R
Research Areas
Biography and Research Information
OverviewAI-generated summary
Ruqaiza Muhyudin's research investigates cellular and molecular adaptations in skeletal muscle, particularly in the context of disease and physiological challenges. Her recent publications examine the role of mitochondrial function and antioxidant strategies in mitigating muscle wasting associated with cancer cachexia, exploring these effects across biological sexes. Muhyudin also studies the impact of exercise on muscle adaptations during tumor induction and investigates myocellular responses to conditions simulating anorexia nervosa and subsequent weight restoration. Her work includes transcriptional analysis of cancer cachexia to identify conserved and sex-specific features. Muhyudin collaborates with Nicholas P. Greene, Tyrone A. Washington, Ana Regina Cabrera, and Eleanor R. Schrems, all at the University of Arkansas at Fayetteville, with whom she has co-authored multiple publications.
Metrics
- h-index: 3
- Publications: 10
- Citations: 29
Selected Publications
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Exercise-induced antioxidant programming in oxidative muscle: a critical IL1β-NBR1-p62 axis (2026)
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Mitochondrial capacities and quality control following short‐ and long‐term weight restoration after simulated anorexia nervosa (2025)
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Changes in muscle strength and moderators of protein turnover in a rodent model of anorexia nervosa and recovery (2025)
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Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females (2025)
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Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice (2025)
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Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice (2025)
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Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex (2024)
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Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice (2024)
Collaboration Network
Top Collaborators
Kevin A. Murach
University of Arkansas at Fayetteville
6 shared publications
In database
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
Showing 5 of 6 shared publications
Francielly Morena da Silva
University of Arkansas at Fayetteville
5 shared publications
In database
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
Ana Regina Cabrera
University of Arkansas at Fayetteville
5 shared publications
In database
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
Tyrone A. Washington
University of Arkansas at Fayetteville
5 shared publications
In database
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
Nicholas P. Greene
5 shared publications
In database
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
Stavroula Tsitkanou
University of Arkansas at Fayetteville
4 shared publications
In database
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
Eleanor R. Schrems
University of Arkansas at Fayetteville
4 shared publications
In database
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Transcriptional analysis of cancer cachexia: conserved and unique features across preclinical models and biological sex
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
Sabin Khadgi
University of Arkansas at Fayetteville
4 shared publications
In database
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
Pieter J. Koopmans
University of Arkansas at Fayetteville (US)
3 shared publications
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
Seongkyun Lim
University of Arkansas at Fayetteville
3 shared publications
In database
- Mitochondrial antioxidant SkQ1 attenuates C26 cancer-induced muscle wasting in males and improves muscle contractility in female tumor-bearing mice
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
Calvin Peterson
University of Arkansas at Fayetteville
3 shared publications
In database
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Myocellular adaptations to short‐term weighted wheel‐running exercise are largely conserved during C26‐tumour induction in male and female mice
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
Toby L. Chambers
University of Arkansas at Fayetteville
3 shared publications
In database
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
- Mitochondrial capacities and quality control following short‐ and long‐term weight restoration after simulated anorexia nervosa
Sepideh Shakeri
University of Arkansas at Fayetteville
2 shared publications
In database
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
Kevin Zhao
University of Arkansas at Fayetteville
2 shared publications
In database
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
Dipak Kumar Mishra
University of Arkansas at Fayetteville
2 shared publications
In database
- Promoting mitochondrial fusion is protective against cancer-induced muscle detriments in males and females
- Global mitophagy inhibition via BNIP3 ablation is not sufficient to alleviate skeletal muscle impairments in male and female tumor-bearing mice
Similar Researchers
Based on overlapping research topics
Nicholas P. Greene
University of Arkansas at Fayetteville
Mitochondrial Function and Pathology
Cancer-related molecular mechanisms research
Muscle Physiology and Disorders
Calvin Peterson
University of Arkansas at Fayetteville
Mitochondrial Function and Pathology
Cancer-related molecular mechanisms research
Muscle Physiology and Disorders
Landen W. Saling
University of Arkansas at Fayetteville
Mitochondrial Function and Pathology
Cancer-related molecular mechanisms research
Muscle Physiology and Disorders
Eleanor R. Schrems
University of Arkansas at Fayetteville
Mitochondrial Function and Pathology
Cancer-related molecular mechanisms research
Muscle Physiology and Disorders
Pieter J. Koopmans
University of Arkansas at Fayetteville
Mitochondrial Function and Pathology
Cancer-related molecular mechanisms research
Muscle Physiology and Disorders
Sepideh Shakeri
University of Arkansas at Fayetteville
Mitochondrial Function and Pathology
Cancer-related molecular mechanisms research
Muscle Physiology and Disorders