Ashim K. Bagchi
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Formerly Arkansas Affiliated with UAMS through 2024; recent publications list University of Manitoba.
Research Areas
Biomedical Subjects
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Biography and Research Information
OverviewAI-generated summary
Ashim K. Bagchi's research focuses on understanding the molecular mechanisms underlying cardiovascular diseases, particularly in the context of diabetes and oxidative stress. His work investigates how cellular processes like apoptosis and signal transduction are altered in conditions such as heart failure and doxorubicin-induced cardiomyopathy. Bagchi has explored the protective effects of various compounds, including Vitamin C and oleic acid, against oxidative and nitrosative stress in cardiac cells. He also studies the role of signaling pathways, such as NF-κB and Akt, in regulating inflammatory responses and cell survival in the cardiovascular system. His research has been supported by collaborations with colleagues at the University of Arkansas for Medical Sciences, including Amanda J. Stolarz, Rushita A. Bagchi, Soumiya Pal, and Jawahar L. Mehta. Bagchi has published 67 papers, accumulating 1,290 citations, with an h-index of 18.
Metrics
- h-index: 18
- Publications: 67
- Citations: 1,290
Selected Publications
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Obesity, histone acetylation, and insulin resistance (2024)
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List of contributors (2024)
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Rhythmic Contractions of Lymph Vessels and Lymph Flow Are Disrupted in Hypertensive Rats (2024)
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Proteomics Approach to Identify Anthracycline-induced Cardiotoxicity Mechanisms in Human Cardiac Fibroblasts (2024)
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JoVE Video Dataset (2024)
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Real-Time Evaluation of Absolute, Cytosolic, Free Ca2+ and Corresponding Contractility in Isolated, Pressurized Lymph Vessels (2024)
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Doxorubicin‑induced cardiomyopathy is mitigated by empagliflozin via the modulation of endoplasmic reticulum stress pathways (2024)
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The cardioprotective effects of Empagliflozin in Doxorubicin-induced endoplasmic reticulum (ER) stress (2023)
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Lipidomic Predictors of Coronary No-Reflow (2023)
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Olive oil protects against progression of heart failure by inhibiting remodeling of heart subsequent to myocardial infarction in rats (2022)
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NF-κB, A Potential Therapeutic Target in Cardiovascular Diseases (2022)
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The Structural Basis of Effective LOX-1 Inhibition (2022)
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Interleukin-10 Mitigates Doxorubicin-Induced Endoplasmic Reticulum Stress as Well as Cardiomyopathy (2022)
Collaboration Network
Top Collaborators
- Interleukin-10 Mitigates Doxorubicin-Induced Endoplasmic Reticulum Stress as Well as Cardiomyopathy
- Olive oil protects against progression of heart failure by inhibiting remodeling of heart subsequent to myocardial infarction in rats
- Doxorubicin‑induced cardiomyopathy is mitigated by empagliflozin via the modulation of endoplasmic reticulum stress pathways
- Lipidomic Predictors of Coronary No-Reflow
- The cardioprotective effects of Empagliflozin in Doxorubicin-induced endoplasmic reticulum (ER) stress
- Interleukin-10 Mitigates Doxorubicin-Induced Endoplasmic Reticulum Stress as Well as Cardiomyopathy
- Olive oil protects against progression of heart failure by inhibiting remodeling of heart subsequent to myocardial infarction in rats
- Doxorubicin‑induced cardiomyopathy is mitigated by empagliflozin via the modulation of endoplasmic reticulum stress pathways
- The cardioprotective effects of Empagliflozin in Doxorubicin-induced endoplasmic reticulum (ER) stress
- Interleukin-10 Mitigates Doxorubicin-Induced Endoplasmic Reticulum Stress as Well as Cardiomyopathy
- Olive oil protects against progression of heart failure by inhibiting remodeling of heart subsequent to myocardial infarction in rats
- Doxorubicin‑induced cardiomyopathy is mitigated by empagliflozin via the modulation of endoplasmic reticulum stress pathways
- The cardioprotective effects of Empagliflozin in Doxorubicin-induced endoplasmic reticulum (ER) stress
- Real-Time Evaluation of Absolute, Cytosolic, Free Ca2+ and Corresponding Contractility in Isolated, Pressurized Lymph Vessels
- JoVE Video Dataset
- Proteomics Approach to Identify Anthracycline-induced Cardiotoxicity Mechanisms in Human Cardiac Fibroblasts
- Rhythmic Contractions of Lymph Vessels and Lymph Flow Are Disrupted in Hypertensive Rats
- Real-Time Evaluation of Absolute, Cytosolic, Free Ca2+ and Corresponding Contractility in Isolated, Pressurized Lymph Vessels
- JoVE Video Dataset
- Rhythmic Contractions of Lymph Vessels and Lymph Flow Are Disrupted in Hypertensive Rats
- Proteomics Approach to Identify Anthracycline-induced Cardiotoxicity Mechanisms in Human Cardiac Fibroblasts
- List of contributors
- Obesity, histone acetylation, and insulin resistance
- Proteomics Approach to Identify Anthracycline-induced Cardiotoxicity Mechanisms in Human Cardiac Fibroblasts
- List of contributors
- Obesity, histone acetylation, and insulin resistance
- NF-κB, A Potential Therapeutic Target in Cardiovascular Diseases
- The Structural Basis of Effective LOX-1 Inhibition
- The Structural Basis of Effective LOX-1 Inhibition
- The Structural Basis of Effective LOX-1 Inhibition
- NF-κB, A Potential Therapeutic Target in Cardiovascular Diseases
- NF-κB, A Potential Therapeutic Target in Cardiovascular Diseases
- NF-κB, A Potential Therapeutic Target in Cardiovascular Diseases
- NF-κB, A Potential Therapeutic Target in Cardiovascular Diseases
- NF-κB, A Potential Therapeutic Target in Cardiovascular Diseases
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