Mohit Bansal

This is a likely match — the affiliation was inferred from OpenAlex, ORCID, and web sources but has not been fully confirmed. Treat with appropriate caution.

Research Scientist

University of Arkansas

Also affiliated: Agriculture and Agri-Food Canada (2011); Guru Angad Dev Veterinary and Animal Sciences University (2017); University of Alabama at Birmingham (2024–2025); Public Works Department Buildings and Roads (2017); Summerland Research and Development Centre (2011); Mississippi State University (2018–2021)

13 h-index 40 pubs 488 cited

Research Areas

Biomedical Subjects

Links

ORCID Research Group

OverviewAI-generated summary

Mohit Bansal's research focuses on host-pathogen interactions and the role of microbial metabolites in disease. His work has investigated the impact of specific secondary bile acids, such as deoxycholic acid, in controlling chicken necrotic enteritis and Campylobacter jejuni colonization. Bansal has also studied the mechanisms by which microbial metabolites attenuate inflammation and disease progression in poultry. His research has explored the response of Listeria monocytogenes to oxidative stress and the formation of Salmonella biofilms on food-contact surfaces. Additionally, Bansal has examined the seroepidemiology of hepatitis E virus in swine and its occupational exposure risks to humans. He has authored 40 publications with 488 citations and an h-index of 13. Key collaborators at the University of Arkansas at Fayetteville include Xiaolun Sun, Tahrir Alenezi, Rohana Liyanage, and Anamika Gupta.

Metrics

h-index: 13
Publications: 40
Citations: 488

Positions

Research Scientist 2021–present

University of Alabama at Birmingham Pathology ORCID
Research Scientist publications 2018–2025

University of Arkansas at Fayetteville ORCID

Selected Publications

The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid (2025)

Microorganisms 1 citation DOI OpenAlex
Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens (2022)

Microorganisms 11 citations DOI OpenAlex
Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization (2021)

Pathogens 3 citations DOI OpenAlex
Specific Secondary Bile Acids Control Chicken Necrotic Enteritis (2021)

Pathogens 32 citations DOI OpenAlex
Sodium butyrate modulates chicken macrophage proteins essential for Salmonella Enteritidis invasion (2021)

PLoS ONE 15 citations DOI OpenAlex
Microbiota attenuates chicken transmission-exacerbated campylobacteriosis in Il10−/− mice (2020)

Scientific Reports 7 citations DOI OpenAlex
Sodium Butyrate Reduces Salmonella Enteritidis Infection of Chicken Enterocytes and Expression of Inflammatory Host Genes in vitro (2020)

Frontiers in Microbiology 35 citations DOI OpenAlex
A secondary bile acid from microbiota metabolism attenuates ileitis and bile acid reduction in subclinical necrotic enteritis in chickens (2020)

Journal of Animal Science and Biotechnology/Journal of animal science and biotechnology 49 citations DOI OpenAlex
Microbial metabolite deoxycholic acid controls Clostridium perfringens-induced chicken necrotic enteritis through attenuating inflammatory cyclooxygenase signaling (2019)

Scientific Reports 46 citations DOI OpenAlex
Microbial metabolite deoxycholic acid shapes microbiota against Campylobacter jejuni chicken colonization (2019)

PLoS ONE 36 citations DOI OpenAlex
Microbial metabolite deoxycholic acid shapes microbiota against Campylobacter jejuni chicken colonization (2019)

bioRxiv (Cold Spring Harbor Laboratory) 9 citations DOI OpenAlex
Microbial metabolite deoxycholic acid controls Clostridium perfringens -induced chicken necrotic enteritis through attenuating cyclooxygenase signaling (2018)

bioRxiv (Cold Spring Harbor Laboratory) DOI OpenAlex