Biography and Research Information
OverviewAI-generated summary
J. Tucker Andrews' research focuses on the immunological mechanisms underlying hypertension and host defense against bacterial infections. His work investigates the role of T-cell interactions, specifically CD8+ T-lymphocytes and their interaction with IFNγ-PDL1 pathways, in the development of salt-sensitive hypertension. Andrews also studies the metabolic regulation of immune cells, such as neutrophils and alveolar macrophages, in the context of Mycobacterium tuberculosis infection. His recent publications explore how these metabolic activities influence immune cell function, contributing to either disease progression or host protection. Collaborations with researchers at the University of Arkansas for Medical Sciences, including Lin-Xi Li and Lu Huang, have resulted in multiple shared publications, indicating a network focused on advancing understanding in these areas of immunology and disease.
Metrics
- h-index: 4
- Publications: 8
- Citations: 90
Selected Publications
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β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395 (2025)
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Fatty acid oxidation regulates alveolar macrophage homeostasis and resistance to infection 4350 (2025)
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Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis (2024)
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Metabolism and ontogeny of alveolar macrophages contribute to peripheral trained immunity and confer protection against <i>Mycobacterium tuberculosis</i> (2023)
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Breathe In, Breathe Out: Metabolic Regulation of Lung Macrophages in Host Defense Against Bacterial Infection (2022)
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Formation of Supported Lipid Bilayers (SLBs) from Buffers Containing Low Concentrations of Group I Chloride Salts (2021)
Collaboration Network
Top Collaborators
Lu Huang
University of Arkansas for Medical Sciences
7 shared publications
In database
- The IFNγ-PDL1 Pathway Enhances CD8T-DCT Interaction to Promote Hypertension
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- <i>P2X7</i> -Mediated Antigen-Independent Activation of CD8 <sup>+</sup> T Cells Promotes Salt-Sensitive Hypertension
- Breathe In, Breathe Out: Metabolic Regulation of Lung Macrophages in Host Defense Against Bacterial Infection
- Metabolism and ontogeny of alveolar macrophages contribute to peripheral trained immunity and confer protection against <i>Mycobacterium tuberculosis</i>
Showing 5 of 7 shared publications
Lin‐Xi Li
University of Arkansas for Medical Sciences
5 shared publications
In database
- The IFNγ-PDL1 Pathway Enhances CD8T-DCT Interaction to Promote Hypertension
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- <i>P2X7</i> -Mediated Antigen-Independent Activation of CD8 <sup>+</sup> T Cells Promotes Salt-Sensitive Hypertension
- Metabolism and ontogeny of alveolar macrophages contribute to peripheral trained immunity and confer protection against <i>Mycobacterium tuberculosis</i>
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
Shengyu Mu
University of Arkansas for Medical Sciences
3 shared publications
In database
- The IFNγ-PDL1 Pathway Enhances CD8T-DCT Interaction to Promote Hypertension
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- <i>P2X7</i> -Mediated Antigen-Independent Activation of CD8 <sup>+</sup> T Cells Promotes Salt-Sensitive Hypertension
Ananya Ranaraja
University of Arkansas for Medical Sciences
3 shared publications
In database
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- Fatty acid oxidation regulates alveolar macrophage homeostasis and resistance to infection 4350
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
Lance Benson
2 shared publications
- The IFNγ-PDL1 Pathway Enhances CD8T-DCT Interaction to Promote Hypertension
- <i>P2X7</i> -Mediated Antigen-Independent Activation of CD8 <sup>+</sup> T Cells Promotes Salt-Sensitive Hypertension
Yunping Guo
2 shared publications
In database
- The IFNγ-PDL1 Pathway Enhances CD8T-DCT Interaction to Promote Hypertension
- <i>P2X7</i> -Mediated Antigen-Independent Activation of CD8 <sup>+</sup> T Cells Promotes Salt-Sensitive Hypertension
Katherine Deck
2 shared publications
In database
- The IFNγ-PDL1 Pathway Enhances CD8T-DCT Interaction to Promote Hypertension
- <i>P2X7</i> -Mediated Antigen-Independent Activation of CD8 <sup>+</sup> T Cells Promotes Salt-Sensitive Hypertension
Christoph Mora
2 shared publications
In database
- The IFNγ-PDL1 Pathway Enhances CD8T-DCT Interaction to Promote Hypertension
- <i>P2X7</i> -Mediated Antigen-Independent Activation of CD8 <sup>+</sup> T Cells Promotes Salt-Sensitive Hypertension
Zhiqiang Qin
University of Arkansas for Medical Sciences (US)
2 shared publications
- The IFNγ-PDL1 Pathway Enhances CD8T-DCT Interaction to Promote Hypertension
- <i>P2X7</i> -Mediated Antigen-Independent Activation of CD8 <sup>+</sup> T Cells Promotes Salt-Sensitive Hypertension
Robert S. Hoover
Tulane University (US)
2 shared publications
- The IFNγ-PDL1 Pathway Enhances CD8T-DCT Interaction to Promote Hypertension
- <i>P2X7</i> -Mediated Antigen-Independent Activation of CD8 <sup>+</sup> T Cells Promotes Salt-Sensitive Hypertension
Daniel E. Voth
University of Arkansas for Medical Sciences
2 shared publications
In database
- Breathe In, Breathe Out: Metabolic Regulation of Lung Macrophages in Host Defense Against Bacterial Infection
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
Stanley Ching‐Cheng Huang
2 shared publications
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- Breathe In, Breathe Out: Metabolic Regulation of Lung Macrophages in Host Defense Against Bacterial Infection
Fischer Huey
University of Arkansas for Medical Sciences (US)
2 shared publications
- Metabolism and ontogeny of alveolar macrophages contribute to peripheral trained immunity and confer protection against <i>Mycobacterium tuberculosis</i>
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
Yasir H. Ahmed-Braimah
Syracuse University (US)
2 shared publications
- Metabolism and ontogeny of alveolar macrophages contribute to peripheral trained immunity and confer protection against <i>Mycobacterium tuberculosis</i>
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
Zijing Zhang
University of Arkansas for Medical Sciences
2 shared publications
In database
- Metabolically active neutrophils represent a permissive niche for Mycobacterium tuberculosis
- β-glucan-induced monocyte-derived alveolar macrophages confer protection against Mycobacterium tuberculosis 4395
Similar Researchers
Based on overlapping research topics
Katherine Deck
University of Arkansas for Medical Sciences
Immune Response and Inflammation
Cardiovascular Function and Risk Factors
Blood Pressure and Hypertension Studies
Pamela I. Rogers
University of Arkansas for Medical Sciences
Immune Response and Inflammation
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Lin‐Xi Li
University of Arkansas for Medical Sciences
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Changhoon Oh
University of Arkansas for Medical Sciences
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Immune Cell Function and Interaction
Shengyu Mu
University of Arkansas for Medical Sciences
Immune Response and Inflammation
Cardiovascular Function and Risk Factors
Blood Pressure and Hypertension Studies
Shiv Kumar Agarwal
University of Arkansas for Medical Sciences
Immune Response and Inflammation
Immune Cell Function and Interaction
T-cell and B-cell Immunology