Tahrir Alenezi
This is a likely match — the affiliation was inferred from OpenAlex, ORCID, and web sources but has not been fully confirmed. Treat with appropriate caution.
Researcher
Also affiliated: Northern Border University (2023–2025); Long Island University (2020)
Faculty Researcher
Research Areas
Biomedical Subjects
Links
Biography and Research Information
OverviewAI-generated summary
Tahrir Alenezi's research primarily investigates the mechanisms of bacterial infections, focusing on *Clostridium perfringens* and its impact on animal health. His work has explored the role of specific secondary bile acids in controlling necrotic enteritis in chickens, a significant disease in poultry farming. Alenezi has also investigated the use of vaccines based on *Clostridium perfringens* sporulation proteins to reduce the incidence of this disease.
Further research by Alenezi delves into the modulation of bacterial virulence through microbial metabolites, specifically examining how bile acid metabolites and enzymes like taurodeoxycholic acid can inhibit *Clostridium perfringens*. His publications also touch upon the influence of the gut microbiota on bacterial colonization in animals and the broader role of immune responses in infectious diseases such as campylobacteriosis. More recently, Alenezi has contributed to systematic reviews and meta-analyses concerning the impact of surgical interventions on chronic health conditions in children, such as asthma control following adenotonsillectomy.
Metrics
- h-index: 6
- Publications: 10
- Citations: 128
Selected Publications
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The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid (2025)
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Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence (2024)
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Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence (2023)
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Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens (2022)
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Clostridium perfringens-Induced Necrotic Diseases: An Overview (2022)
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Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization (2021)
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Specific Secondary Bile Acids Control Chicken Necrotic Enteritis (2021)
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Microbiota attenuates chicken transmission-exacerbated campylobacteriosis in Il10−/− mice (2020)
Collaboration Network
Top Collaborators
- Clostridium perfringens-Induced Necrotic Diseases: An Overview
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens
- Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence
- Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence
Showing 5 of 7 shared publications
- Clostridium perfringens-Induced Necrotic Diseases: An Overview
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens
- Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence
- Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence
Showing 5 of 7 shared publications
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens
- Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence
- Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens
- Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
- Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence
- Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence
- Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
- Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
- Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization
- Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens
- Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence
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