Biography and Research Information
OverviewAI-generated summary
Katie Pierce's research focuses on understanding infectious diseases and the immune system's response. Her work includes investigating suitable laboratory strains for studying *Coxiella burnetii*, a bacterium that causes Q fever, and exploring the development and efficacy of vaccines against this disease. Pierce also examines the role of alveolar macrophages in trained immunity and their contribution to protection against *Mycobacterium tuberculosis*. She has contributed to publications on designing effective questionnaires for research studies. Pierce is affiliated with the University of Arkansas for Medical Sciences, where she collaborates with researchers such as Daniel E. Voth and Lin-Xi Li.
Metrics
- h-index: 3
- Publications: 8
- Citations: 174
Selected Publications
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<i>Coxiella burnetii</i> Nine Mile II Δ <i>cbu0533</i> as a suitable laboratory replacement strain (2025)
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Control of human Q fever by vaccination: the journey to Q-VAX and beyond (2025)
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Metabolism and ontogeny of alveolar macrophages contribute to peripheral trained immunity and confer protection against <i>Mycobacterium tuberculosis</i> (2023)
Collaboration Network
Top Collaborators
- Metabolism and ontogeny of alveolar macrophages contribute to peripheral trained immunity and confer protection against <i>Mycobacterium tuberculosis</i>
- Metabolism and ontogeny of alveolar macrophages contribute to peripheral trained immunity and confer protection against <i>Mycobacterium tuberculosis</i>
- Metabolism and ontogeny of alveolar macrophages contribute to peripheral trained immunity and confer protection against <i>Mycobacterium tuberculosis</i>
- Metabolism and ontogeny of alveolar macrophages contribute to peripheral trained immunity and confer protection against <i>Mycobacterium tuberculosis</i>
- Metabolism and ontogeny of alveolar macrophages contribute to peripheral trained immunity and confer protection against <i>Mycobacterium tuberculosis</i>
- Metabolism and ontogeny of alveolar macrophages contribute to peripheral trained immunity and confer protection against <i>Mycobacterium tuberculosis</i>
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