David L. Davies
Professor
Also affiliated: Statens Serum Institut (2000); University of North Carolina at Chapel Hill (2000); Tulane University (1977); Louisiana State University (1975–1981); Texas Tech University (1980); St Bartholomew's Hospital (1987); The Royal Melbourne Hospital (1988); East Malling Research (United Kingdom) (1986); Environment and Climate Change Canada (1989–1992); University of British Columbia (2014); University of Liverpool (1989); University of Leicester (1989–2023); University of Auckland (2011); The University of Melbourne (1998–2000); The University of Western Australia (1975); University of Basilicata (2014); University of Derby (2015–2022); University of Arkansas Medical Center (1990); Western General Hospital (1991); Tulane Medical Center (1977); Glasgow Royal Infirmary (1978–1991); Liverpool Hospital (2000); Western Infirmary (1968–2015); University Medical Center New Orleans (1975); Gartnavel General Hospital (1993); Southern General Hospital (1983–1989); Edinburgh Royal Infirmary (2000); University of Surrey (1997); Heriot-Watt University (2001–2021); United States Public Health Service (1977); University of Bristol (1973–1990); Institut National de Recherche pour l'Agriculture, l'Alimentation et l'Environnement (1994); St Mary's Hospital (1965); Royal Marsden Hospital (1978); King's College Hospital (1983); Pratt & Whitney (United States) (1979–1980); Institut National de la Recherche Agronomique (1994); MRC Human Genetics Unit (1991); Belnet (1970); Pathologie végétale (1994); Department of Agriculture (1994); University College London (1980); University of Colorado Anschutz (1983–1986); University of Tennessee at Knoxville (1979); Louisiana State University Health Sciences Center New Orleans (1975–1981); University of Glasgow (1973–2002); Cardiff University (1942); KPMG (United Kingdom) (1995); University of Gothenburg (1988); Medical Research Council (1972–2015); University of Colorado Denver (1983–1986); University of Wales (1942)
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
David Lewis Davies' research has focused on computational studies of chemical reaction mechanisms, particularly those involving transition metal centers and C-H bond activation. His work investigates the interplay between computational methods and experimental findings to elucidate complex chemical processes.
Davies has published extensively on topics including the mechanisms of cyclometalation by palladium acetate and the synergistic roles of computation and experiment in understanding C-H bond activation. His research group also explores the properties and applications of novel solvent systems, such as deep eutectic solvents formed between choline chloride and carboxylic acids, and moisture-stable ionic liquids containing quaternary ammonium salts.
With an h-index of 60 and over 593 publications, Davies is recognized as a highly cited researcher. He collaborates with colleagues at the University of Arkansas for Medical Sciences, including Noor Akhter, Mohsin Syed, Tiffany Huitt, and Erica Malone.
Metrics
- h-index: 60
- Publications: 568
- Citations: 33,132
Positions
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Professor publications 1990–2025University of Arkansas for Medical Sciences Institution web page
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University of Leicester 1985–2019Chemistry ORCID
Selected Publications
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Vascular Pathology: A Multifocal Cadaveric Analysis of Arterial Aneurysms (2025)
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Unilateral Absence of the Psoas Major and Iliacus Muscles and Downstream Effects of Surgical Intervention in a 96 year-old Female Donor (2025)
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Essential anatomy for core clerkships: A clinical perspective (2023)
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Retroaortic Left Renal Vein: An Incidental Finding During Cadaver Dissection (2022)
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Anatomy Scholars Program for Medical Students Entering a Surgical Residency (2021)
Grants & Funding
As listed on this researcher's institutional profile.
- ETHANOL EFFECTS ON GLIAL RESPONSES TO CNS TISSUE INSULT NIH
- Medical Student Ed grant for Arkansas - Continuation - Revision Health Resources & Services Administration
- ETHANOL AND ACETALDEHYDE EFFECTS ON ASTROCYTES IN CULTUR NIH
Collaboration Network
Top Collaborators
- Parenterally administered 3-nitropropionic acid and amphetamine can combine to produce damage to terminals and cell bodies in the striatum
- Biochemical and morphological effects of fumonisin B1 on primary cultures of rat cerebrum
- Proliferation of astroglia from the adult human cerebrum is inhibited by ethanol in vitro
- Heterogeneity of astroglia cultured from adult human temporal lobe
- Parenterally administered 3-nitropropionic acid and amphetamine can combine to produce damage to terminals and cell bodies in the striatum
- Changes in mRNA Levels for Heat‐Shock/Stress Proteins (Hsp) and a Secretory Vesicle Associated Cysteine‐String Protein (Csp1) after Amphetamine (AMPH) Exposure
- Heterogeneity of astroglia cultured from adult human temporal lobe
- Retroaortic Left Renal Vein: An Incidental Finding During Cadaver Dissection
- Retroaortic Left Renal Vein: An Incidental Finding During Cadaver Dissection
- Vascular Pathology: A Multifocal Cadaveric Analysis of Arterial Aneurysms
- Retroaortic Left Renal Vein: An Incidental Finding During Cadaver Dissection
- Vascular Pathology: A Multifocal Cadaveric Analysis of Arterial Aneurysms
- Unilateral Absence of the Psoas Major and Iliacus Muscles and Downstream Effects of Surgical Intervention in a 96 year-old Female Donor
- Vascular Pathology: A Multifocal Cadaveric Analysis of Arterial Aneurysms
- Unilateral Absence of the Psoas Major and Iliacus Muscles and Downstream Effects of Surgical Intervention in a 96 year-old Female Donor
- Vascular Pathology: A Multifocal Cadaveric Analysis of Arterial Aneurysms
- Long-term ethanol-exposure markedly changes the cellular composition of cerebral glial cultures
- Biochemical and morphological effects of fumonisin B1 on primary cultures of rat cerebrum
- Proliferation of astroglia from the adult human cerebrum is inhibited by ethanol in vitro
- Proliferation of astroglia from the adult human cerebrum is inhibited by ethanol in vitro
- Glial development in primary cultures established from normal and X‐irradiated neonatal spinal cord
- Glial development in primary cultures established from normal and X‐irradiated neonatal spinal cord
- Parenterally administered 3-nitropropionic acid and amphetamine can combine to produce damage to terminals and cell bodies in the striatum
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