Match tier Listed
Presence Current · Arkansas
Last published 2025
Sources OpenAlex · ORCID
Refreshed 2026-08-15

J. Tucker Andrews

Research Technologist

Also affiliated: Conway School of Landscape Design (2021); L-3 Communications (United States) (2024)

Unknown Researcher

4 h-index 8 pubs 99 cited

  • Hypertension
  • Animals
  • Mice
  • CD8-Positive T-Lymphocytes
  • Disease Models, Animal
  • Humans
  • Lung
  • Mycobacterium tuberculosis
  • Neutrophils
  • Neutrophil Activation
  • Sodium
  • Sodium Chloride, Dietary
  • Desoxycorticosterone Acetate
  • Bacterial Infections
  • Macrophages, Alveolar

Biography and Research Information

OverviewAI-generated summary

J. Tucker Andrews' research focuses on the immunological mechanisms underlying hypertension and host defense against bacterial infections. His work investigates the role of T-cell interactions, specifically CD8+ T-lymphocytes and their interaction with IFNγ-PDL1 pathways, in the development of salt-sensitive hypertension. Andrews also studies the metabolic regulation of immune cells, such as neutrophils and alveolar macrophages, in the context of Mycobacterium tuberculosis infection. His recent publications explore how these metabolic activities influence immune cell function, contributing to either disease progression or host protection. Collaborations with researchers at the University of Arkansas for Medical Sciences, including Lin-Xi Li and Lu Huang, have resulted in multiple shared publications, indicating a network focused on advancing understanding in these areas of immunology and disease.

Metrics

  • h-index: 4
  • Publications: 8
  • Citations: 99

Selected Publications

  • β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395 (2025)
    The Journal of Immunology DOI OpenAlex
  • Fatty acid oxidation regulates alveolar macrophage homeostasis and resistance to infection 4350 (2025)
    The Journal of Immunology DOI OpenAlex
  • Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis (2024)
    Mucosal Immunology 35 citations DOI OpenAlex
  • <i>P2X7</i> -Mediated Antigen-Independent Activation of CD8 <sup>+</sup> T Cells Promotes Salt-Sensitive Hypertension (2024)
    Hypertension 8 citations DOI OpenAlex
  • Metabolism and ontogeny of alveolar macrophages contribute to peripheral trained immunity and confer protection against <i>Mycobacterium tuberculosis</i> (2023)
    The Journal of Immunology DOI OpenAlex
  • Breathe In, Breathe Out: Metabolic Regulation of Lung Macrophages in Host Defense Against Bacterial Infection (2022)
    Frontiers in Cellular and Infection Microbiology 8 citations DOI OpenAlex
  • The IFNγ-PDL1 Pathway Enhances CD8T-DCT Interaction to Promote Hypertension (2022)
    Circulation Research 42 citations DOI OpenAlex
  • Formation of Supported Lipid Bilayers (SLBs) from Buffers Containing Low Concentrations of Group I Chloride Salts (2021)
    Langmuir 4 citations DOI OpenAlex

View all publications on OpenAlex →

Collaboration Network

30 Collaborators 13 Institutions 2 Countries

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