Andrew Shen
Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.
PhD Student
Also affiliated: Broad Institute (2025); Northwestern University (2024); United States Food and Drug Administration (2021); Harvard University (2024–2026); University of Kentucky (2016–2018); The University of Melbourne (2022); Behavioral Pharma (United States) (2015); Auburn University (2015–2016)
Graduate Student Researcher
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Andrew Shen's research focuses on the neurobehavioral and neurochemical effects of environmental exposures, particularly in the context of aging. His work investigates the implications of substances like methylmercury and arsenite on developing and aging organisms. Shen also explores the application of artificial intelligence in analyzing molecular datasets and in developing safe reinforcement learning models. His publications include studies on the generalizability of AI models, quantitative proteome responses of bacterial biofilms to antibiotics, and neurobehavioral effects of perinatal chemical exposures in animal models. Shen has a scholarly h-index of 7, with 25 total publications and 179 citations. He collaborates with researchers at the National Center for Toxicological Research, including Héctor Rosas-Hernández and Katelin S. Matazel.
Metrics
- h-index: 7
- Publications: 24
- Citations: 208
Selected Publications
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Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model (2026)
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Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model (2026)
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Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model (2026)
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Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model (2026)
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Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model (2026)
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Concentration-Dependent Global Quantitative Proteome Response of Staphylococcus epidermidis RP62A Biofilms to Subinhibitory Tigecycline (2022)
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Neurobehavioral and neurochemical effects of perinatal arsenite exposure in Sprague-Dawley rats (2021)
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Methylmercury exposure and its implications for aging (2021)
Collaboration Network
Top Collaborators
- Neurobehavioral and neurochemical effects of perinatal arsenite exposure in Sprague-Dawley rats
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
Showing 5 of 6 shared publications
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Methylmercury exposure and its implications for aging
- Neurobehavioral and neurochemical effects of perinatal arsenite exposure in Sprague-Dawley rats
- Neurobehavioral and neurochemical effects of perinatal arsenite exposure in Sprague-Dawley rats
- Neurobehavioral and neurochemical effects of perinatal arsenite exposure in Sprague-Dawley rats
- Neurobehavioral and neurochemical effects of perinatal arsenite exposure in Sprague-Dawley rats
- Neurobehavioral and neurochemical effects of perinatal arsenite exposure in Sprague-Dawley rats
- Concentration-Dependent Global Quantitative Proteome Response of Staphylococcus epidermidis RP62A Biofilms to Subinhibitory Tigecycline
- Concentration-Dependent Global Quantitative Proteome Response of Staphylococcus epidermidis RP62A Biofilms to Subinhibitory Tigecycline
- Concentration-Dependent Global Quantitative Proteome Response of Staphylococcus epidermidis RP62A Biofilms to Subinhibitory Tigecycline
- Concentration-Dependent Global Quantitative Proteome Response of Staphylococcus epidermidis RP62A Biofilms to Subinhibitory Tigecycline
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