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Biography and Research Information
OverviewAI-generated summary
Zinia D’Souza's research focuses on the molecular mechanisms governing Golgi apparatus function, particularly in relation to protein glycosylation and vesicular transport. Her work has investigated the role of specific protein complexes, such as the COG complex and the GARP complex, in maintaining Golgi physiology. D'Souza has examined how the dysfunction of these complexes can lead to disruptions in protein modification, including glycosylation, and impact cellular homeostasis, such as calcium regulation. Her publications also explore the flexibility of SNARE proteins, like Syntaxin-5, in mediating vesicle fusion and supporting Golgi trafficking. D'Souza collaborates extensively with researchers at the University of Arkansas for Medical Sciences, including Vladimir Lupashin and Irina D. Pokrovskaya, with whom she has co-authored multiple publications. Her scholarly output includes 13 publications with a total of 321 citations and an h-index of 9.
Metrics
- h-index: 9
- Publications: 13
- Citations: 328
Selected Publications
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Syntaxin‐5's flexibility in <scp>SNARE</scp> pairing supports Golgi functions (2023)
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GARP dysfunction results in COPI displacement, depletion of Golgi v-SNAREs and calcium homeostasis proteins (2022)
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Acute COG complex inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra‐Golgi recycling vesicles (2022)
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GARP complex controls Golgi physiology by stabilizing COPI machinery and Golgi v-SNAREs (2022)
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Acute COG inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra-Golgi recycling vesicles (2022)
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STX5’s flexibility in SNARE pairing supports Golgi functions (2022)
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Getting Sugar Coating Right! The Role of the Golgi Trafficking Machinery in Glycosylation (2021)
Collaboration Network
Top Collaborators
Vladimir Lupashin
University of Arkansas for Medical Sciences
7 shared publications
In database
- Getting Sugar Coating Right! The Role of the Golgi Trafficking Machinery in Glycosylation
- Syntaxin‐5's flexibility in <scp>SNARE</scp> pairing supports Golgi functions
- Acute COG complex inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra‐Golgi recycling vesicles
- STX5’s flexibility in SNARE pairing supports Golgi functions
- GARP dysfunction results in COPI displacement, depletion of Golgi v-SNAREs and calcium homeostasis proteins
Showing 5 of 7 shared publications
Farhana Taher Sumya
University of Arkansas for Medical Sciences
3 shared publications
In database
- Getting Sugar Coating Right! The Role of the Golgi Trafficking Machinery in Glycosylation
- Acute COG complex inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra‐Golgi recycling vesicles
- Acute COG inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra-Golgi recycling vesicles
Amrita Khakurel
University of Arkansas for Medical Sciences
3 shared publications
In database
- Getting Sugar Coating Right! The Role of the Golgi Trafficking Machinery in Glycosylation
- GARP dysfunction results in COPI displacement, depletion of Golgi v-SNAREs and calcium homeostasis proteins
- GARP complex controls Golgi physiology by stabilizing COPI machinery and Golgi v-SNAREs
Irina D. Pokrovskaya
University of Arkansas for Medical Sciences
3 shared publications
In database
- Acute COG complex inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra‐Golgi recycling vesicles
- STX5’s flexibility in SNARE pairing supports Golgi functions
- Acute COG inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra-Golgi recycling vesicles
I. V. Pokrovskaya
University of Arkansas for Medical Sciences (US)
3 shared publications
- Syntaxin‐5's flexibility in <scp>SNARE</scp> pairing supports Golgi functions
- GARP dysfunction results in COPI displacement, depletion of Golgi v-SNAREs and calcium homeostasis proteins
- GARP complex controls Golgi physiology by stabilizing COPI machinery and Golgi v-SNAREs
Tetyana Kudlyk
University of Arkansas for Medical Sciences
2 shared publications
In database
- GARP dysfunction results in COPI displacement, depletion of Golgi v-SNAREs and calcium homeostasis proteins
- GARP complex controls Golgi physiology by stabilizing COPI machinery and Golgi v-SNAREs
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