Biography and Research Information
OverviewAI-generated summary
Pritam Biswas's research focuses on the molecular mechanisms underlying cellular apoptosis, particularly in the context of cancer, and the potential of nanomaterials in therapeutic applications. His work investigates the role of specific lipid mediators and cytokines, such as IL-13 and 13(S)HpODE, in initiating apoptotic pathways through signaling cascades involving MAO-A, ROS, and p53/p21. Biswas also explores the application of redox nanomedicine for conditions like chronic kidney disease, aiming to recondition mitochondria. His research has involved preclinical animal studies to assess the safety and efficacy of nanomaterials, including their redox buffering capacity in relation to ROS-based therapeutics and toxicity. He has co-authored publications with researchers at the University of Arkansas for Medical Sciences, including Samrat Roy Choudhury and Akhilesh Kaushal. Biswas has an h-index of 15 and has authored 85 publications with 745 citations.
Metrics
- h-index: 15
- Publications: 85
- Citations: 745
Selected Publications
-
Transcriptional rewiring by enhancer methylation in CBFA2T3-GLIS2–driven pediatric acute megakaryoblastic leukemia (2025)
-
Abstract 2281 Epigenetic Drivers and Subtype-Specific Roles of DNMT3B in Acute Myeloid Leukemia (2025)
-
Abstract P16: CBFA2T3-GLIS2 fusion leads to a distinct DNA methylation enhancer landscape in pediatric acute myeloid leukemia (2024)
Collaboration Network
Top Collaborators
- Abstract P16: CBFA2T3-GLIS2 fusion leads to a distinct DNA methylation enhancer landscape in pediatric acute myeloid leukemia
- Abstract 2281 Epigenetic Drivers and Subtype-Specific Roles of DNMT3B in Acute Myeloid Leukemia
- Transcriptional rewiring by enhancer methylation in CBFA2T3-GLIS2–driven pediatric acute megakaryoblastic leukemia
- Abstract P16: CBFA2T3-GLIS2 fusion leads to a distinct DNA methylation enhancer landscape in pediatric acute myeloid leukemia
- Abstract 2281 Epigenetic Drivers and Subtype-Specific Roles of DNMT3B in Acute Myeloid Leukemia
- Transcriptional rewiring by enhancer methylation in CBFA2T3-GLIS2–driven pediatric acute megakaryoblastic leukemia
- Abstract P16: CBFA2T3-GLIS2 fusion leads to a distinct DNA methylation enhancer landscape in pediatric acute myeloid leukemia
- Transcriptional rewiring by enhancer methylation in CBFA2T3-GLIS2–driven pediatric acute megakaryoblastic leukemia
- Abstract 2281 Epigenetic Drivers and Subtype-Specific Roles of DNMT3B in Acute Myeloid Leukemia
- Transcriptional rewiring by enhancer methylation in CBFA2T3-GLIS2–driven pediatric acute megakaryoblastic leukemia
- Transcriptional rewiring by enhancer methylation in CBFA2T3-GLIS2–driven pediatric acute megakaryoblastic leukemia
- Transcriptional rewiring by enhancer methylation in CBFA2T3-GLIS2–driven pediatric acute megakaryoblastic leukemia
- Transcriptional rewiring by enhancer methylation in CBFA2T3-GLIS2–driven pediatric acute megakaryoblastic leukemia
- Transcriptional rewiring by enhancer methylation in CBFA2T3-GLIS2–driven pediatric acute megakaryoblastic leukemia
Similar Researchers
Based on overlapping research topics