M
Mohit Bansal Data-verified
Affiliation confirmed via AI analysis of OpenAlex, ORCID, and web sources.
Research Scientist
staff
12 h-index
40 pubs
474 cited
Research Areas
Links
Biography and Research Information
OverviewAI-generated summary
Mohit Bansal's research investigates molecular mechanisms underlying cellular stress, proliferation, and adaptation in both cancer development and animal health. His work has explored how MYC drives mRNA pseudouridylation, a process critical for mitigating proliferation-induced cellular stress in cancer. Bansal has also studied the role of DKC1-mediated pseudouridylation of rRNA in sustaining translation and metabolic adaptation. In the realm of animal health, his research has focused on poultry diseases, examining how specific bile acids and vaccines using *Clostridium perfringens* sporulation proteins can reduce necrotic enteritis. He has also investigated the modulation of chicken macrophage proteins by sodium butyrate for *Salmonella Enteritidis* invasion and the impact of microbiota on *Campylobacter jejuni* chicken colonization. His scholarship metrics include an h-index of 12, with 40 total publications and 464 total citations.
Metrics
- h-index: 12
- Publications: 40
- Citations: 474
Selected Publications
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The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid (2025)
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Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens (2022)
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Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization (2021)
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Specific Secondary Bile Acids Control Chicken Necrotic Enteritis (2021)
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Sodium butyrate modulates chicken macrophage proteins essential for Salmonella Enteritidis invasion (2021)
Collaboration Network
Top Collaborators
Jane Ding
University of Alabama at Birmingham (US)
6 shared publications
- MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
- DKC1-mediated pseudouridylation of rRNA targets hnRNP A1 to sustain IRES-dependent translation and ATF4-driven metabolic adaptation
- DKC1-mediated pseudouridylation of rRNA targets hnRNP A1 to sustain IRES-dependent translation and ATF4-driven metabolic adaptation
- Data from MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
- Supplementary Tables from MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
Showing 5 of 6 shared publications
Anamika Gupta
University of Alabama at Birmingham (US)
6 shared publications
- MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
- DKC1-mediated pseudouridylation of rRNA targets hnRNP A1 to sustain IRES-dependent translation and ATF4-driven metabolic adaptation
- DKC1-mediated pseudouridylation of rRNA targets hnRNP A1 to sustain IRES-dependent translation and ATF4-driven metabolic adaptation
- Data from MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
- Supplementary Tables from MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
Showing 5 of 6 shared publications
Sunil Sudarshan
University of Alabama at Birmingham (US)
6 shared publications
- MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
- DKC1-mediated pseudouridylation of rRNA targets hnRNP A1 to sustain IRES-dependent translation and ATF4-driven metabolic adaptation
- DKC1-mediated pseudouridylation of rRNA targets hnRNP A1 to sustain IRES-dependent translation and ATF4-driven metabolic adaptation
- Data from MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
- Supplementary Tables from MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
Showing 5 of 6 shared publications
Han‐Fei Ding
University of Alabama at Birmingham (US)
6 shared publications
- MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
- DKC1-mediated pseudouridylation of rRNA targets hnRNP A1 to sustain IRES-dependent translation and ATF4-driven metabolic adaptation
- DKC1-mediated pseudouridylation of rRNA targets hnRNP A1 to sustain IRES-dependent translation and ATF4-driven metabolic adaptation
- Data from MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
- Supplementary Tables from MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
Showing 5 of 6 shared publications
Xiaolun Sun
University of Arkansas at Fayetteville
5 shared publications
In database
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Sodium butyrate modulates chicken macrophage proteins essential for Salmonella Enteritidis invasion
- Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens
- Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
Tahrir Alenezi
University of Arkansas at Fayetteville
4 shared publications
In database
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens
- Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
Ying Fu
University of Arkansas at Fayetteville
4 shared publications
In database
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens
- Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
Ayidh Almansour
University of Arkansas at Fayetteville (US)
4 shared publications
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens
- Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
Yuxia Cao
4 shared publications
- MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
- Data from MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
- Supplementary Tables from MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
- Supplementary Data from MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
Bingwei Ye
4 shared publications
- MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
- Data from MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
- Supplementary Tables from MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
- Supplementary Data from MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
Rui Mao
4 shared publications
- MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
- Data from MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
- Supplementary Tables from MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
- Supplementary Data from MYC Drives mRNA Pseudouridylation to Mitigate Proliferation-Induced Cellular Stress during Cancer Development
Anamika Gupta
University of Arkansas at Fayetteville
3 shared publications
In database
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Sodium butyrate modulates chicken macrophage proteins essential for Salmonella Enteritidis invasion
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
Rohana Liyanage
University of Arkansas at Fayetteville
3 shared publications
In database
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Sodium butyrate modulates chicken macrophage proteins essential for Salmonella Enteritidis invasion
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
Abhinav Upadhyay
University of Connecticut
1 shared publication
In database
- Sodium butyrate modulates chicken macrophage proteins essential for Salmonella Enteritidis invasion
N.C. Rath
United States Department of Agriculture (US)
1 shared publication
- Sodium butyrate modulates chicken macrophage proteins essential for Salmonella Enteritidis invasion
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Host-Pathogen Interactions