Kathleen M. Gilbert
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Also affiliated: Tulane University (1980); Scripps Research Institute (1992–1994); Arkansas Children's Hospital (2003–2016); United States Food and Drug Administration (2009); Los Angeles County Department of Health Services (1992); Memorial Sloan Kettering Cancer Center (1983); University of Arkansas Medical Center (2017); Scripps Clinic (1991–1992); Arkansas Children's Research Institute (2003–2019)
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Kathleen M. Gilbert's research has focused on health disparities and outcomes, with a particular emphasis on maternal and perinatal health interventions. Her work also extends to understanding the impact of health and medical research, and explores themes within health sciences research and education. Gilbert has contributed to the literature through recent publications.
Metrics
- h-index: 21
- Publications: 57
- Citations: 1,214
Selected Publications
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Developmental exposure to environmental toxicant trichloroethylene alters DNA methylation in polycomb protein binding regions in effector/memory CD4 + T cells from autoimmune-prone mice (2019)
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Continuous Developmental and Early Life Trichloroethylene Exposure Promoted DNA Methylation Alterations in Polycomb Protein Binding Sites in Effector/Memory CD4+ T Cells (2019)
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Irreversible effects of trichloroethylene on the gut microbial community and gut‐associated immune responses in autoimmune‐prone mice (2018)
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Opposing Actions of Developmental Trichloroethylene and High-Fat Diet Coexposure on Markers of Lipogenesis and Inflammation in Autoimmune-Prone Mice (2018)
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Epigenetic underpinnings of developmental immunotoxicity and autoimmune disease (2017)
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Trichloroethylene-induced alterations in DNA methylation were enriched in polycomb protein binding sites in effector/memory CD4+ T cells (2017)
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Oxidative Stress Challenge Uncovers Trichloroacetaldehyde Hydrate-Induced Mitoplasticity in Autistic and Control Lymphoblastoid Cell Lines (2017)
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Exposure Cessation During Adulthood Did Not Prevent Immunotoxicity Caused by Developmental Exposure to Low-Level Trichloroethylene in Drinking Water (2017)
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Chronic exposure to trichloroethylene increases DNA methylation of the Ifng promoter in CD4+ T cells (2016)
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Chronic Exposure to Water Pollutant Trichloroethylene Increased Epigenetic Drift in CD4 + T cells (2016)
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Hepatitis, Autoimmune (2014)
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Trichloroethylene: Toxicity and Health Risks (2014)
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Modeling toxicodynamic effects of trichloroethylene on liver in mouse model of autoimmune hepatitis (2014)
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Differential Immunotoxicity Induced by Two Different Windows of Developmental Trichloroethylene Exposure (2014)
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Water pollutant trichloroethylene epigenetically alters CD4+ T cells in mouse model of autoimmune disease (2014)
Collaboration Network
Top Collaborators
- Trichloroethylene accelerates an autoimmune response by Th1 T cell activation in MRL+/+ mice
- Delineating Liver Events in Trichloroethylene-Induced Autoimmune Hepatitis
- Activation and attenuation of apoptosis of CD4+ T cells following in vivo exposure to two common environmental toxicants, trichloroacetaldehyde hydrate and trichloroacetic acid
- Epigenetic Alterations May Regulate Temporary Reversal of CD4+ T Cell Activation Caused by Trichloroethylene Exposure
- Epigenetic underpinnings of developmental immunotoxicity and autoimmune disease
Showing 5 of 21 shared publications
- Epigenetic Alterations May Regulate Temporary Reversal of CD4+ T Cell Activation Caused by Trichloroethylene Exposure
- Chronic Exposure to Water Pollutant Trichloroethylene Increased Epigenetic Drift in CD4 + T cells
- Postnatal exposure to trichloroethylene alters glutathione redox homeostasis, methylation potential, and neurotrophin expression in the mouse hippocampus
- Chronic exposure to trichloroethylene increases DNA methylation of the Ifng promoter in CD4+ T cells
- Trichloroethylene-induced alterations in DNA methylation were enriched in polycomb protein binding sites in effector/memory CD4+ T cells
Showing 5 of 7 shared publications
- Trichloroethylene accelerates an autoimmune response by Th1 T cell activation in MRL+/+ mice
- Delineating Liver Events in Trichloroethylene-Induced Autoimmune Hepatitis
- Activation and attenuation of apoptosis of CD4+ T cells following in vivo exposure to two common environmental toxicants, trichloroacetaldehyde hydrate and trichloroacetic acid
- TRICHLOROETHYLENE ACTIVATES CD4+T CELLS: POTENTIAL ROLE IN AN AUTOIMMUNE RESPONSE*
- Environmental contaminant and disinfection by-product trichloroacetaldehyde stimulates T cells in vitro
Showing 5 of 7 shared publications
- Chronic Exposure to Water Pollutant Trichloroethylene Increased Epigenetic Drift in CD4 + T cells
- Chronic exposure to trichloroethylene increases DNA methylation of the Ifng promoter in CD4+ T cells
- Irreversible effects of trichloroethylene on the gut microbial community and gut‐associated immune responses in autoimmune‐prone mice
- Exposure Cessation During Adulthood Did Not Prevent Immunotoxicity Caused by Developmental Exposure to Low-Level Trichloroethylene in Drinking Water
- Trichloroethylene-induced alterations in DNA methylation were enriched in polycomb protein binding sites in effector/memory CD4+ T cells
Showing 5 of 6 shared publications
- Chronic Exposure to Water Pollutant Trichloroethylene Increased Epigenetic Drift in CD4 + T cells
- Chronic exposure to trichloroethylene increases DNA methylation of the Ifng promoter in CD4+ T cells
- Modeling toxicodynamic effects of trichloroethylene on liver in mouse model of autoimmune hepatitis
- Trichloroethylene-induced alterations in DNA methylation were enriched in polycomb protein binding sites in effector/memory CD4+ T cells
- Water pollutant trichloroethylene epigenetically alters CD4+ T cells in mouse model of autoimmune disease
- Structure–activity relationship between carboxylic acids and T cell cycle blockade
- Butyric Acid Derivative Induces Allospecific T Cell Anergy and Prevents Graft-Versus-Host Disease
- T Cell Tolerance Induced by Histone Deacetylase Inhibitor is Mediated by P21cip1
- Macrophage production of inflammatory mediators is potently inhibited by a butyric acid derivative demonstrated to inactivate antigen-stimulated T cells
- Chronic Exposure to Water Pollutant Trichloroethylene Increased Epigenetic Drift in CD4 + T cells
- Chronic exposure to trichloroethylene increases DNA methylation of the Ifng promoter in CD4+ T cells
- Trichloroethylene-induced alterations in DNA methylation were enriched in polycomb protein binding sites in effector/memory CD4+ T cells
- Water pollutant trichloroethylene epigenetically alters CD4+ T cells in mouse model of autoimmune disease
- Epigenetic Alterations May Regulate Temporary Reversal of CD4+ T Cell Activation Caused by Trichloroethylene Exposure
- Chronic Exposure to Water Pollutant Trichloroethylene Increased Epigenetic Drift in CD4 + T cells
- Modeling toxicodynamic effects of trichloroethylene on liver in mouse model of autoimmune hepatitis
- Trichloroethylene-induced alterations in DNA methylation were enriched in polycomb protein binding sites in effector/memory CD4+ T cells
- Induction of Anergy in Th1 Cells Associated with Increased Levels of Cyclin-Dependent Kinase Inhibitors p21Cip1 and p27Kip1 1
- The ability of antigen, but not interleukin‐2, to promote n‐butyrate‐induced T helper 1 cell anergy is associated with increased expression and altered association patterns of cyclin‐dependent kinase inhibitors
- Structure–activity relationship between carboxylic acids and T cell cycle blockade
- Butyric Acid Derivative Induces Allospecific T Cell Anergy and Prevents Graft-Versus-Host Disease
- T Cell Tolerance Induced by Histone Deacetylase Inhibitor is Mediated by P21cip1
- Macrophage production of inflammatory mediators is potently inhibited by a butyric acid derivative demonstrated to inactivate antigen-stimulated T cells
- Trichloroethylene accelerates an autoimmune response by Th1 T cell activation in MRL+/+ mice
- Induction of Anergy in Th1 Cells Associated with Increased Levels of Cyclin-Dependent Kinase Inhibitors p21Cip1 and p27Kip1 1
- The ability of antigen, but not interleukin‐2, to promote n‐butyrate‐induced T helper 1 cell anergy is associated with increased expression and altered association patterns of cyclin‐dependent kinase inhibitors
- Chronic Exposure to Water Pollutant Trichloroethylene Increased Epigenetic Drift in CD4 + T cells
- Chronic exposure to trichloroethylene increases DNA methylation of the Ifng promoter in CD4+ T cells
- Trichloroethylene-induced alterations in DNA methylation were enriched in polycomb protein binding sites in effector/memory CD4+ T cells
- Trichloroethylene-induced alterations in DNA methylation were enriched in polycomb protein binding sites in effector/memory CD4+ T cells
- Continuous Developmental and Early Life Trichloroethylene Exposure Promoted DNA Methylation Alterations in Polycomb Protein Binding Sites in Effector/Memory CD4+ T Cells
- Developmental exposure to environmental toxicant trichloroethylene alters DNA methylation in polycomb protein binding regions in effector/memory CD4 + T cells from autoimmune-prone mice
- Histone deacetylase inhibitor uses p21Cip1 to maintain anergy in CD4+ T cells
- p21Cip1 up‐regulated during histone deacetylase inhibitor‐induced CD4+ T‐cell anergy selectively associates with mitogen‐activated protein kinases
- Recent Advances and Opportunities in Research on Lupus: Environmental Influences and Mechanisms of Disease
- Recent advances and opportunities in research on lupus: environmental influences and mechanisms of disease
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