Match tier Listed
Presence Current · Arkansas
Last published 2026
Sources OpenAlex · ORCID
Refreshed 2026-10-03
Md Nazmul Huda profile photo

Md Nazmul Huda

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Biochemistry & Molecular Biology, College of Medicine

1 h-index 3 pubs 21 cited

  • Chromatin
  • Humans
  • Phosphatidylinositol 3-Kinases
  • Phosphoproteins
  • Repressor Proteins
  • Transcription Factors
  • Protein Serine-Threonine Kinases
  • Adaptor Proteins, Signal Transducing
  • Sin3 Histone Deacetylase and Corepressor Complex
  • Histone Deacetylase 1
  • Cell Transformation, Neoplastic
  • Wnt Signaling Pathway
  • Carcinogenesis
  • Hippo Signaling Pathway
  • YAP-Signaling Proteins

Biography and Research Information

OverviewAI-generated summary

Md Nazmul Huda's research focuses on the molecular mechanisms underlying cancer development, particularly how oncogenic signaling pathways regulate chromatin structure. His work investigates the disruption of normal cellular processes by genetic alterations and the subsequent promotion of uncontrolled cell growth. Huda has published research on the role of specific proteins and complexes, such as FAM60A and the Sin3/HDAC complex, in controlling signaling pathways like the Hippo pathway and the activation of YAP1, a key factor in tumorigenesis. His scholarship metrics include an h-index of 1, with 3 total publications and 21 citations. Huda collaborates with researchers at the University of Arkansas for Medical Sciences, including Sayem Miah, Brendan Frett, Baku Acharya, and Alan J. Tackett, with whom he shares multiple publications.

Metrics

  • h-index: 1
  • Publications: 3
  • Citations: 21

Selected Publications

  • Loss of FAM60A disrupts Sin3/HDAC control of the Hippo signaling and promotes oncogenic YAP1 activation (2026)
    Cell Death and Disease DOI OpenAlex
  • Oncogenic signaling-mediated regulation of chromatin during tumorigenesis (2023)
    Cancer and Metastasis Reviews 19 citations DOI OpenAlex
  • 443 Team Science (2023)
    Journal of Clinical and Translational Science DOI OpenAlex

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Collaboration Network

17 Collaborators 6 Institutions 1 Country

Top Collaborators

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