Match tier
Listed
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-08-08
F
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Farhana Taher Sumya's research focuses on the molecular mechanisms underlying protein transport and glycosylation within the Golgi apparatus. Her work investigates the COG complex, a multi-subunit tethering factor crucial for intra-Golgi vesicle trafficking. Sumya has studied the impact of COG complex inactivation on Golgi function and the nature of intra-Golgi recycling vesicles. Her research also explores congenital disorders of glycosylation (CDG), specifically CDG-II diseases, and has involved the development and characterization of cellular models for these conditions. She has published on the role of the COG complex in glycosylation and has investigated the effects of mutations in COG subunits, such as COG3, on vesicular trafficking and disease pathogenesis. Sumya's publications also include proteomic characterization of intra-Golgi trafficking intermediates and methods for rapid COG depletion in mammalian cells. She has a history of collaboration with researchers at the University of Arkansas for Medical Sciences, including Vladimir Lupashin and Irina D. Pokrovskaya.
Metrics
- h-index: 5
- Publications: 10
- Citations: 83
Selected Publications
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Golgi CATCHR complexes function as organizing hubs for vesicle tethering and fusion (2026)
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COG Complex in Golgi Trafficking and Glycosylation (2026)
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Deep proteomic profiling of the intra-Golgi trafficking intermediates (2025)
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Comprehensive Proteomic Characterization of the Intra-Golgi Trafficking Intermediates (2024)
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Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking (2023)
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Rapid COG Depletion in Mammalian Cell by Auxin-Inducible Degradation System (2022)
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Acute COG complex inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra‐Golgi recycling vesicles (2022)
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Acute COG inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra-Golgi recycling vesicles (2022)
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Getting Sugar Coating Right! The Role of the Golgi Trafficking Machinery in Glycosylation (2021)
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Development and Initial Characterization of Cellular Models for COG Complex-Related CDG-II Diseases (2021)
Collaboration Network
Top Collaborators
Vladimir Lupashin
University of Arkansas for Medical Sciences
8 shared publications
In database
- Getting Sugar Coating Right! The Role of the Golgi Trafficking Machinery in Glycosylation
- Development and Initial Characterization of Cellular Models for COG Complex-Related CDG-II Diseases
- Acute COG complex inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra‐Golgi recycling vesicles
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
- Rapid COG Depletion in Mammalian Cell by Auxin-Inducible Degradation System
Showing 5 of 8 shared publications
Irina D. Pokrovskaya
University of Arkansas for Medical Sciences
4 shared publications
In database
- Development and Initial Characterization of Cellular Models for COG Complex-Related CDG-II Diseases
- Acute COG complex inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra‐Golgi recycling vesicles
- Rapid COG Depletion in Mammalian Cell by Auxin-Inducible Degradation System
- Acute COG inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra-Golgi recycling vesicles
Zinia D’Souza
University of Arkansas for Medical Sciences
3 shared publications
In database
- Getting Sugar Coating Right! The Role of the Golgi Trafficking Machinery in Glycosylation
- Acute COG complex inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra‐Golgi recycling vesicles
- Acute COG inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra-Golgi recycling vesicles
Walter S. Aragon‐Ramirez
University of Arkansas for Medical Sciences
2 shared publications
In database
- Comprehensive Proteomic Characterization of the Intra-Golgi Trafficking Intermediates
- Deep proteomic profiling of the intra-Golgi trafficking intermediates
Amrita Khakurel
University of Arkansas for Medical Sciences
1 shared publication
In database
- Getting Sugar Coating Right! The Role of the Golgi Trafficking Machinery in Glycosylation
Ruizhi Duan
Baylor College of Medicine (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Dana Marafi
Baylor College of Medicine (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Zhi‐Jie Xia
Sanford Burnham Prebys Medical Discovery Institute (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Bobby G. Ng
Sanford Burnham Prebys Medical Discovery Institute (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Reza Maroofian
Queen Mary University of London (GB)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Ahmed K. Saad
Baylor College of Medicine (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Haowei Du
Baylor College of Medicine (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Jawid M. Fatih
Baylor College of Medicine (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Jill V. Hunter
Baylor College of Medicine (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Hasnaa M. Elbendary
National Human Genome Research Institute (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
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