Noor Akhter
Sourced from institutional research profiles (UAMS TRI or ARA).
Associate Professor
Also affiliated: Nagoya University (1993)
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Noor Akhter's research focuses on the metabolic signaling pathways that regulate pituitary cell differentiation, with a particular emphasis on the role of leptin. Her work has investigated leptin as a metabolic signal influencing somatotroph differentiation and explored sex-specific responses to diet under thermoneutral conditions, using single-cell transcriptomics to identify reduced pituitary plasticity. Akhter also contributes to research on the impacts of the COVID-19 pandemic on medical students, examining academic performance, stress levels, and the effectiveness of online learning environments. Additionally, her publications include studies on vascular pathology, such as retroaortic left renal veins and arterial aneurysms, often utilizing cadaveric analysis. Akhter is an Associate Professor at the University of Arkansas for Medical Sciences and collaborates with several researchers within the institution, including Mohsin Syed, Kevin D. Phelan, Tiffany Huitt, and Gwen V. Childs.
Metrics
- h-index: 12
- Publications: 43
- Citations: 466
Positions
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Associate Professor publications 2004–2025University of Arkansas for Medical Sciences Neurobiology & Developmental Sciences Institutional directory
Selected Publications
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Vascular Pathology: A Multifocal Cadaveric Analysis of Arterial Aneurysms (2025)
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THU043 Single-cell Transcriptomics Show Sex-specific Responses To A High Fat Diet, Under Thermoneutral Conditions, Pointing To Reduced Pituitary Plasticity (2023)
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Bridging the Technology Divide in the COVID-19 Era: Using Virtual Outreach to Expose Middle and High School Students to Imaging Technology (2022)
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Bridging the Technology Divide in the COVID-19 Era: Using Virtual Outreach to Expose Middle and High School Students to Imaging Technology (2022)
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Retroaortic Left Renal Vein: An Incidental Finding During Cadaver Dissection (2022)
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Leptin: A Metabolic Signal for the Differentiation of Pituitary Cells (2021)
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Persistence and Academic Performance of Medical Students in Online Learning Environment During the COVID-19 Pandemic Lockdown (2021)
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The Effects of COVID‐19 Pandemic Lockdown on Medical Students’ Academic Performance and Stress Levels Based on their Gender and Age (2021)
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Right to Left Crossed Fused Renal Ectopia: An Incidental Finding in Cadaveric Dissection (2019)
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Leptin Regulation of Gonadotrope Gonadotropin-Releasing Hormone Receptors As a Metabolic Checkpoint and Gateway to Reproductive Competence (2018)
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Complete Bilateral Ureteral Duplication: an Incidental Finding in Cadaveric Dissection (2017)
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Association of Gnrhr mRNA With the Stem Cell Determinant Musashi: A Mechanism for Leptin-Mediated Modulation of GnRHR Expression (2017)
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Purified Somatotrope Populations Include Cells with Multihormonal Potential That Produce GH, TSH and Prolactin in Response to Leptin Signals (2016)
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A Sex-Dependent, Tropic Role for Leptin in the Somatotrope as a Regulator of POU1F1 and POU1F1-Dependent Hormones (2016)
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Adipocyte Versus Somatotrope Leptin: Regulation of Metabolic Functions in the Mouse (2016)
Grants & Funding
As listed on this researcher's institutional profile.
- The Significance of Leptin Signals to Neonatal Somatotropes and Gonadotropes NIH
- ArkanSONO: A Technology Based Outreach Exposure Program for High School Students NIH
- Center for Translational Neuroscience NIH
- Cellular Basis for Non-Parallel Gonadotropin Release NIH
Collaboration Network
Top Collaborators
- Leptin Regulation of Gonadotrope Gonadotropin-Releasing Hormone Receptors As a Metabolic Checkpoint and Gateway to Reproductive Competence
- The Somatotrope as a Metabolic Sensor: Deletion of Leptin Receptors Causes Obesity
- Selective Deletion of Leptin Receptors in Gonadotropes Reveals Activin and GnRH-Binding Sites as Leptin Targets in Support of Fertility
- Association of Gnrhr mRNA With the Stem Cell Determinant Musashi: A Mechanism for Leptin-Mediated Modulation of GnRHR Expression
- Ablation of Leptin Signaling to Somatotropes: Changes in Metabolic Factors that Cause Obesity
Showing 5 of 23 shared publications
- Leptin Regulation of Gonadotrope Gonadotropin-Releasing Hormone Receptors As a Metabolic Checkpoint and Gateway to Reproductive Competence
- The Somatotrope as a Metabolic Sensor: Deletion of Leptin Receptors Causes Obesity
- Selective Deletion of Leptin Receptors in Gonadotropes Reveals Activin and GnRH-Binding Sites as Leptin Targets in Support of Fertility
- Adipocyte Versus Pituitary Leptin in the Regulation of Pituitary Hormones: Somatotropes Develop Normally in the Absence of Circulating Leptin
- Bipotential Effects of Estrogen on Growth Hormone Synthesis and Storage<i>in Vitro</i>
Showing 5 of 19 shared publications
- Leptin Regulation of Gonadotrope Gonadotropin-Releasing Hormone Receptors As a Metabolic Checkpoint and Gateway to Reproductive Competence
- The Somatotrope as a Metabolic Sensor: Deletion of Leptin Receptors Causes Obesity
- Selective Deletion of Leptin Receptors in Gonadotropes Reveals Activin and GnRH-Binding Sites as Leptin Targets in Support of Fertility
- Adipocyte Versus Pituitary Leptin in the Regulation of Pituitary Hormones: Somatotropes Develop Normally in the Absence of Circulating Leptin
- Association of Gnrhr mRNA With the Stem Cell Determinant Musashi: A Mechanism for Leptin-Mediated Modulation of GnRHR Expression
Showing 5 of 13 shared publications
- The Somatotrope as a Metabolic Sensor: Deletion of Leptin Receptors Causes Obesity
- Selective Deletion of Leptin Receptors in Gonadotropes Reveals Activin and GnRH-Binding Sites as Leptin Targets in Support of Fertility
- Adipocyte Versus Pituitary Leptin in the Regulation of Pituitary Hormones: Somatotropes Develop Normally in the Absence of Circulating Leptin
- Association of Gnrhr mRNA With the Stem Cell Determinant Musashi: A Mechanism for Leptin-Mediated Modulation of GnRHR Expression
- Ablation of Leptin Signaling to Somatotropes: Changes in Metabolic Factors that Cause Obesity
Showing 5 of 10 shared publications
- Leptin Regulation of Gonadotrope Gonadotropin-Releasing Hormone Receptors As a Metabolic Checkpoint and Gateway to Reproductive Competence
- Selective Deletion of Leptin Receptors in Gonadotropes Reveals Activin and GnRH-Binding Sites as Leptin Targets in Support of Fertility
- Adipocyte Versus Pituitary Leptin in the Regulation of Pituitary Hormones: Somatotropes Develop Normally in the Absence of Circulating Leptin
- Association of Gnrhr mRNA With the Stem Cell Determinant Musashi: A Mechanism for Leptin-Mediated Modulation of GnRHR Expression
- Ablation of Leptin Signaling to Somatotropes: Changes in Metabolic Factors that Cause Obesity
Showing 5 of 10 shared publications
- Leptin Regulation of Gonadotrope Gonadotropin-Releasing Hormone Receptors As a Metabolic Checkpoint and Gateway to Reproductive Competence
- The Somatotrope as a Metabolic Sensor: Deletion of Leptin Receptors Causes Obesity
- Selective Deletion of Leptin Receptors in Gonadotropes Reveals Activin and GnRH-Binding Sites as Leptin Targets in Support of Fertility
- Association of Gnrhr mRNA With the Stem Cell Determinant Musashi: A Mechanism for Leptin-Mediated Modulation of GnRHR Expression
- Medical gross anatomy: conversion of a traditional discipline‐based course to a briefer and more clinically focused foundational module in a revamped preclinical curriculum (532.1)
Showing 5 of 7 shared publications
- Medical gross anatomy: conversion of a traditional discipline‐based course to a briefer and more clinically focused foundational module in a revamped preclinical curriculum (532.1)
- Retroaortic Left Renal Vein: An Incidental Finding During Cadaver Dissection
- Bridging the Technology Divide in the COVID-19 Era: Using Virtual Outreach to Expose Middle and High School Students to Imaging Technology
- Multiple changes in teaching procedures increase student engagement and success in gross anatomy during revision of a medical curriculum
- Right to Left Crossed Fused Renal Ectopia: An Incidental Finding in Cadaveric Dissection
Showing 5 of 7 shared publications
- Medical gross anatomy: conversion of a traditional discipline‐based course to a briefer and more clinically focused foundational module in a revamped preclinical curriculum (532.1)
- Retroaortic Left Renal Vein: An Incidental Finding During Cadaver Dissection
- Methods used in the gross anatomy lab to facilitate learning by first‐year students in a new Physician Assistant Program (722.1)
- Multiple changes in teaching procedures increase student engagement and success in gross anatomy during revision of a medical curriculum
- Right to Left Crossed Fused Renal Ectopia: An Incidental Finding in Cadaveric Dissection
Showing 5 of 6 shared publications
- Leptin Regulation of Gonadotrope Gonadotropin-Releasing Hormone Receptors As a Metabolic Checkpoint and Gateway to Reproductive Competence
- Association of Gnrhr mRNA With the Stem Cell Determinant Musashi: A Mechanism for Leptin-Mediated Modulation of GnRHR Expression
- A Sex-Dependent, Tropic Role for Leptin in the Somatotrope as a Regulator of POU1F1 and POU1F1-Dependent Hormones
- Leptin: A Metabolic Signal for the Differentiation of Pituitary Cells
- THU043 Single-cell Transcriptomics Show Sex-specific Responses To A High Fat Diet, Under Thermoneutral Conditions, Pointing To Reduced Pituitary Plasticity
Showing 5 of 6 shared publications
- Leptin Regulation of Gonadotrope Gonadotropin-Releasing Hormone Receptors As a Metabolic Checkpoint and Gateway to Reproductive Competence
- Association of Gnrhr mRNA With the Stem Cell Determinant Musashi: A Mechanism for Leptin-Mediated Modulation of GnRHR Expression
- A Sex-Dependent, Tropic Role for Leptin in the Somatotrope as a Regulator of POU1F1 and POU1F1-Dependent Hormones
- Leptin: A Metabolic Signal for the Differentiation of Pituitary Cells
- THU043 Single-cell Transcriptomics Show Sex-specific Responses To A High Fat Diet, Under Thermoneutral Conditions, Pointing To Reduced Pituitary Plasticity
Showing 5 of 6 shared publications
- The Somatotrope as a Metabolic Sensor: Deletion of Leptin Receptors Causes Obesity
- Selective Deletion of Leptin Receptors in Gonadotropes Reveals Activin and GnRH-Binding Sites as Leptin Targets in Support of Fertility
- Ablation of Leptin Signaling to Somatotropes: Changes in Metabolic Factors that Cause Obesity
- Ghrelin Restoration of Function In Vitro in Somatotropes from Male Mice Lacking the Janus Kinase (JAK)-Binding Site of the Leptin Receptor
- Functional defects in mice somatotropes caused by ablation of leptin receptors is reversed, in vitro, by stimulation with Ghrelin
- The Somatotrope as a Metabolic Sensor: Deletion of Leptin Receptors Causes Obesity
- Anterior Pituitary Leptin Expression Changes in Different Reproductive States: In Vitro Stimulation by Gonadotropin-releasing Hormone
- Fasting and Glucose Effects on Pituitary Leptin Expression
- Ghrelin Restoration of Function In Vitro in Somatotropes from Male Mice Lacking the Janus Kinase (JAK)-Binding Site of the Leptin Receptor
- Functional defects in mice somatotropes caused by ablation of leptin receptors is reversed, in vitro, by stimulation with Ghrelin
- Bipotential Effects of Estrogen on Growth Hormone Synthesis and Storage<i>in Vitro</i>
- Anterior Pituitary Leptin Expression Changes in Different Reproductive States: In Vitro Stimulation by Gonadotropin-releasing Hormone
- Fasting and Glucose Effects on Pituitary Leptin Expression
- Estrogen mediated cross talk between the ovary and pituitary somatotrope
- The Effects of COVID‐19 Pandemic Lockdown on Medical Students’ Academic Performance and Stress Levels Based on their Gender and Age
- Persistence and Academic Performance of Medical Students in Online Learning Environment During the COVID-19 Pandemic Lockdown
- Medical gross anatomy: conversion of a traditional discipline‐based course to a briefer and more clinically focused foundational module in a revamped preclinical curriculum (532.1)
- Anterior Pituitary Leptin Expression Changes in Different Reproductive States: In Vitro Stimulation by Gonadotropin-releasing Hormone
- Fasting and Glucose Effects on Pituitary Leptin Expression
- Estrogen mediated cross talk between the ovary and pituitary somatotrope
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