N
Nicole Massoll
High Impact
Professor
faculty
Pathology, College of Medicine
27 h-index
80 pubs
2,386 cited
Research Areas
Biography and Research Information
OverviewAI-generated summary
Nicole Massoll's research focuses on the pathology of various cancers, particularly thyroid and breast neoplasms. Her work investigates the molecular mechanisms driving cancer progression and explores potential diagnostic and therapeutic strategies. Recent publications demonstrate her engagement with developing novel cancer models, including murine models for papillary thyroid cancer and cell lines that progress to poorly differentiated and anaplastic thyroid cancer. She has also explored the diagnostic performance of microRNA expression analysis in indeterminate thyroid nodules and examined the role of fibroblast-mediated collagen remodeling and macrophage abundance in thyroid cancer progression.
Massoll's research group has contributed to understanding the tumor microenvironment, specifically the interplay between fibroblasts, collagen, and cancer development in BRAFV600E-driven and Pten loss-associated thyroid cancers. Her work also extends to the expression of PD-L1 in oncocytic thyroid cancer in relation to macrophage abundance. Additionally, she has been involved in developing entrustable professional activities for cytopathology fellowship education, indicating a commitment to advancing pathology training.
Metrics
- h-index: 27
- Publications: 80
- Citations: 2,386
Selected Publications
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Macrophage abundance in oncocytic thyroid cancer is associated with PD-L1 expression (2025)
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Entrustable professional activities for cytopathology fellowship (2024)
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Development of Novel Murine BRAFV600E-Driven Papillary Thyroid Cancer Cell Lines for Modeling of Disease Progression and Preclinical Evaluation of Therapeutics (2023)
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A Retrospective Evaluation of the Diagnostic Performance of an Interdependent Pairwise MicroRNA Expression Analysis with a Mutation Panel in Indeterminate Thyroid Nodules (2022)
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Development of Novel Follicular Thyroid Cancer Models Which Progress to Poorly Differentiated and Anaplastic Thyroid Cancer (2021)
Grants & Funding
- Tumor-stromal crosstalk in papillary thyroid cancer- Resubmission American Cancer Society, Inc. Co-Investigator
- MRNA translational Control of Cell Cycle Progression American Cancer Society, Inc. Principal Investigator
Collaboration Network
Top Collaborators
Aime T. Franco
University of Pennsylvania (US)
17 shared publications
- Development of Novel Follicular Thyroid Cancer Models Which Progress to Poorly Differentiated and Anaplastic Thyroid Cancer
- Development of Novel Murine BRAFV600E-Driven Papillary Thyroid Cancer Cell Lines for Modeling of Disease Progression and Preclinical Evaluation of Therapeutics
- Data from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Figure S1 from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Figure S3 from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
Showing 5 of 17 shared publications
Lee Ann Jolly
15 shared publications
- Development of Novel Follicular Thyroid Cancer Models Which Progress to Poorly Differentiated and Anaplastic Thyroid Cancer
- Data from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Figure S1 from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Figure S3 from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Materials and Methods from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
Showing 5 of 15 shared publications
Sergey V. Novitskiy
14 shared publications
- Data from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Figure S1 from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Figure S3 from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Materials and Methods from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Figure S2 from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
Showing 5 of 14 shared publications
Phillip Owens
14 shared publications
- Data from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Figure S1 from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Figure S3 from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Materials and Methods from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Figure S2 from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
Showing 5 of 14 shared publications
Nikki Cheng
14 shared publications
- Data from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Figure S1 from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Figure S3 from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Materials and Methods from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Figure S2 from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
Showing 5 of 14 shared publications
Wei Fang
14 shared publications
- Data from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Figure S1 from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Figure S3 from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Materials and Methods from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Figure S2 from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
Showing 5 of 14 shared publications
Harold L. Moses
14 shared publications
- Data from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Figure S1 from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Figure S3 from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Materials and Methods from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
- Supplemental Figure S2 from Fibroblast-Mediated Collagen Remodeling Within the Tumor Microenvironment Facilitates Progression of Thyroid Cancers Driven by Braf<sup>V600E</sup> and Pten Loss
Showing 5 of 14 shared publications
Lingbao Ai
8 shared publications
- Data from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Data from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Supplementary Figure 1 from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Supplementary Figure 1 from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Supplementary Figure 3 from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
Showing 5 of 8 shared publications
Wan-Ju Kim
8 shared publications
- Data from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Data from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Supplementary Figure 1 from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Supplementary Figure 1 from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Supplementary Figure 3 from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
Showing 5 of 8 shared publications
Tae‐You Kim
8 shared publications
- Data from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Data from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Supplementary Figure 1 from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Supplementary Figure 1 from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Supplementary Figure 3 from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
Showing 5 of 8 shared publications
C. Robert Fields
8 shared publications
- Data from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Data from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Supplementary Figure 1 from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Supplementary Figure 1 from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Supplementary Figure 3 from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
Showing 5 of 8 shared publications
Keith D. Robertson
8 shared publications
- Data from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Data from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Supplementary Figure 1 from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Supplementary Figure 1 from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Supplementary Figure 3 from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
Showing 5 of 8 shared publications
Kevin D. Brown
8 shared publications
- Data from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Data from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Supplementary Figure 1 from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Supplementary Figure 1 from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
- Supplementary Figure 3 from Epigenetic Silencing of the Tumor Suppressor Cystatin M Occurs during Breast Cancer Progression
Showing 5 of 8 shared publications
Caitlin Caperton
University of Arkansas for Medical Sciences
2 shared publications
In database
- Development of Novel Follicular Thyroid Cancer Models Which Progress to Poorly Differentiated and Anaplastic Thyroid Cancer
- Macrophage abundance in oncocytic thyroid cancer is associated with PD-L1 expression
Andrew J. Bauer
Children's Hospital of Philadelphia (US)
2 shared publications
- Development of Novel Follicular Thyroid Cancer Models Which Progress to Poorly Differentiated and Anaplastic Thyroid Cancer
- Development of Novel Murine BRAFV600E-Driven Papillary Thyroid Cancer Cell Lines for Modeling of Disease Progression and Preclinical Evaluation of Therapeutics
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