Match tier
Listed
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-10-05
Biography and Research Information
OverviewAI-generated summary
Cecile Bustamante-Gomez studies bone remodeling and its therapeutic modulation, with a focus on murine models. Her research investigates the role of specific molecular pathways, such as RANKL and Wnt10b, in bone loss and healing. She has examined the effects of denosumab and romosozumab on bone remodeling in mice and explored the potential of sclerostin inhibition for treating lytic lesions in multiple myeloma.
Bustamante-Gomez also works on defining mesenchymal cell populations associated with periosteal and endosteal bone in mice. Her publications include research on osteomyelitis, bone loss in disease models, and the anabolic response to bone-modulating agents. Her collaborations at the University of Arkansas for Medical Sciences include work with Charles A. O’Brien, Qiang Fu, Visanu Wanchai, and Jinhu Xiong.
Metrics
- h-index: 3
- Publications: 7
- Citations: 63
Positions
-
research associate 2019–presentUniversity of Arkansas for Medical Sciences Endocrinology ORCID
Selected Publications
-
Healing of lytic lesions and restoration of bone health in multiple myeloma through sclerostin inhibition (2025)
-
Potent suppression of bone remodeling by denosumab does not blunt the anabolic response to romosozumab in mice (2025)
-
RANKL-mediated osteoclast formation is required for bone loss in a murine model of Staphylococcus aureus osteomyelitis (2024)
-
Refining the identity of mesenchymal cell types associated with murine periosteal and endosteal bone (2024)
-
A framework for defining mesenchymal cell types associated with murine periosteal and endosteal bone (2023)
Collaboration Network
Top Collaborators
Charles A. O’Brien
University of Arkansas for Medical Sciences
4 shared publications
In database
- Refining the identity of mesenchymal cell types associated with murine periosteal and endosteal bone
- RANKL-mediated osteoclast formation is required for bone loss in a murine model of Staphylococcus aureus osteomyelitis
- Potent suppression of bone remodeling by denosumab does not blunt the anabolic response to romosozumab in mice
- A framework for defining mesenchymal cell types associated with murine periosteal and endosteal bone
Intawat Nookaew
University of Arkansas for Medical Sciences
3 shared publications
In database
- Refining the identity of mesenchymal cell types associated with murine periosteal and endosteal bone
- A framework for defining mesenchymal cell types associated with murine periosteal and endosteal bone
- Healing of lytic lesions and restoration of bone health in multiple myeloma through sclerostin inhibition
Qiang Fu
University of Arkansas for Medical Sciences
3 shared publications
In database
- Refining the identity of mesenchymal cell types associated with murine periosteal and endosteal bone
- Potent suppression of bone remodeling by denosumab does not blunt the anabolic response to romosozumab in mice
- A framework for defining mesenchymal cell types associated with murine periosteal and endosteal bone
Jinhu Xiong
University of Arkansas for Medical Sciences
2 shared publications
In database
- Refining the identity of mesenchymal cell types associated with murine periosteal and endosteal bone
- A framework for defining mesenchymal cell types associated with murine periosteal and endosteal bone
Melda Onal
University of Arkansas for Medical Sciences
2 shared publications
In database
- Refining the identity of mesenchymal cell types associated with murine periosteal and endosteal bone
- A framework for defining mesenchymal cell types associated with murine periosteal and endosteal bone
Visanu Wanchai
University of Arkansas for Medical Sciences
2 shared publications
In database
- Refining the identity of mesenchymal cell types associated with murine periosteal and endosteal bone
- A framework for defining mesenchymal cell types associated with murine periosteal and endosteal bone
Ha‐Neui Kim
University of Arkansas for Medical Sciences
2 shared publications
In database
- Refining the identity of mesenchymal cell types associated with murine periosteal and endosteal bone
- A framework for defining mesenchymal cell types associated with murine periosteal and endosteal bone
Maria Almeida
University of Arkansas for Medical Sciences
2 shared publications
In database
- Refining the identity of mesenchymal cell types associated with murine periosteal and endosteal bone
- A framework for defining mesenchymal cell types associated with murine periosteal and endosteal bone
Humberto Reyes-Pardo
University of Arkansas for Medical Sciences
2 shared publications
In database
- RANKL-mediated osteoclast formation is required for bone loss in a murine model of Staphylococcus aureus osteomyelitis
- Potent suppression of bone remodeling by denosumab does not blunt the anabolic response to romosozumab in mice
Mara J. Campbell
University of Arkansas for Medical Sciences
1 shared publication
In database
- RANKL-mediated osteoclast formation is required for bone loss in a murine model of Staphylococcus aureus osteomyelitis
Qiang Fu
University of Arkansas for Medical Sciences (US)
1 shared publication
- RANKL-mediated osteoclast formation is required for bone loss in a murine model of Staphylococcus aureus osteomyelitis
Karen E. Beenken
University of Arkansas for Medical Sciences
1 shared publication
In database
- RANKL-mediated osteoclast formation is required for bone loss in a murine model of Staphylococcus aureus osteomyelitis
Mark S. Smeltzer
University of Arkansas for Medical Sciences
1 shared publication
In database
- RANKL-mediated osteoclast formation is required for bone loss in a murine model of Staphylococcus aureus osteomyelitis
Joseph J. Goellner
University of Arkansas for Medical Sciences
1 shared publication
In database
- Potent suppression of bone remodeling by denosumab does not blunt the anabolic response to romosozumab in mice
Jeff D. Thostenson
University of Arkansas for Medical Sciences
1 shared publication
In database
- Potent suppression of bone remodeling by denosumab does not blunt the anabolic response to romosozumab in mice
Similar Researchers
Based on overlapping research topics
Qiang Fu
University of Arkansas for Medical Sciences
Bone Metabolism and Diseases
Disease Models, Animal
Mesenchymal stem cell research
Elysia A. Masters
National Center for Toxicological Research
Bone Metabolism and Diseases
Disease Models, Animal
Pharmacological Effects and Toxicity Studies
Rebekah Margaret Samsonraj
University of Arkansas at Fayetteville
Bone Metabolism and Diseases
Mesenchymal stem cell research
Pharmacological Effects and Toxicity Studies
Gabriel Gifford
University of Central Arkansas
Disease Models, Animal
Pharmacological Effects and Toxicity Studies
Host-Pathogen Interactions
Michael Zotti
University of Arkansas at Fayetteville
Disease Models, Animal
Mesenchymal stem cell research
Pharmacological Effects and Toxicity Studies
Sanja Novak
University of Arkansas for Medical Sciences
Bone Metabolism and Diseases
Disease Models, Animal
Mesenchymal stem cell research