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Presence Current · Arkansas
Last published 2026
Sources OpenAlex · ORCID
Refreshed 2026-08-18

Fumou Sun

Assistant Professor

Faculty Researcher

18 h-index 67 pubs 860 cited

  • Multiple Myeloma
  • Humans
  • Animals
  • Mice
  • Cell Line, Tumor
  • Tumor Microenvironment
  • Xenograft Model Antitumor Assays
  • Female
  • Monoclonal Gammopathy of Undetermined Significance
  • Mice, Inbred BALB C
  • T-Lymphocytes
  • CD24 Antigen
  • Killer Cells, Natural
  • Mice, Nude
  • NK Cell Lectin-Like Receptor Subfamily K

Biography and Research Information

OverviewAI-generated summary

Fumou Sun investigates multiple myeloma, focusing on cellular mechanisms and potential therapeutic strategies. His research explores the role of specific genes and proteins, such as FOXM1 and NEK2, in regulating cancer cell metabolism, energy production, and immune suppression within the tumor microenvironment. Sun has also examined the efficacy of novel immunotherapies, including bispecific CAR-T cells targeting BCMA and CD24, and fusion antibodies that enhance immunotherapy effects by blocking PD-1. Additionally, his work has investigated the impact of certain compounds, like Baicalin, on autophagy and graft-versus-host disease, as well as the mechanisms by which CST6 suppresses osteolytic bone disease in multiple myeloma.

Sun's scholarship metrics include an h-index of 18, with 67 total publications and 848 total citations. He has a significant collaborative network at the University of Arkansas for Medical Sciences, with numerous shared publications with John D. Shaughnessy, Guido Tricot, David E. Mery, and Dongzheng Gai.

Metrics

  • h-index: 18
  • Publications: 67
  • Citations: 860

Selected Publications

  • Polyclonal plasma cell (PolyPC) signature as a key indicator for predicting the progression of MGUS to multiple myeloma (2025)
    Cancer Biomarkers 1 citation DOI OpenAlex
  • Cystatin M/E Ameliorates Multiple Myeloma-Induced Hyper Osteolytic Bone Resorption (2025)
    Cancers 1 citation DOI OpenAlex
  • Multi-Omics Reveal Immune Microenvironment Alterations in Multiple Myeloma and Its Precursor Stages (2024)
    Blood 1 citation DOI OpenAlex
  • Of Seven Molecular Subtypes in MGUS the Low Bone Disease (LB) Subtype Exhibits Resistance to Multiple Myeloma Progression (2024)
    Blood DOI OpenAlex
  • Multi-omics reveal immune microenvironment alterations in multiple myeloma and its precursor stages (2024)
    Blood Cancer Journal 35 citations DOI OpenAlex
  • Cystatin M/E ameliorates bone resorption through increasing osteoclastic cell estrogen influx (2024)
    Research Square 1 citation DOI OpenAlex
  • A Risk Stratification System in Myeloma Patients with Autologous Stem Cell Transplantation (2024)
    Cancers 4 citations DOI OpenAlex
  • Enhancing prognostic power in multiple myeloma using a plasma cell signature derived from single-cell RNA sequencing (2024)
    Blood Cancer Journal 7 citations DOI OpenAlex
  • Prognostic value of ferritin in ASCT MM patients: integration with GEP models and ISS series systems (2024)
    Blood Cancer Journal 7 citations DOI OpenAlex
  • Bispecific BCMA/CD24 CAR-T cells control multiple myeloma growth (2024)
    Nature Communications 46 citations DOI OpenAlex
  • A 5-Stage System for ASCT Multiple Myeloma (2023)
    Blood DOI OpenAlex
  • Prognostic Value of Ferritin in ASCT MM Patients: Integration with GEP Models and ISS Series Systems (2023)
    Blood 1 citation DOI OpenAlex
  • BCMA- and CST6-specific CAR T cells lyse multiple myeloma cells and suppress murine osteolytic lesions (2023)
    Journal of Clinical Investigation 7 citations DOI OpenAlex
  • High NEK2 expression in myeloid progenitors suppresses T cell immunity in multiple myeloma (2023)
    Cell Reports Medicine 17 citations DOI OpenAlex
  • P-066 BCMA-CAR-T cells with synthetic circuits of CST6 lyse tumor cells and suppress osteolytic lesions in multiple myeloma (2023)
    Clinical Lymphoma Myeloma & Leukemia DOI OpenAlex

View all publications on OpenAlex →

Collaboration Network

100 Collaborators 23 Institutions 3 Countries

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