Research Areas
Links
Biography and Research Information
OverviewAI-generated summary
Elisabeth Ferreira's research group at the University of Arkansas for Medical Sciences investigates mechanisms of bone healing and regeneration. Her work has explored the role of gene transfer in potentiating bone formation, specifically examining how IL-1Ra gene transfer can redirect osteogenesis toward endochondral ossification. This research has led to publications on the efficacy of such interventions in promoting bone repair.
Further research interests include the cellular and molecular processes underlying other physiological and pathological conditions. Publications indicate studies on the structural basis of protein interactions involved in apoptosis, and the attenuation of epithelial-mesenchymal transition by natural compounds. Ferreira's group has also developed tools for <i>in vivo</i> assessment of chondrogenesis, utilizing transgenic animal models. Collaborations within the University of Arkansas system and with external institutions contribute to the breadth of her research network.
Metrics
- h-index: 22
- Publications: 46
- Citations: 1,668
Selected Publications
-
Fibrocartilage repair involves chronic cellular senescence in a rat model of bone marrow stimulation (2025)
-
Structural basis of BAK sequestration by MCL-1 in apoptosis (2025)
-
CHRONIC SENESCENCE CONTRIBUTES TO FIBROCARTILAGE REPAIR IN A PRECLINICAL MODEL OF BONE MARROW STIMULATION (2024)
-
IL-1Ra gene transfer potentiates BMP2-mediated bone healing by redirecting osteogenesis toward endochondral ossification (2022)
-
A Transgenic Rat for Noninvasive Assessment of Chondrogenesis <i>in Vivo</i> (2021)
Collaboration Network
Top Collaborators
- IL-1Ra gene transfer potentiates BMP2-mediated bone healing by redirecting osteogenesis toward endochondral ossification
- A Transgenic Rat for Noninvasive Assessment of Chondrogenesis <i>in Vivo</i>
- CHRONIC SENESCENCE CONTRIBUTES TO FIBROCARTILAGE REPAIR IN A PRECLINICAL MODEL OF BONE MARROW STIMULATION
- Fibrocartilage repair involves chronic cellular senescence in a rat model of bone marrow stimulation
- A Transgenic Rat for Noninvasive Assessment of Chondrogenesis <i>in Vivo</i>
- CHRONIC SENESCENCE CONTRIBUTES TO FIBROCARTILAGE REPAIR IN A PRECLINICAL MODEL OF BONE MARROW STIMULATION
- Fibrocartilage repair involves chronic cellular senescence in a rat model of bone marrow stimulation
- A Transgenic Rat for Noninvasive Assessment of Chondrogenesis <i>in Vivo</i>
- CHRONIC SENESCENCE CONTRIBUTES TO FIBROCARTILAGE REPAIR IN A PRECLINICAL MODEL OF BONE MARROW STIMULATION
- Fibrocartilage repair involves chronic cellular senescence in a rat model of bone marrow stimulation
- Uvaol attenuates TGF-β1-induced epithelial-mesenchymal transition in human alveolar epithelial cells by modulating expression and membrane localization of β-catenin
- Luminescent Nanocomposite SiO<sub>2</sub>/EuTTA/ZIF-8 Loaded with Uvaol: Synthesis, Characterization, Anti-Inflammatory Effects, and Molecular Docking Analysis
- New Cinnamamide and Derivatives as New Larvicides against <i>Aedes aegypti</i> Vector Larvae: Facile Synthesis, <i>In Silico</i> Study, <i>In Vitro</i> Noncytotoxicity, and Nontoxicity against Zebrafish
- A Transgenic Rat for Noninvasive Assessment of Chondrogenesis <i>in Vivo</i>
- Fibrocartilage repair involves chronic cellular senescence in a rat model of bone marrow stimulation
- CHRONIC SENESCENCE CONTRIBUTES TO FIBROCARTILAGE REPAIR IN A PRECLINICAL MODEL OF BONE MARROW STIMULATION
- Fibrocartilage repair involves chronic cellular senescence in a rat model of bone marrow stimulation
- Structural basis of BAK sequestration by MCL-1 in apoptosis
- Abstract 1442 Efficacy of BCL-2 Guardian-Inhibiting Drugs
- IL-1Ra gene transfer potentiates BMP2-mediated bone healing by redirecting osteogenesis toward endochondral ossification
- IL-1Ra gene transfer potentiates BMP2-mediated bone healing by redirecting osteogenesis toward endochondral ossification
- IL-1Ra gene transfer potentiates BMP2-mediated bone healing by redirecting osteogenesis toward endochondral ossification
- IL-1Ra gene transfer potentiates BMP2-mediated bone healing by redirecting osteogenesis toward endochondral ossification
- IL-1Ra gene transfer potentiates BMP2-mediated bone healing by redirecting osteogenesis toward endochondral ossification
- IL-1Ra gene transfer potentiates BMP2-mediated bone healing by redirecting osteogenesis toward endochondral ossification
- IL-1Ra gene transfer potentiates BMP2-mediated bone healing by redirecting osteogenesis toward endochondral ossification
- IL-1Ra gene transfer potentiates BMP2-mediated bone healing by redirecting osteogenesis toward endochondral ossification
Similar Researchers
Based on overlapping research topics