miah, Mohammad Alinoor Rahman, and Md. Rafikul Islam, as indicated by shared publications.
Metrics
- Publications: 1
Selected Publications
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Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing (2024)
Collaboration Network
Top Collaborators
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
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