Biography and Research Information
OverviewAI-generated summary
Eunice Odiase's research investigates the human gastrointestinal microbiome and its relationship with feeding modes in infants, as well as its connection to growth status. She has published work on how gut microbiota differs between exclusively breastfed and formula-fed infants in the United States. Odiase also studies the mechanisms behind eosinophilic esophagitis, specifically how Th2 cytokines influence eotaxin-3 secretion in esophageal squamous cells. Her work has explored the role of proton pumps and intracellular calcium in this process.
Additionally, Odiase has contributed to research on sedation-free transnasal esophagoscopy for monitoring esophageal diseases in children with esophageal atresia-tracheoesophageal fistula. She has also identified anemia as a presenting sign for gastrinoma. Her scholarship metrics include an h-index of 4, with 9 total publications and 136 total citations. Odiase collaborates with P Strebeck and J Cotoco at the University of Arkansas for Medical Sciences.
Metrics
- h-index: 5
- Publications: 9
- Citations: 146
Selected Publications
-
Anemia as a presenting sign for gastrinoma (2024)
Collaboration Network
Top Collaborators
Daniel N. Frank
University of Colorado Anschutz Medical Campus (US)
2 shared publications
- The Gut Microbiota Differ in Exclusively Breastfed and Formula-Fed United States Infants and are Associated with Growth Status
- Gut Microbiota Is Significantly Impacted by Feeding Mode and Associated With Growth Status in Exclusively Breastfed and Formula-Fed US Infants
Bridget E. Young
University of Rochester (US)
2 shared publications
- The Gut Microbiota Differ in Exclusively Breastfed and Formula-Fed United States Infants and are Associated with Growth Status
- Gut Microbiota Is Significantly Impacted by Feeding Mode and Associated With Growth Status in Exclusively Breastfed and Formula-Fed US Infants
Charles E. Robertson
University of Colorado Anschutz Medical Campus (US)
2 shared publications
- The Gut Microbiota Differ in Exclusively Breastfed and Formula-Fed United States Infants and are Associated with Growth Status
- Gut Microbiota Is Significantly Impacted by Feeding Mode and Associated With Growth Status in Exclusively Breastfed and Formula-Fed US Infants
Jennifer M. Kofonow
University of Colorado Anschutz Medical Campus (US)
2 shared publications
- The Gut Microbiota Differ in Exclusively Breastfed and Formula-Fed United States Infants and are Associated with Growth Status
- Gut Microbiota Is Significantly Impacted by Feeding Mode and Associated With Growth Status in Exclusively Breastfed and Formula-Fed US Infants
Lillian M. Berman
University of Colorado Anschutz Medical Campus (US)
2 shared publications
- The Gut Microbiota Differ in Exclusively Breastfed and Formula-Fed United States Infants and are Associated with Growth Status
- Gut Microbiota Is Significantly Impacted by Feeding Mode and Associated With Growth Status in Exclusively Breastfed and Formula-Fed US Infants
Nancy F. Krebs
University of Colorado Anschutz Medical Campus (US)
2 shared publications
- The Gut Microbiota Differ in Exclusively Breastfed and Formula-Fed United States Infants and are Associated with Growth Status
- Gut Microbiota Is Significantly Impacted by Feeding Mode and Associated With Growth Status in Exclusively Breastfed and Formula-Fed US Infants
Minghua Tang
University of Colorado Anschutz Medical Campus (US)
2 shared publications
- The Gut Microbiota Differ in Exclusively Breastfed and Formula-Fed United States Infants and are Associated with Growth Status
- Gut Microbiota Is Significantly Impacted by Feeding Mode and Associated With Growth Status in Exclusively Breastfed and Formula-Fed US Infants
Xi Zhang
Baylor Medical Center at Garland (US)
1 shared publication
- In Esophageal Squamous Cells From Eosinophilic Esophagitis Patients, Th2 Cytokines Increase Eotaxin-3 Secretion Through Effects on Intracellular Calcium and a Non-Gastric Proton Pump
Yan Chang
The University of Texas at Arlington (US)
1 shared publication
- In Esophageal Squamous Cells From Eosinophilic Esophagitis Patients, Th2 Cytokines Increase Eotaxin-3 Secretion Through Effects on Intracellular Calcium and a Non-Gastric Proton Pump
Melissa R. Nelson
Baylor University Medical Center (US)
1 shared publication
- In Esophageal Squamous Cells From Eosinophilic Esophagitis Patients, Th2 Cytokines Increase Eotaxin-3 Secretion Through Effects on Intracellular Calcium and a Non-Gastric Proton Pump
Uthra Balaji
Baylor Scott & White Health (US)
1 shared publication
- In Esophageal Squamous Cells From Eosinophilic Esophagitis Patients, Th2 Cytokines Increase Eotaxin-3 Secretion Through Effects on Intracellular Calcium and a Non-Gastric Proton Pump
Jinghua Gu
Baylor Scott & White Health (US)
1 shared publication
- In Esophageal Squamous Cells From Eosinophilic Esophagitis Patients, Th2 Cytokines Increase Eotaxin-3 Secretion Through Effects on Intracellular Calcium and a Non-Gastric Proton Pump
Qiuyang Zhang
Baylor University Medical Center (US)
1 shared publication
- In Esophageal Squamous Cells From Eosinophilic Esophagitis Patients, Th2 Cytokines Increase Eotaxin-3 Secretion Through Effects on Intracellular Calcium and a Non-Gastric Proton Pump
Zui Pan
The University of Texas at Arlington (US)
1 shared publication
- In Esophageal Squamous Cells From Eosinophilic Esophagitis Patients, Th2 Cytokines Increase Eotaxin-3 Secretion Through Effects on Intracellular Calcium and a Non-Gastric Proton Pump
Stuart J. Spechler
Baylor University Medical Center (US)
1 shared publication
- In Esophageal Squamous Cells From Eosinophilic Esophagitis Patients, Th2 Cytokines Increase Eotaxin-3 Secretion Through Effects on Intracellular Calcium and a Non-Gastric Proton Pump
Similar Researchers
Based on overlapping research topics
Prateek Wali
University of Arkansas for Medical Sciences
Gut microbiota and health
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Annalyse Marie Kohley
University of Arkansas for Medical Sciences
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Renny S. Lan
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David Kawatu
University of Arkansas for Medical Sciences
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Arya Kapil
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Misty Virmani
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