Bonnie Robinson
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Researcher
Also affiliated: United States Food and Drug Administration (2008–2023); Children's Hospital of Los Angeles (1992)
Faculty Researcher
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Bonnie Robinson's research focuses on understanding the mechanisms of toxicity and the biological effects of chemical exposures, particularly on the nervous system. Her work utilizes various animal models, including zebrafish and rodents, to investigate how substances like inorganic arsenic and ketamine impact neuronal development and function. Recent publications highlight her investigations into how inorganic arsenic alters dopaminergic and motor neuron development in zebrafish, exploring the underlying pathways such as the Sonic hedgehog pathway. She also examines the potential mechanisms behind the antidepressant actions of ketamine, considering the role of L-type calcium channels.
Further research by Robinson involves the development and application of proteomic approaches to identify circulating biomarkers of neurotoxicity. This work aims to find measurable indicators of central nervous system damage in rodent models. Her collaborations include researchers at the National Center for Toxicological Research, with whom she has co-authored multiple publications, indicating a consistent research network. Robinson's scholarship metrics, including an h-index of 21 and over 1,900 citations, reflect her significant contributions to the field.
Metrics
- h-index: 21
- Publications: 43
- Citations: 1,918
Selected Publications
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Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish (2023)
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Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity (2021)
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Tauroursodeoxycholic acid (TUDCA) is neuroprotective in a chronic mouse model of Parkinson’s disease (2020)
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N-acetylcysteine prevents verapamil-induced cardiotoxicity with no effect on the noradrenergic arch-associated neurons in zebrafish (2020)
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Impaired Amyloid Beta Clearance and Brain Microvascular Dysfunction are Present in the Tg-SwDI Mouse Model of Alzheimer’s Disease (2020)
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P4‐064: EVALUATION OF TAU NEUROPATHOLOGY AND OTHER KEY PROTEINS IN THE OLFACTORY BULB AND ITS CORRELATION WITH HIPPOCAMPUS IN HUMAN ALZHEIMER'S DISEASE (2019)
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Nifedipine toxicity is exacerbated by acetyl <scp>l</scp>‐carnitine but alleviated by low‐dose ketamine in zebrafish in vivo (2019)
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Ketamine-induced attenuation of reactive oxygen species in zebrafish is prevented by acetyl l-carnitine in vivo (2019)
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Characterization of Serum Exosomes from a Transgenic Mouse Model of Alzheimer’s Disease (2019)
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Neuroprotective effects of acetyl-l-carnitine (ALC) in a chronic MPTP-induced Parkinson’s disease mouse model: Endothelial and microglial effects (2019)
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The time course of blood brain barrier leakage and its implications on the progression of methamphetamine-induced seizures (2018)
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Identification of altered microRNAs in serum of a mouse model of Parkinson’s disease (2018)
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N-acetylcysteine prevents ketamine-induced adverse effects on development, heart rate and monoaminergic neurons in zebrafish (2018)
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Mechanistic studies on ketamine-induced mitochondrial toxicity in zebrafish embryos (2017)
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Changes in the metabolome and microRNA levels in biological fluids might represent biomarkers of neurotoxicity: A trimethyltin study (2017)
Collaboration Network
Top Collaborators
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
- Circulating biomarkers of neurotoxicity: Proteomics approach reveals fluidic endpoints of central nervous system toxicity in a rodent model of neurotoxicity
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