Md Nazmul Huda
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Biochemistry & Molecular Biology, College of Medicine
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Md Nazmul Huda's research focuses on the molecular mechanisms underlying cancer development, particularly how oncogenic signaling pathways regulate chromatin structure. His work investigates the disruption of normal cellular processes by genetic alterations and the subsequent promotion of uncontrolled cell growth. Huda has published research on the role of specific proteins and complexes, such as FAM60A and the Sin3/HDAC complex, in controlling signaling pathways like the Hippo pathway and the activation of YAP1, a key factor in tumorigenesis. His scholarship metrics include an h-index of 1, with 3 total publications and 21 citations. Huda collaborates with researchers at the University of Arkansas for Medical Sciences, including Sayem Miah, Brendan Frett, Baku Acharya, and Alan J. Tackett, with whom he shares multiple publications.
Metrics
- h-index: 1
- Publications: 3
- Citations: 21
Selected Publications
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Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation (2026)
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Oncogenic signaling-mediated regulation of chromatin during tumorigenesis (2023)
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443 Team Science (2023)
Collaboration Network
Top Collaborators
- Oncogenic signaling-mediated regulation of chromatin during tumorigenesis
- 443 Team Science
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Oncogenic signaling-mediated regulation of chromatin during tumorigenesis
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- 443 Team Science
- Oncogenic signaling-mediated regulation of chromatin during tumorigenesis
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
- Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation
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