Akash Saha
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Researcher
Also affiliated: Winthrop Rockefeller Foundation (2026)
Graduate Student Researcher
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Akash Saha's research focuses on the molecular mechanisms underlying cancer, specifically investigating the role of FAM60A in the Hippo signaling pathway. His recent publication details how the loss of FAM60A disrupts the Sin3/HDAC complex, leading to the activation of YAP1, a key factor in oncogenic processes. This work contributes to understanding how disruptions in protein complexes can promote cancer development. Saha collaborates with researchers at the University of Arkansas for Medical Sciences, including Michael Bauer, Mohammad Alinoor Rahman, Bolni Marius Nagalo, and Md. Rafikul Islam, with whom he has co-authored publications.
Metrics
- Publications: 1
Selected Publications
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Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation (2026)
Collaboration Network
Top Collaborators
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
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