Match tier
Confirmed
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-10-05
J
Joanna L. Fiddler
Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.
Assistant Professor
Also affiliated: Oklahoma State University (2014–2021); Cornell University (2018–2024); Clemson University (2023–2025)
7 h-index
31 pubs
121 cited
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Joanna L. Fiddler investigates the role of mitochondria in cellular function and disease. Her research has explored how deficiencies in specific enzymes, such as those involved in folate metabolism (e.g., SHMT2, methionine synthase), impact mitochondrial respiration and lead to uracil accumulation in mitochondrial DNA. Fiddler's work also examines the effects of environmental factors and substances on cellular health, including the cardiac toxicity induced by selective serotonin reuptake inhibitors through mitochondrial and sarcomere dysfunction. Additionally, her research has touched upon nutritional interventions, such as zinc supplementation and vitamin B12 and folic acid status, and their influence on cellular processes and genetic material. Fiddler has published 32 papers with 122 citations and an h-index of 7. She has collaborated with Abigail L. Zirbel and Makenzie A. Tharpe at the University of Arkansas at Fayetteville.
Metrics
- h-index: 7
- Publications: 31
- Citations: 121
Positions
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Assistant Professor 2024–presentUniversity of Arkansas at Fayetteville Human Environmental Sciences ORCID
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Assistant Professor 2023–2024Clemson University Food, Nutrition, and Packaging Sciences ORCID
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Postdoctoral Research Associate 2019–2022Cornell University Division of Nutritional Sciences ORCID
Selected Publications
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Transient MYC Mimicking the Exercise Response Orchestrates Multifaceted Skeletal Muscle Adaptations (2026)
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Perfluorooctane sulfonic acid (PFOS) perturbs skeletal muscle oxidative phosphorylation by a different mechanism than liver (2025)
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Loss of SHMT2 and Folate Deficiency Impair Protein Levels Involved in Oxidative Phosphorylation, Folate Transport, and Mitochondrial Dynamics in Mouse C2C12 Myoblast Cells (2025)
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Marginal Iron Depletion Impairs Mitochondrial Complex I Activity and Reduces NDUFB8 and ISCU Protein Levels in Mouse C2C12 Myoblast Cells (2025)
Collaboration Network
Top Collaborators
Abigail L Zirbel
University of Arkansas at Fayetteville
2 shared publications
In database
- Marginal Iron Depletion Impairs Mitochondrial Complex I Activity and Reduces NDUFB8 and ISCU Protein Levels in Mouse C2C12 Myoblast Cells
- Loss of SHMT2 and Folate Deficiency Impair Protein Levels Involved in Oxidative Phosphorylation, Folate Transport, and Mitochondrial Dynamics in Mouse C2C12 Myoblast Cells
Makenzie A Tharpe
University of Arkansas at Fayetteville
2 shared publications
In database
- Marginal Iron Depletion Impairs Mitochondrial Complex I Activity and Reduces NDUFB8 and ISCU Protein Levels in Mouse C2C12 Myoblast Cells
- Loss of SHMT2 and Folate Deficiency Impair Protein Levels Involved in Oxidative Phosphorylation, Folate Transport, and Mitochondrial Dynamics in Mouse C2C12 Myoblast Cells
Aidan J Sievers
Clemson University (US)
1 shared publication
- Marginal Iron Depletion Impairs Mitochondrial Complex I Activity and Reduces NDUFB8 and ISCU Protein Levels in Mouse C2C12 Myoblast Cells
Jazmine A. Eccles-Miller
Clemson University (US)
1 shared publication
- Perfluorooctane sulfonic acid (PFOS) perturbs skeletal muscle oxidative phosphorylation by a different mechanism than liver
Lanie Williams
Clemson University (US)
1 shared publication
- Perfluorooctane sulfonic acid (PFOS) perturbs skeletal muscle oxidative phosphorylation by a different mechanism than liver
Elizabeth Leonard
Clemson University (US)
1 shared publication
- Perfluorooctane sulfonic acid (PFOS) perturbs skeletal muscle oxidative phosphorylation by a different mechanism than liver
Nishanth Tharayil
Clemson University (US)
1 shared publication
- Perfluorooctane sulfonic acid (PFOS) perturbs skeletal muscle oxidative phosphorylation by a different mechanism than liver
Elliet L. Buslee
Clemson University (US)
1 shared publication
- Perfluorooctane sulfonic acid (PFOS) perturbs skeletal muscle oxidative phosphorylation by a different mechanism than liver
William S. Baldwin
Clemson University (US)
1 shared publication
- Perfluorooctane sulfonic acid (PFOS) perturbs skeletal muscle oxidative phosphorylation by a different mechanism than liver
Ronald G. Jones
University of Arkansas at Fayetteville
1 shared publication
In database
- Transient MYC Mimicking the Exercise Response Orchestrates Multifaceted Skeletal Muscle Adaptations
Pieter J. Koopmans
University of Arkansas at Fayetteville
1 shared publication
In database
- Transient MYC Mimicking the Exercise Response Orchestrates Multifaceted Skeletal Muscle Adaptations
Nathan Serrano
University of Arkansas at Fayetteville
1 shared publication
In database
- Transient MYC Mimicking the Exercise Response Orchestrates Multifaceted Skeletal Muscle Adaptations
Ana Regina Cabreara
University of Arkansas at Fayetteville
1 shared publication
In database
- Transient MYC Mimicking the Exercise Response Orchestrates Multifaceted Skeletal Muscle Adaptations
Francielly Morena
University of Florida
1 shared publication
In database
- Transient MYC Mimicking the Exercise Response Orchestrates Multifaceted Skeletal Muscle Adaptations
Toby L. Chambers
University of Arkansas at Fayetteville
1 shared publication
In database
- Transient MYC Mimicking the Exercise Response Orchestrates Multifaceted Skeletal Muscle Adaptations
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