Biography and Research Information
OverviewAI-generated summary
Farhana Taher Sumya's research investigates the intricate mechanisms of intracellular transport within the Golgi apparatus, with a particular focus on its role in glycosylation. Her work has examined the function of the COG complex, a key player in vesicular trafficking, and its impact on protein modification. Sumya has contributed to understanding how disruptions in this machinery can lead to congenital disorders of glycosylation (CDGs). Her publications detail the development of cellular models for studying these diseases and the proteomic characterization of cellular components involved in intra-Golgi transport. She has collaborated extensively with Vladimir Lupashin, Irina D. Pokrovskaya, Zinia D’Souza, and Walter S. Aragon‐Ramirez at the University of Arkansas for Medical Sciences on these research endeavors.
Metrics
- h-index: 4
- Publications: 9
- Citations: 74
Selected Publications
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COG Complex in Golgi Trafficking and Glycosylation (2026)
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Deep proteomic profiling of the intra-Golgi trafficking intermediates (2025)
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Comprehensive Proteomic Characterization of the Intra-Golgi Trafficking Intermediates (2024)
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Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking (2023)
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Rapid COG Depletion in Mammalian Cell by Auxin-Inducible Degradation System (2022)
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Acute COG complex inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra‐Golgi recycling vesicles (2022)
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Acute COG inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra-Golgi recycling vesicles (2022)
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Getting Sugar Coating Right! The Role of the Golgi Trafficking Machinery in Glycosylation (2021)
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Development and Initial Characterization of Cellular Models for COG Complex-Related CDG-II Diseases (2021)
Collaboration Network
Top Collaborators
Vladimir Lupashin
University of Arkansas for Medical Sciences
8 shared publications
In database
- Getting Sugar Coating Right! The Role of the Golgi Trafficking Machinery in Glycosylation
- Development and Initial Characterization of Cellular Models for COG Complex-Related CDG-II Diseases
- Acute COG complex inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra‐Golgi recycling vesicles
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
- Rapid COG Depletion in Mammalian Cell by Auxin-Inducible Degradation System
Showing 5 of 8 shared publications
Irina D. Pokrovskaya
University of Arkansas for Medical Sciences
4 shared publications
In database
- Development and Initial Characterization of Cellular Models for COG Complex-Related CDG-II Diseases
- Acute COG complex inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra‐Golgi recycling vesicles
- Rapid COG Depletion in Mammalian Cell by Auxin-Inducible Degradation System
- Acute COG inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra-Golgi recycling vesicles
Zinia D’Souza
University of Arkansas for Medical Sciences
3 shared publications
In database
- Getting Sugar Coating Right! The Role of the Golgi Trafficking Machinery in Glycosylation
- Acute COG complex inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra‐Golgi recycling vesicles
- Acute COG inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra-Golgi recycling vesicles
Walter S. Aragon‐Ramirez
University of Arkansas for Medical Sciences
2 shared publications
In database
- Comprehensive Proteomic Characterization of the Intra-Golgi Trafficking Intermediates
- Deep proteomic profiling of the intra-Golgi trafficking intermediates
Amrita Khakurel
University of Arkansas for Medical Sciences
1 shared publication
In database
- Getting Sugar Coating Right! The Role of the Golgi Trafficking Machinery in Glycosylation
Ruizhi Duan
Baylor College of Medicine (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Dana Marafi
Baylor College of Medicine (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Zhi‐Jie Xia
Sanford Burnham Prebys Medical Discovery Institute (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Bobby G. Ng
Sanford Burnham Prebys Medical Discovery Institute (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Reza Maroofian
Queen Mary University of London (GB)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Ahmed K. Saad
Baylor College of Medicine (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Haowei Du
Baylor College of Medicine (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Jawid M. Fatih
Baylor College of Medicine (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Jill V. Hunter
Baylor College of Medicine (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Hasnaa M. Elbendary
National Human Genome Research Institute (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
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