Claire Greenhill
This is a likely match — the affiliation was inferred from OpenAlex, ORCID, and web sources but has not been fully confirmed. Treat with appropriate caution.
Chief Editor, Nature Reviews Endocrinology
Also affiliated: The University of Melbourne (2017–2018); Hudson Institute of Medical Research (2014); University of Bristol (2012); Monash University (2006–2014); Monash Institute of Medical Research (2006–2014); Cardiff University (2013–2020)
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Claire Greenhill's research has focused on the molecular mechanisms underlying inflammatory responses and their contribution to disease. Her work has investigated the roles of various signaling pathways, including those involving Interleukin-6 (IL-6), STAT proteins, and Toll-like receptors (TLRs), in modulating immune cell function and driving pathological processes. Specific investigations have explored how IL-6 trans-signaling influences TLR4-dependent inflammation through STAT3 activation and how STAT1 participates in TLR signal transduction. Greenhill has also examined the involvement of cytokines like Interleukin-27 in regulating inflammatory arthritis and the role of Interleukin-10 in inflammasome-driven joint destruction. Furthermore, her research has delved into the mechanisms of nerve growth factor signaling in bone nociceptors and its contribution to inflammatory bone pain, as well as the activation of bone afferent neurons by GDNF family ligands. With a career spanning over 400 publications and an h-index of 17, Greenhill has collaborated extensively with researchers at the University of Arkansas at Fayetteville.
Metrics
- h-index: 17
- Publications: 416
- Citations: 2,485
Positions
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Chief Editor, Nature Reviews Endocrinology 2010–presentNature Publishing Group Nature Reviews ORCID
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Chief Editor, Nature Reviews Endocrinology publications 2025–2026University of Arkansas at Fayetteville ORCID
Selected Publications
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Bone Alterations During the Development of Anorexia Nervosa (2026)
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The Effect of Anorexia Nervosa on Muscle Metabolism in Rat Models. (2026)
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Mitochondrial capacities and quality control following short‐ and long‐term weight restoration after simulated anorexia nervosa (2025)
Collaboration Network
Top Collaborators
- The Effect of Anorexia Nervosa on Muscle Metabolism in Rat Models.
- Bone Alterations During the Development of Anorexia Nervosa
- The Effect of Anorexia Nervosa on Muscle Metabolism in Rat Models.
- Bone Alterations During the Development of Anorexia Nervosa
- The Effect of Anorexia Nervosa on Muscle Metabolism in Rat Models.
- Bone Alterations During the Development of Anorexia Nervosa
- The Effect of Anorexia Nervosa on Muscle Metabolism in Rat Models.
- Bone Alterations During the Development of Anorexia Nervosa
- The Effect of Anorexia Nervosa on Muscle Metabolism in Rat Models.
- Bone Alterations During the Development of Anorexia Nervosa
- The Effect of Anorexia Nervosa on Muscle Metabolism in Rat Models.
- Bone Alterations During the Development of Anorexia Nervosa
- The Effect of Anorexia Nervosa on Muscle Metabolism in Rat Models.
- Bone Alterations During the Development of Anorexia Nervosa
- The Effect of Anorexia Nervosa on Muscle Metabolism in Rat Models.
- Bone Alterations During the Development of Anorexia Nervosa
- Mitochondrial capacities and quality control following short‐ and long‐term weight restoration after simulated anorexia nervosa
- Mitochondrial capacities and quality control following short‐ and long‐term weight restoration after simulated anorexia nervosa
- Mitochondrial capacities and quality control following short‐ and long‐term weight restoration after simulated anorexia nervosa
- Mitochondrial capacities and quality control following short‐ and long‐term weight restoration after simulated anorexia nervosa
- Mitochondrial capacities and quality control following short‐ and long‐term weight restoration after simulated anorexia nervosa
- Mitochondrial capacities and quality control following short‐ and long‐term weight restoration after simulated anorexia nervosa
- Mitochondrial capacities and quality control following short‐ and long‐term weight restoration after simulated anorexia nervosa
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