Mojnu Miah
Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.
Graduate Assistant
Also affiliated: International Centre for Diarrhoeal Disease Research (1993–2024); University of Arkansas Medical Center (2025)
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Mojnu Miah's research has focused on microbial infections and disease transmission, with a significant emphasis on Vibrio cholerae and SARS-CoV-2. His work includes the detection of non-culturable Vibrio cholerae O1 associated with cyanobacteria in aquatic environments in Bangladesh and the isolation of Vibrio cholerae O139 from similar settings, exploring implications for disease transmission. More recently, Miah has investigated the evaluation of isothermal amplification assays for point-of-need detection of SARS-CoV-2 in resource-limited settings and culture-independent workflows for sequencing influenza A virus. He has also studied patterns and risk factors of avian influenza virus infection in pigeons and quail in Bangladesh, and contributed to vaccine design targeting hemagglutinin protein of H5 influenza viruses. Miah's scholarship metrics include an h-index of 12, with 41 publications and 610 citations.
Metrics
- h-index: 12
- Publications: 31
- Citations: 596
Positions
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Graduate Assistant 2025–presentUniversity of Arkansas for Medical Sciences Biochemistry and Molecular Biology ORCID
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Research Assistant I 2023–2025University of Arkansas for Medical Sciences Biochemistry and Molecular Biology ORCID
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Research Technologist 2023University of Arkansas for Medical Sciences Biochemistry and Molecular Biology ORCID
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Research Officer 2022–2023International Centre for Diarrhoeal Disease Research One Health Laboratory ORCID
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Research officer 2018–2022International Centre for Diarrhoeal Disease Research, Bangladesh Virology Laboratory ORCID
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Senior Research Assistant 2017–2018International Centre for Diarrhoeal Disease Research, Bangladesh Virology Laboratory ORCID
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Elective Fellow (Intern) 2016–2017International Centre for Diarrhoeal Disease Research Virology Laboratory ORCID
Selected Publications
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Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation (2026)
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DNA secondary structures in <i>BCL2</i> and <i>MYC</i> elicit activation-induced cytidine deaminase binding and activity (2026)
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Low detection of H5N1 virus in commercial chickens with a low-level of vaccination coverage against H5N1 virus infection in Bangladesh (2024)
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Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing (2024)
Collaboration Network
Top Collaborators
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
- Beyond the Sin3/HDAC Complex: FAM60A emerges as a regulator of RNA Splicing
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