Match tier Institution-verified
Presence Current · Arkansas
Last published 2021
Sources Institutional record
Refreshed 2026-10-01
Kristen M. Sterba profile photo

Kristen M. Sterba

Sourced from institutional research profiles (UAMS TRI or ARA).

Associate Provost for Students and Admin

Academic Affairs, College of Medicine

4 h-index 4 pubs 150 cited

  • Gene Expression Regulation, Bacterial
  • Humans
  • Bacterial Proteins
  • Base Sequence
  • DNA-Binding Proteins
  • DNA, Bacterial
  • Staphylococcus aureus
  • Adhesins, Bacterial
  • Electrophoretic Mobility Shift Assay
  • Academic Success
  • Binding Sites
  • Enhancer Elements, Genetic
  • Ligands
  • Oligonucleotides
  • Promoter Regions, Genetic

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Biography and Research Information

OverviewAI-generated summary

Kristen M. Sterba's research has focused on the molecular mechanisms of bacterial gene regulation, particularly within *Staphylococcus aureus*. Her work has investigated the binding sites and regulatory functions of the SarA protein, exploring its responsiveness to environmental factors such as redox potential and pH. Sterba has also examined the role of SarA in bacteriophage integration and excision processes. Additionally, her research has touched upon cellular responses to chemotherapy, specifically studying the impact of c-Jun overexpression on vinblastine resistance in MCF7 cells by influencing apoptosis and senescence pathways. More recently, Sterba's scholarship has extended to evaluating the outcomes of Ph.D. students from underrepresented racial groups in biomedical sciences, analyzing performance measures within programs like the UAMS IMSD Program.

Metrics

  • h-index: 4
  • Publications: 4
  • Citations: 150

Selected Publications

  • Performance measures of racially underrepresented Ph.D. students in biomedical sciences: The UAMS IMSD Program Outcomes (2021)
    PLoS ONE 4 citations DOI OpenAlex
  • Staphylococcus aureus SarA is a regulatory protein responsive to redox and pH that can support bacteriophage lambda integrase‐mediated excision/recombination (2009)
    Molecular Microbiology 57 citations DOI OpenAlex
  • Inducible overexpression of c-Jun in MCF7 cells causes resistance to vinblastine via inhibition of drug-induced apoptosis and senescence at a step subsequent to mitotic arrest (2006)
    Biochemical Pharmacology 22 citations DOI OpenAlex
  • Characterization of Staphylococcus aureus SarA Binding Sites (2003)
    Journal of Bacteriology 75 citations DOI OpenAlex

Grants & Funding

As listed on this researcher's institutional profile.

  • The University of Arkansas for Medical Sciences Initiative for Maximizing Student Development Program NIH Co-Investigator
  • COVID-19 Higher Education Emergency Relief Fund-Institutional Portion University of Arkansas for Medical Sciences US Department of Education Principal Investigator
  • UAMS Summer Undergraduate Research Program to Increase Diversity in Research NIH Co-Investigator

Collaboration Network

18 Collaborators 6 Institutions 1 Country

Top Collaborators

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