Match tier
Listed
Presence
Current · Arkansas
Last published
2026
Sources
OpenAlex · ORCID
Refreshed
2026-08-08
A
Arundhati Chavan
Researcher
Also affiliated: Arkansas Children's Hospital (2025–2026)
Unknown Researcher
1 h-index
5 pubs
2 cited
Research Areas
Biography and Research Information
OverviewAI-generated summary
Arundhati Chavan's research focuses on the molecular mechanisms underlying acute megakaryoblastic leukemia (AMKL) and acute myeloid leukemia (AML). Her work investigates how genetic fusions, such as CBFA2T3-GLIS2, drive transcriptional rewiring through epigenetic modifications, specifically enhancer methylation. Chavan's publications explore the role of enzymes like DNMT3B in controlling chromatin states and cell cycle networks in AML, identifying potential vulnerabilities for therapeutic intervention. She has co-authored publications with researchers at the University of Arkansas for Medical Sciences, including Samrat Roy Choudhury and Giselle Almeida Gonzalez. Chavan's research is characterized by an h-index of 1 and 5 total publications.
Metrics
- h-index: 1
- Publications: 5
- Citations: 2
Selected Publications
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Abstract 3496: Epigenetic repurposing by CBFA2T3-GLIS2 reveals a druggable DNA methylation axis in high-risk pediatric AML vulnerability in pediatric AML (2026)
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Abstract PR006: CBFA2T3–GLIS2 fusion reprograms enhancer-linked DNA methylation to enforce apoptotic resistance and defines an epigenetic vulnerability in pediatric AML (2026)
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Transcriptional rewiring by enhancer methylation in CBFA2T3-GLIS2–driven pediatric acute megakaryoblastic leukemia (2025)
Collaboration Network
Top Collaborators
Samrat Roy Choudhury
University of Arkansas for Medical Sciences
3 shared publications
In database
- Transcriptional rewiring by enhancer methylation in CBFA2T3-GLIS2–driven pediatric acute megakaryoblastic leukemia
- Abstract PR006: CBFA2T3–GLIS2 fusion reprograms enhancer-linked DNA methylation to enforce apoptotic resistance and defines an epigenetic vulnerability in pediatric AML
- Abstract 3496: Epigenetic repurposing by CBFA2T3-GLIS2 reveals a druggable DNA methylation axis in high-risk pediatric AML vulnerability in pediatric AML
Giselle Almeida Gonzalez
University of Arkansas for Medical Sciences
3 shared publications
In database
- Transcriptional rewiring by enhancer methylation in CBFA2T3-GLIS2–driven pediatric acute megakaryoblastic leukemia
- Abstract PR006: CBFA2T3–GLIS2 fusion reprograms enhancer-linked DNA methylation to enforce apoptotic resistance and defines an epigenetic vulnerability in pediatric AML
- Abstract 3496: Epigenetic repurposing by CBFA2T3-GLIS2 reveals a druggable DNA methylation axis in high-risk pediatric AML vulnerability in pediatric AML
Jason E. Farrar
University of Arkansas for Medical Sciences
3 shared publications
In database
- Transcriptional rewiring by enhancer methylation in CBFA2T3-GLIS2–driven pediatric acute megakaryoblastic leukemia
- Abstract PR006: CBFA2T3–GLIS2 fusion reprograms enhancer-linked DNA methylation to enforce apoptotic resistance and defines an epigenetic vulnerability in pediatric AML
- Abstract 3496: Epigenetic repurposing by CBFA2T3-GLIS2 reveals a druggable DNA methylation axis in high-risk pediatric AML vulnerability in pediatric AML
Akhilesh Kaushal
Arkansas Children's Hospital
1 shared publication
In database
- Transcriptional rewiring by enhancer methylation in CBFA2T3-GLIS2–driven pediatric acute megakaryoblastic leukemia
Pritam Biswas
Arkansas Children's Hospital
1 shared publication
In database
- Transcriptional rewiring by enhancer methylation in CBFA2T3-GLIS2–driven pediatric acute megakaryoblastic leukemia
Cory C. Padilla
Biota Pharmaceuticals (United States) (US)
1 shared publication
- Transcriptional rewiring by enhancer methylation in CBFA2T3-GLIS2–driven pediatric acute megakaryoblastic leukemia
Jay F. Sarthy
1 shared publication
- Transcriptional rewiring by enhancer methylation in CBFA2T3-GLIS2–driven pediatric acute megakaryoblastic leukemia
Soheil Meshinchi
Fred Hutch Cancer Center (US)
1 shared publication
- Transcriptional rewiring by enhancer methylation in CBFA2T3-GLIS2–driven pediatric acute megakaryoblastic leukemia
Rhonda E. Ries
Cape Town HVTN Immunology Laboratory / Hutchinson Centre Research Institute of South Africa (ZA)
1 shared publication
- Abstract PR006: CBFA2T3–GLIS2 fusion reprograms enhancer-linked DNA methylation to enforce apoptotic resistance and defines an epigenetic vulnerability in pediatric AML
Soheil Meshinchi
Cape Town HVTN Immunology Laboratory / Hutchinson Centre Research Institute of South Africa (ZA)
1 shared publication
- Abstract PR006: CBFA2T3–GLIS2 fusion reprograms enhancer-linked DNA methylation to enforce apoptotic resistance and defines an epigenetic vulnerability in pediatric AML
Rhonda E. Ries
Fred Hutch Cancer Center (US)
1 shared publication
- Abstract 3496: Epigenetic repurposing by CBFA2T3-GLIS2 reveals a druggable DNA methylation axis in high-risk pediatric AML vulnerability in pediatric AML
Soheil Meshinchi
Fred Hutch Cancer Center (US)
1 shared publication
- Abstract 3496: Epigenetic repurposing by CBFA2T3-GLIS2 reveals a druggable DNA methylation axis in high-risk pediatric AML vulnerability in pediatric AML
Similar Researchers
Based on overlapping research topics
Giselle Almeida Gonzalez
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Akhilesh Kaushal
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Jason E. Farrar
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