Tahrir Alenezi Data-verified
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Biography and Research Information
OverviewAI-generated summary
Tahrir Alenezi's research focuses on microbial infections and disease mechanisms, particularly those involving Clostridium perfringens in poultry. His work has investigated the role of specific secondary bile acids and microbial metabolites in controlling and modulating the virulence of C. perfringens. Alenezi has explored the use of vaccines based on C. perfringens sporulation proteins to reduce necrotic enteritis in chickens. He also studies the impact of microbiota on bacterial colonization in chickens, examining the relationship between immune response and microbiota in Campylobacteriosis. Additionally, Alenezi has contributed to systematic reviews and meta-analyses on clinical outcomes, including the impact of adenotonsillectomy on asthma control in children. His collaborations include work with Xiaolun Sun, Ying Fu, Mohit Bansal, and Rohana Liyanage at the University of Arkansas at Fayetteville.
Metrics
- h-index: 6
- Publications: 11
- Citations: 125
Selected Publications
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The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid (2025)
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Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence (2024)
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Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence (2023)
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Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens (2022)
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Clostridium perfringens-Induced Necrotic Diseases: An Overview (2022)
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Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization (2021)
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Specific Secondary Bile Acids Control Chicken Necrotic Enteritis (2021)
Collaboration Network
Top Collaborators
- Clostridium perfringens-Induced Necrotic Diseases: An Overview
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens
- Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence
- Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence
Showing 5 of 8 shared publications
- Clostridium perfringens-Induced Necrotic Diseases: An Overview
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens
- Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence
- Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence
Showing 5 of 8 shared publications
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens
- Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence
- Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization
- The Role of Immune Response and Microbiota on Campylobacteriosis
Showing 5 of 6 shared publications
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Vaccines Using Clostridium perfringens Sporulation Proteins Reduce Necrotic Enteritis in Chickens
- Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
- Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence
- Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence
- Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
- Potent Bile Acid Microbial Metabolites Modulate Clostridium perfringens Virulence
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
- Recombinant Bile Salt Hydrolase Enhances the Inhibition Efficiency of Taurodeoxycholic Acid against Clostridium perfringens Virulence
- The Mechanistic Target of Rapamycin Mediates Clostridium perfringens-Induced Chicken Necrotic Enteritis Attenuated by Secondary Bile Acid Deoxycholic Acid
- The Role of Immune Response and Microbiota on Campylobacteriosis
- The Role of Immune Response and Microbiota on Campylobacteriosis
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Specific Secondary Bile Acids Control Chicken Necrotic Enteritis
- Microbiota from Specific Pathogen-Free Mice Reduces Campylobacter jejuni Chicken Colonization
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