J. Hanks Data-verified
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Biography and Research Information
OverviewAI-generated summary
J. Hanks' research investigates the molecular mechanisms underlying liver injury, with a recent focus on non-alcoholic steatohepatitis (NASH)-like conditions in preclinical models. Their work has explored the use of chronic high-fat and high-sucrose diets in mice to induce severe liver injury, as detailed in a 2024 publication. Further research has focused on developing signature gene expression models for the quantitative evaluation of NASH-like liver injury, as presented in a 2025 publication. Hanks has a publication record of 26 articles with 970 citations and an h-index of 15. Key collaborators include Volodymyr Tryndyak, Frederick A. Beland, Rose Willett, and Igor P. Pogribny, all affiliated with the National Center for Toxicological Research, with whom Hanks has co-authored multiple publications.
Metrics
- h-index: 15
- Publications: 26
- Citations: 970
Selected Publications
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Signature gene expression model for quantitative evaluation of MASH-like liver injury in mice (2025)
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- A preclinical model of severe NASH-like liver injury by chronic administration of a high-fat and high-sucrose diet in mice
- Signature gene expression model for quantitative evaluation of MASH-like liver injury in mice
- A preclinical model of severe NASH-like liver injury by chronic administration of a high-fat and high-sucrose diet in mice
- Signature gene expression model for quantitative evaluation of MASH-like liver injury in mice
- A preclinical model of severe NASH-like liver injury by chronic administration of a high-fat and high-sucrose diet in mice
- Signature gene expression model for quantitative evaluation of MASH-like liver injury in mice
- A preclinical model of severe NASH-like liver injury by chronic administration of a high-fat and high-sucrose diet in mice
- Signature gene expression model for quantitative evaluation of MASH-like liver injury in mice
- A preclinical model of severe NASH-like liver injury by chronic administration of a high-fat and high-sucrose diet in mice
- Signature gene expression model for quantitative evaluation of MASH-like liver injury in mice
- A preclinical model of severe NASH-like liver injury by chronic administration of a high-fat and high-sucrose diet in mice
- Signature gene expression model for quantitative evaluation of MASH-like liver injury in mice
- A preclinical model of severe NASH-like liver injury by chronic administration of a high-fat and high-sucrose diet in mice
- A preclinical model of severe NASH-like liver injury by chronic administration of a high-fat and high-sucrose diet in mice
- A preclinical model of severe NASH-like liver injury by chronic administration of a high-fat and high-sucrose diet in mice
- A preclinical model of severe NASH-like liver injury by chronic administration of a high-fat and high-sucrose diet in mice
- Signature gene expression model for quantitative evaluation of MASH-like liver injury in mice
- Signature gene expression model for quantitative evaluation of MASH-like liver injury in mice
- Signature gene expression model for quantitative evaluation of MASH-like liver injury in mice
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