Match tier
Listed
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-10-05
Biography and Research Information
OverviewAI-generated summary
Farhana Taher Sumya's research focuses on the molecular mechanisms underlying glycosylation, a critical post-translational modification of proteins. Her work investigates the role of the Golgi apparatus trafficking machinery, specifically the COG complex, in ensuring proper protein modification and transport. Sumya has studied the impact of mutations within COG complex genes, such as COG3, on cellular function and has characterized these as causes of congenital disorders of glycosylation (CDGs). Her publications detail the development and use of cellular models to study these diseases, employing techniques like auxin-inducible degradation systems for rapid COG depletion. Sumya has also contributed to proteomic analyses of intra-Golgi trafficking intermediates. She has a significant collaborative network at the University of Arkansas for Medical Sciences, with nine shared publications with Vladimir Lupashin.
Metrics
- h-index: 5
- Publications: 10
- Citations: 85
Positions
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Post Doctoral Fellow 2025–presentUniversity of California, San Francisco Medicine ORCID
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Post Doctoral Fellow publications 2021–2026University of Arkansas for Medical Sciences ORCID
Selected Publications
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Golgi CATCHR complexes function as organizing hubs for vesicle tethering and fusion (2026)
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COG Complex in Golgi Trafficking and Glycosylation (2026)
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Deep proteomic profiling of the intra-Golgi trafficking intermediates (2025)
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Comprehensive Proteomic Characterization of the Intra-Golgi Trafficking Intermediates (2024)
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Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking (2023)
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Rapid COG Depletion in Mammalian Cell by Auxin-Inducible Degradation System (2022)
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Acute COG complex inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra‐Golgi recycling vesicles (2022)
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Acute COG inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra-Golgi recycling vesicles (2022)
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Getting Sugar Coating Right! The Role of the Golgi Trafficking Machinery in Glycosylation (2021)
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Development and Initial Characterization of Cellular Models for COG Complex-Related CDG-II Diseases (2021)
Collaboration Network
Top Collaborators
Vladimir Lupashin
University of Arkansas for Medical Sciences
9 shared publications
In database
- Getting Sugar Coating Right! The Role of the Golgi Trafficking Machinery in Glycosylation
- Development and Initial Characterization of Cellular Models for COG Complex-Related CDG-II Diseases
- Acute COG complex inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra‐Golgi recycling vesicles
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
- Rapid COG Depletion in Mammalian Cell by Auxin-Inducible Degradation System
Showing 5 of 9 shared publications
Irina D. Pokrovskaya
University of Arkansas for Medical Sciences
5 shared publications
In database
- Development and Initial Characterization of Cellular Models for COG Complex-Related CDG-II Diseases
- Acute COG complex inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra‐Golgi recycling vesicles
- Rapid COG Depletion in Mammalian Cell by Auxin-Inducible Degradation System
- Acute COG inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra-Golgi recycling vesicles
- Golgi CATCHR complexes function as organizing hubs for vesicle tethering and fusion
Zinia D’Souza
University of Arkansas for Medical Sciences
3 shared publications
In database
- Getting Sugar Coating Right! The Role of the Golgi Trafficking Machinery in Glycosylation
- Acute COG complex inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra‐Golgi recycling vesicles
- Acute COG inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra-Golgi recycling vesicles
Walter S. Aragon‐Ramirez
University of Arkansas for Medical Sciences
3 shared publications
In database
- Deep proteomic profiling of the intra-Golgi trafficking intermediates
- Comprehensive Proteomic Characterization of the Intra-Golgi Trafficking Intermediates
- Golgi CATCHR complexes function as organizing hubs for vesicle tethering and fusion
Amrita Khakurel
University of Arkansas for Medical Sciences
2 shared publications
In database
- Getting Sugar Coating Right! The Role of the Golgi Trafficking Machinery in Glycosylation
- Golgi CATCHR complexes function as organizing hubs for vesicle tethering and fusion
Ruizhi Duan
Baylor College of Medicine (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Dana Marafi
Baylor College of Medicine (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Zhi‐Jie Xia
Sanford Burnham Prebys Medical Discovery Institute (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Bobby G. Ng
Sanford Burnham Prebys Medical Discovery Institute (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Reza Maroofian
Queen Mary University of London (GB)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Ahmed K. Saad
Baylor College of Medicine (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Haowei Du
Baylor College of Medicine (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Jawid M. Fatih
Baylor College of Medicine (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Jill V. Hunter
Baylor College of Medicine (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
Hasnaa M. Elbendary
National Human Genome Research Institute (US)
1 shared publication
- Biallelic missense variants in <scp><i>COG3</i></scp> cause a congenital disorder of glycosylation with impairment of retrograde vesicular trafficking
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