Héctor Rosas-Hernández Data-verified
Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.
Staff Fellow
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Research Areas
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Biography and Research Information
OverviewAI-generated summary
Héctor Rosas-Hernández's research centers on neurovascular dysfunction, particularly in the context of toxicological exposure and neurodegenerative diseases. He has investigated the neurobehavioral and neurochemical effects of perinatal arsenite exposure in Sprague-Dawley rats. His work also explores mechanisms of central nervous system infection, including the potential for SARS-CoV-2 to cross the blood-brain barrier via the olfactory bulb. Rosas-Hernández has contributed to modeling neurovascular dysfunction in Alzheimer's disease using an isogenic brain-chip model. He has published 64 papers, accumulating over 1,005 citations and an h-index of 19. Key collaborators include Katelin S. Matazel and W. Drew Gill from the National Center for Toxicological Research, with whom he has co-authored multiple publications.
Metrics
- h-index: 19
- Publications: 64
- Citations: 1,011
Selected Publications
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Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model (2026)
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Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model (2026)
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Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model (2026)
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Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model (2026)
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Neurobehavioral and neurochemical effects of perinatal arsenite exposure in Sprague-Dawley rats (2021)
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Can SARS-CoV-2 infect the central nervous system via the olfactory bulb or the blood-brain barrier? (2021)
Collaboration Network
Top Collaborators
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Neurobehavioral and neurochemical effects of perinatal arsenite exposure in Sprague-Dawley rats
- Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Additional file 1 of Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Modeling neurovascular dysfunction in Alzheimer’s disease using an isogenic brain-chip model
- Can SARS-CoV-2 infect the central nervous system via the olfactory bulb or the blood-brain barrier?
- Can SARS-CoV-2 infect the central nervous system via the olfactory bulb or the blood-brain barrier?
- Can SARS-CoV-2 infect the central nervous system via the olfactory bulb or the blood-brain barrier?
- Can SARS-CoV-2 infect the central nervous system via the olfactory bulb or the blood-brain barrier?
- Neurobehavioral and neurochemical effects of perinatal arsenite exposure in Sprague-Dawley rats
- Neurobehavioral and neurochemical effects of perinatal arsenite exposure in Sprague-Dawley rats
- Neurobehavioral and neurochemical effects of perinatal arsenite exposure in Sprague-Dawley rats
- Neurobehavioral and neurochemical effects of perinatal arsenite exposure in Sprague-Dawley rats
- Neurobehavioral and neurochemical effects of perinatal arsenite exposure in Sprague-Dawley rats
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