Match tier
Likely match
Presence
Current · Arkansas
Last published
2025
Sources
OpenAlex · ORCID
Refreshed
2026-08-08
M
Mohit Bansal
This is a likely match — the affiliation was inferred from OpenAlex, ORCID, and web sources but has not been fully confirmed. Treat with appropriate caution.
Research Scientist
Also affiliated: Agriculture and Agri-Food Canada (2011); Guru Angad Dev Veterinary and Animal Sciences University (2017); University of Alabama at Birmingham (2024–2025); Public Works Department Buildings and Roads (2017); Summerland Research and Development Centre (2011); Mississippi State University (2018–2021)
Staff Researcher
13 h-index
40 pubs
483 cited
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Mohit Bansal's research investigates molecular mechanisms underlying cellular stress, proliferation, and adaptation in both cancer development and animal health. His work has explored how MYC drives mRNA pseudouridylation, a process critical for mitigating proliferation-induced cellular stress in cancer. Bansal has also studied the role of DKC1-mediated pseudouridylation of rRNA in sustaining translation and metabolic adaptation. In the realm of animal health, his research has focused on poultry diseases, examining how specific bile acids and vaccines using *Clostridium perfringens* sporulation proteins can reduce necrotic enteritis. He has also investigated the modulation of chicken macrophage proteins by sodium butyrate for *Salmonella Enteritidis* invasion and the impact of microbiota on *Campylobacter jejuni* chicken colonization. His scholarship metrics include an h-index of 12, with 40 total publications and 464 total citations.
Metrics
- h-index: 13
- Publications: 40
- Citations: 483
Selected Publications
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The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid (2025)
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Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens (2022)
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Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization (2021)
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Specific Secondary Bile Acids Control Chicken Necrotic Enteritis (2021)
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Sodium butyrate modulates chicken macrophage proteins essential for Salmonella Enteritidis invasion (2021)
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Microbiota attenuates chicken transmission-exacerbated campylobacteriosis in Il10−/− mice (2020)
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Sodium Butyrate Reduces Salmonella Enteritidis Infection of Chicken Enterocytes and Expression of Inflammatory Host Genes in vitro (2020)
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A secondary bile acid from microbiota metabolism attenuates ileitis and bile acid reduction in subclinical necrotic enteritis in chickens (2020)
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Microbial metabolite deoxycholic acid controls Clostridium perfringens-induced chicken necrotic enteritis through attenuating inflammatory cyclooxygenase signaling (2019)
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Microbial metabolite deoxycholic acid shapes microbiota against Campylobacter jejuni chicken colonization (2019)
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Microbial metabolite deoxycholic acid shapes microbiota against <i>Campylobacter jejuni</i> chicken colonization (2019)
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Microbial metabolite deoxycholic acid controls <i>Clostridium perfringens</i> -induced chicken necrotic enteritis through attenuating cyclooxygenase signaling (2018)
Collaboration Network
Top Collaborators
Xiaolun Sun
University of Arkansas at Fayetteville
5 shared publications
In database
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Sodium butyrate modulates chicken macrophage proteins essential for Salmonella Enteritidis invasion
- Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens
- Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
Tahrir Alenezi
University of Arkansas at Fayetteville
4 shared publications
In database
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens
- Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
Ying Fu
University of Arkansas at Fayetteville
4 shared publications
In database
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens
- Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
Ayidh Almansour
University of Arkansas at Fayetteville (US)
4 shared publications
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens
- Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
Anamika Gupta
University of Arkansas at Fayetteville
3 shared publications
In database
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Sodium butyrate modulates chicken macrophage proteins essential for Salmonella Enteritidis invasion
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
Rohana Liyanage
University of Arkansas at Fayetteville
3 shared publications
In database
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Sodium butyrate modulates chicken macrophage proteins essential for Salmonella Enteritidis invasion
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
Abhinav Upadhyay
University of Connecticut
1 shared publication
In database
- Sodium butyrate modulates chicken macrophage proteins essential for Salmonella Enteritidis invasion
N.C. Rath
United States Department of Agriculture
1 shared publication
In database
- Sodium butyrate modulates chicken macrophage proteins essential for Salmonella Enteritidis invasion
Annie M. Donoghue
United States Department of Agriculture (US)
1 shared publication
- Sodium butyrate modulates chicken macrophage proteins essential for Salmonella Enteritidis invasion
Hong Wang
University of Arkansas at Fayetteville (US)
1 shared publication
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
Danielle Graham
University of Arkansas at Fayetteville
1 shared publication
In database
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
Billy M. Hargis
University of Arkansas at Fayetteville
1 shared publication
In database
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
Bilal Alrubaye
University of Arkansas at Fayetteville
1 shared publication
In database
- Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization
Hong Wang
University of Arkansas at Fayetteville (US)
1 shared publication
- Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization
Hong Wang
University of Arkansas at Fayetteville (US)
1 shared publication
- Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens
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