Jyotshna Kanungo
Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.
Senior (Principal) Investigator
Also affiliated: National Institutes of Health (2006–2008); United States Food and Drug Administration (2013–2026); Augusta University (1993–1996); Augusta University Health (1996); National Institute of Neurological Disorders and Stroke (2006–2018); Bentham Science Publishers (China) (2014); Bentham Science Publishers (United Arab Emirates) (2014)
Faculty Researcher
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Jyotshna Kanungo's research focuses on understanding the neurodevelopmental and neurotoxic effects of environmental agents, utilizing zebrafish as a model organism. Her work investigates how substances like inorganic arsenic and ketamine impact neural development and function, with implications for human health conditions including autism and depression. Kanungo has explored the molecular mechanisms underlying arsenic-induced neurotoxicity, examining its effects on dopaminergic and motor neurons, and its potential involvement with the Sonic hedgehog pathway. She has also studied the antidepressant actions of ketamine, with a focus on the role of L-type calcium channels and gene expression changes in zebrafish embryos.
Her research employs advanced techniques for neurotoxicity assessment, such as combining tissue clearing with Fluoro-Jade C labeling. Kanungo's publication record includes work on screening tools for evaluating the neurotoxic potential of botanicals. Her scholarship metrics include an h-index of 19, with 52 total publications and 996 total citations. She collaborates with researchers at the National Center for Toxicological Research, including Qiang Gu, Nathan C. Twaddle, Frederick A. Beland, and Camila Silva.
Metrics
- h-index: 19
- Publications: 52
- Citations: 1,003
Selected Publications
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A new method for visualizing cerebrovascular vessels (2026)
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N-Acetylcysteine Prevents Arsenic-Induced Apoptosis but Not Supernumerary Motor Neuron Development in Zebrafish Embryos: Assessment of Protein Carbonylation and the p53 Pathway (2026)
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Special Issue “Zebrafish: A Model Organism for Human Health and Disease” (2025)
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Acetyl L‐Carnitine Protects Zebrafish Embryos From Verapamil and Inorganic Arsenic‐Induced Cardiotoxicity and Developmental Toxicity With No Effect on Supernumerary Motor Neuron Development (2025)
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Screening tools to evaluate the neurotoxic potential of botanicals: building a strategy to assess safety (2024)
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Neurogenic Effects of Inorganic Arsenic and Cdk5 Knockdown in Zebrafish Embryos: A Perspective on Modeling Autism (2024)
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Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish (2023)
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Combining tissue clearing and Fluoro-Jade C labeling for neurotoxicity assessments (2023)
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Use of zebrafish embryos to study molecular and cellular neurotoxic effects of drugs: A focus on signaling and ketamine (2022)
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Effect of ketamine on gene expression in zebrafish embryos (2021)
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Effects of acetyl L‐carnitine on zebrafish embryos: Phenotypic and gene expression studies (2020)
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N-acetylcysteine prevents verapamil-induced cardiotoxicity with no effect on the noradrenergic arch-associated neurons in zebrafish (2020)
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Cell & Developmental Biology (2019)
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Nifedipine toxicity is exacerbated by acetyl <scp>l</scp>‐carnitine but alleviated by low‐dose ketamine in zebrafish in vivo (2019)
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Downregulation of 14-3-3 Proteins in Alzheimer’s Disease (2019)
Collaboration Network
Top Collaborators
- Effect of ketamine on gene expression in zebrafish embryos
- Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish
- Neurogenic Effects of Inorganic Arsenic and Cdk5 Knockdown in Zebrafish Embryos: A Perspective on Modeling Autism
- Combining tissue clearing and Fluoro-Jade C labeling for neurotoxicity assessments
- Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish
- Acetyl L‐Carnitine Protects Zebrafish Embryos From Verapamil and Inorganic Arsenic‐Induced Cardiotoxicity and Developmental Toxicity With No Effect on Supernumerary Motor Neuron Development
- Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish
- Acetyl L‐Carnitine Protects Zebrafish Embryos From Verapamil and Inorganic Arsenic‐Induced Cardiotoxicity and Developmental Toxicity With No Effect on Supernumerary Motor Neuron Development
- Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish
- Acetyl L‐Carnitine Protects Zebrafish Embryos From Verapamil and Inorganic Arsenic‐Induced Cardiotoxicity and Developmental Toxicity With No Effect on Supernumerary Motor Neuron Development
- Combining tissue clearing and Fluoro-Jade C labeling for neurotoxicity assessments
- Combining tissue clearing and Fluoro-Jade C labeling for neurotoxicity assessments
- Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish
- Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish
- Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish
- Gene expression analyses reveal potential mechanism of inorganic arsenic‐induced apoptosis in zebrafish
- Screening tools to evaluate the neurotoxic potential of botanicals: building a strategy to assess safety
- Screening tools to evaluate the neurotoxic potential of botanicals: building a strategy to assess safety
- Screening tools to evaluate the neurotoxic potential of botanicals: building a strategy to assess safety
- Screening tools to evaluate the neurotoxic potential of botanicals: building a strategy to assess safety
- Screening tools to evaluate the neurotoxic potential of botanicals: building a strategy to assess safety
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