Andrew Morris
Sourced from institutional research profiles (UAMS TRI or ARA).
Professor, Mehta/Stebbins Chair in Cardiovascular Research
Also affiliated: Central Arkansas Veterans Healthcare System (2023–2026)
Faculty Researcher
Pharmacology and Toxicology
Upstream record may be merged OpenAlex, the source of these figures, lists 76 institutions in 6 countries for this author record — a pattern that usually means it combines several researchers with similar names. The totals above may include work by other people.
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Andrew Morris is a Professor at the University of Arkansas for Medical Sciences with a scholarly record encompassing 414 publications and over 22,000 citations, reflected in an h-index of 77. His research group's work investigates mechanisms of disease and metabolic processes, with recent publications focusing on the role of autotaxin in suppressing anti-tumor immunity, the impact of adipose-specific PPARα knockout on lipogenesis and macrophage polarization, and the effects of phthalate exposure on lipid homeostasis. Morris's group also studies the gut microbiota metabolome using stable isotope probing and the association of short-chain fatty acids with inflammatory markers in acute ischemic stroke patients.
Further research has explored the impact of suppressing hepatic UGT1A1 on plasma bilirubin, kinase signaling, and fatty liver disease. His work also includes the development and application of analytical techniques for per- and polyfluoroalkyl substances in water samples. Morris leads a research group and has been recently active in research, with a publication in 2023 and a recent activity status indicating continued engagement.
Metrics
- h-index: 77
- Publications: 414
- Citations: 22,261
Selected Publications
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How does lipid phosphatase 1 (LPP1) regulate antitumor CD8+ T cell activity? (2025)
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PITPβ promotes COPI vesicle fission through lipid transfer and membrane contact formation (2025)
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Comparison of the capillary and venous blood plasma lipidomes: validation of self-collected blood for plasma lipidomics (2025)
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Glucocorticoid resistance remodels liver lipids and prompts lipogenesis, eicosanoid, and inflammatory pathways (2024)
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Structure and mechanism of biosynthesis of <i>Streptococcus mutans</i> cell wall polysaccharide (2024)
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Intermittent glucocorticoid treatment improves muscle metabolism via the PGC1α/Lipin1 axis in an aging-related sarcopenia model (2024)
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Neurotensin accelerates atherosclerosis and increases circulating levels of short-chain and saturated triglycerides (2024)
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Autotaxin Inhibition with IOA-289 Decreases Breast Tumor Growth in Mice Whereas Knockout of Autotaxin in Adipocytes Does Not (2023)
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Bilirubin Nanoparticle Treatment in Obese Mice Inhibits Hepatic Ceramide Production and Remodels Liver Fat Content (2023)
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Suppressing Hepatic UGT1A1 Increases Plasma Bilirubin, Lowers Plasma Urobilin, Reorganizes Kinase Signaling Pathways and Lipid Species and Improves Fatty Liver Disease (2023)
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PFAS concentrations associate with placental transcriptomic activity: a pilot study (2022)
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Myeloid-Specific Deletion of Lipid Plpp3 (Phosphate Phosphatase 3) Increases Cardiac Inflammation After Myocardial Infarction (2022)
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A Mediterranean Meal Plan Modulates Fatty Acid Content of Human Milk in Women With Obesity (2022)
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Early Life Stress Increases Lipid Storage in Female Mice Fed a High Fat Diet via MR Activation in Adipocytes (2022)
Federal Grants 3
Serum Amyloid as a Critical mediator between inflammation and thrombosis
Grants & Funding
As listed on this researcher's institutional profile. Federal awards with verified records are shown above.
- Advion Triversa Nanomate/ABSciex Nanospray III Ion Source for Targeted Lipidomics NIH
- BIACORE 2000 SURFACE PLASMON RESONANCE BIOSENSOR NIH
- FASEB SUMMER RESEARCH CONFERENCE: BIOACTIVE LYSOLIPIDS NIH
- ShEEP Request For Gas Chromatograph Mass Spectrometer NIH
- Role of Lipid Phosphatases in Cholesterol and Triglyceride Synthesis NIH
- REGULATION OF PHOSPHOLIPASE D NIH
- Acquisition of an HPLC Electrospray/chemical ionization triple quadrupole linear ion trap mass spectrometer system. NIH
- Ryanodine Receptors as Therapeutic Targets to Prevent Doxorubicin-Induced Lymphatic Dysfunction NIH/Nat. Cancer Institute
- FASEB Summer Conference on Phospholipases NIH
- Thermo/Finnigan TSQ AM Triple Stage Quadrupole Mass Spectrometer NIH
- Triple Quadrupole Mass Spectrometer System NIH
- Association of a common variant of the PPAP2B gene with cardiovascular disease. NIH
- Lipid phosphate phosphatase 3 as a novel atherosclerosis suppressor NIH
- Lysophospholipid Mediators in Ovarian Cancer NIH
Collaboration Network
Top Collaborators
- Suppressing Hepatic UGT1A1 Increases Plasma Bilirubin, Lowers Plasma Urobilin, Reorganizes Kinase Signaling Pathways and Lipid Species and Improves Fatty Liver Disease
- Bilirubin Nanoparticle Treatment in Obese Mice Inhibits Hepatic Ceramide Production and Remodels Liver Fat Content
- Glucocorticoid resistance remodels liver lipids and prompts lipogenesis, eicosanoid, and inflammatory pathways
- Early Life Stress Increases Lipid Storage in Female Mice Fed a High Fat Diet via MR Activation in Adipocytes
- Suppressing Hepatic UGT1A1 Increases Plasma Bilirubin, Lowers Plasma Urobilin, Reorganizes Kinase Signaling Pathways and Lipid Species and Improves Fatty Liver Disease
- Bilirubin Nanoparticle Treatment in Obese Mice Inhibits Hepatic Ceramide Production and Remodels Liver Fat Content
- Glucocorticoid resistance remodels liver lipids and prompts lipogenesis, eicosanoid, and inflammatory pathways
- Suppressing Hepatic UGT1A1 Increases Plasma Bilirubin, Lowers Plasma Urobilin, Reorganizes Kinase Signaling Pathways and Lipid Species and Improves Fatty Liver Disease
- Bilirubin Nanoparticle Treatment in Obese Mice Inhibits Hepatic Ceramide Production and Remodels Liver Fat Content
- Glucocorticoid resistance remodels liver lipids and prompts lipogenesis, eicosanoid, and inflammatory pathways
- Suppressing Hepatic UGT1A1 Increases Plasma Bilirubin, Lowers Plasma Urobilin, Reorganizes Kinase Signaling Pathways and Lipid Species and Improves Fatty Liver Disease
- Bilirubin Nanoparticle Treatment in Obese Mice Inhibits Hepatic Ceramide Production and Remodels Liver Fat Content
- Glucocorticoid resistance remodels liver lipids and prompts lipogenesis, eicosanoid, and inflammatory pathways
- Suppressing Hepatic UGT1A1 Increases Plasma Bilirubin, Lowers Plasma Urobilin, Reorganizes Kinase Signaling Pathways and Lipid Species and Improves Fatty Liver Disease
- Early Life Stress Increases Lipid Storage in Female Mice Fed a High Fat Diet via MR Activation in Adipocytes
- A Mediterranean Meal Plan Modulates Fatty Acid Content of Human Milk in Women With Obesity
- PFAS concentrations associate with placental transcriptomic activity: a pilot study
- Suppressing Hepatic UGT1A1 Increases Plasma Bilirubin, Lowers Plasma Urobilin, Reorganizes Kinase Signaling Pathways and Lipid Species and Improves Fatty Liver Disease
- Bilirubin Nanoparticle Treatment in Obese Mice Inhibits Hepatic Ceramide Production and Remodels Liver Fat Content
- Bilirubin Nanoparticle Treatment in Obese Mice Inhibits Hepatic Ceramide Production and Remodels Liver Fat Content
- Neurotensin accelerates atherosclerosis and increases circulating levels of short-chain and saturated triglycerides
- Bilirubin Nanoparticle Treatment in Obese Mice Inhibits Hepatic Ceramide Production and Remodels Liver Fat Content
- Neurotensin accelerates atherosclerosis and increases circulating levels of short-chain and saturated triglycerides
- Early Life Stress Increases Lipid Storage in Female Mice Fed a High Fat Diet via MR Activation in Adipocytes
- Early Life Stress Increases Lipid Storage in Female Mice Fed a High Fat Diet via MR Activation in Adipocytes
- Early Life Stress Increases Lipid Storage in Female Mice Fed a High Fat Diet via MR Activation in Adipocytes
- A Mediterranean Meal Plan Modulates Fatty Acid Content of Human Milk in Women With Obesity
- A Mediterranean Meal Plan Modulates Fatty Acid Content of Human Milk in Women With Obesity
- A Mediterranean Meal Plan Modulates Fatty Acid Content of Human Milk in Women With Obesity
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