Brent J.F. Hill
This is a likely match — the affiliation was inferred from OpenAlex, ORCID, and web sources but has not been fully confirmed. Treat with appropriate caution.
Researcher
Also affiliated: Iowa State University (1997); East Carolina University (2000); Conway School of Landscape Design (2006–2019); University of Missouri (1998–2003)
Faculty Researcher
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Brent J.F. Hill's scholarly work primarily focuses on the field of education, with a specific emphasis on higher education research and the implementation of innovative teaching and learning methods. His recent publications, dating up to 2019, indicate an engagement with pedagogical strategies and their impact within academic settings. While his most recent activity is noted as inactive, his past contributions highlight a dedication to advancing educational practices and understanding within the university context.
Metrics
- h-index: 12
- Publications: 33
- Citations: 394
Selected Publications
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Analysis of leg bones of rats exposed to simulated microgravity and space radiation (2019)
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17<i>β</i>‐estradiol reduces Ca<sub>v</sub>1.2 channel abundance and attenuates Ca<sup>2+</sup>‐dependent contractions in coronary arteries (2017)
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Using three-point bending to evaluate tibia bone strength in ovariectomized young mice (2017)
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Estrogen Induces Coronary Arterial Dilation via Downregulation of Voltage‐gated, Ca <sup>2+</sup> Channels (2015)
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Oestrogen upregulates the sarcoplasmic reticulum Ca<sup>2+</sup><scp>ATP</scp>ase pump in coronary arteries (2014)
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The sarcoplasmic reticulum enhances voltage‐gated Ca <sup>2+</sup> influx in resistance arteries from ovariectomized mice (680.19) (2014)
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Short‐term estrogen depletion does not promote hypertension in mice (2013)
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In vivo loss of estrogen increases voltage‐gated calcium channel function in mesenteric arterial smooth muscle cells (2013)
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Activation of the ERα and ERβ pathway downregulates voltage‐gated Ca <sup>2+</sup> channels in coronary arteries (2012)
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The estrogen metabolite, 2‐MeOH, induces arterial dilation by increasing BK channel expression (2011)
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Regulation of voltage‐gated calcium channel expression by estrogen (2010)
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Nongenomic inhibition of coronary constriction by 17β-estradiol, 2-hydroxyestradiol, and 2-methoxyestradiol (2010)
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An estrogen metabolite, 2‐methoxyestradiol, promotes coronary artery dilation and inhibits breast cancer proliferation (2009)
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An estrogen‐induced decrease in voltage‐gated calcium channel expression attenuates coronary artery contractility (2008)
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The estrogen metabolite, 2‐methoxyestradiol, attenuates coronary arterial tone by inhibiting the influx of calcium (2007)
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