Desislava A. Marinkova
Researcher
Also affiliated: University of Kentucky (2022–2023)
Graduate Student Researcher
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Desislava A. Marinkova's research investigates the role of the TRPA1 cation channel in regulating cochlear sensitivity, particularly following acoustic trauma. Her work has explored how TRPA1 activation in non-sensory supporting cells contributes to these regulatory processes. Marinkova has also examined the relationship between obesity, histone acetylation, and insulin resistance.
Her scholarly contributions include two publications, accumulating 18 citations, and an h-index of 1. Marinkova has collaborated with Rushita A. Bagchi and Ashim K. Bagchi from the University of Arkansas for Medical Sciences, as well as Chris Presley from Hendrix College, with each collaborator sharing one publication with Marinkova.
Metrics
- h-index: 1
- Publications: 2
- Citations: 18
Selected Publications
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Obesity, histone acetylation, and insulin resistance (2024)
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TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma (2023)
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TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma (2022)
Collaboration Network
Top Collaborators
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- TRPA1 activation in non-sensory supporting cells contributes to regulation of cochlear sensitivity after acoustic trauma
- Obesity, histone acetylation, and insulin resistance
- Obesity, histone acetylation, and insulin resistance
- Obesity, histone acetylation, and insulin resistance
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