Biography and Research Information
OverviewAI-generated summary
Rahul Sharma's research investigates the complex interplay of immune responses, inflammation, and disease mechanisms, with a particular focus on conditions such as autoimmune disorders, kidney injury, and cancer. His work explores the role of specific cellular components and molecular pathways in disease progression and potential therapeutic interventions.
Recent publications demonstrate Sharma's engagement with critical areas of immunological research. He has studied the impact of immune checkpoint blockade on the systemic immune landscape and tumor microenvironment in obesity-associated breast cancer. His research also delves into modulating T-follicular helper cells and germinal center B cells to ameliorate autoimmunity, as well as the function of ST2+ T-regulatory cells in renal inflammation and fibrosis following ischemic kidney injury. Furthermore, his work has explored the protective effects of hybrid cytokines in acute graft-versus-host disease and the stabilization of transcription factors by pannexin-3 in vascular oxidative stress.
Sharma's research network includes collaborators at the University of Arkansas for Medical Sciences, such as N. K. Shah, Milenko T Petrovic, and Esther Park. With a scholarly profile including an h-index of 23 and over 1,700 citations across 70 publications, he is recognized as a highly cited researcher. His recent activity and publication dates indicate ongoing contributions to the field.
Metrics
- h-index: 23
- Publications: 70
- Citations: 1,724
Selected Publications
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Sudden Death Due to Hypercoagulability in a Patient With Pancreatic Cancer and Diabetic Ketoacidosis (2025)
Collaboration Network
Top Collaborators
Vikram Sabapathy
University of Virginia (US)
13 shared publications
- Targeting Bcl6 in the TREX1 D18N murine model ameliorates autoimmunity by modulating T‐follicular helper cells and germinal center B cells
- Pannexin-3 stabilizes the transcription factor Bcl6 in a channel-independent manner to protect against vascular oxidative stress
- ST2+ T-Regulatory Cells in Renal Inflammation and Fibrosis after Ischemic Kidney Injury
- Hybrid cytokine IL233 renders protection in murine acute graft vs host disease (aGVHD)
- Spatial Transcriptomics Enables Generation of a Complement Atlas of Mouse AKI
Showing 5 of 13 shared publications
Rajkumar Venkatadri
University of Virginia (US)
6 shared publications
- Targeting Bcl6 in the TREX1 D18N murine model ameliorates autoimmunity by modulating T‐follicular helper cells and germinal center B cells
- ST2+ T-Regulatory Cells in Renal Inflammation and Fibrosis after Ischemic Kidney Injury
- Hybrid cytokine IL233 renders protection in murine acute graft vs host disease (aGVHD)
- IL-33/ST2 Alarmin Signaling Axis in Myeloid Cells Regulates Kidney Injury
- Defective Clearance of Nucleic Acids Exacerbates AKI
Showing 5 of 6 shared publications
Murat Doğan
University of Tennessee Health Science Center (US)
6 shared publications
- Targeting Bcl6 in the TREX1 D18N murine model ameliorates autoimmunity by modulating T‐follicular helper cells and germinal center B cells
- ST2+ T-Regulatory Cells in Renal Inflammation and Fibrosis after Ischemic Kidney Injury
- Hybrid cytokine IL233 renders protection in murine acute graft vs host disease (aGVHD)
- IL-33/ST2 Alarmin Signaling Axis in Myeloid Cells Regulates Kidney Injury
- Defective Clearance of Nucleic Acids Exacerbates AKI
Showing 5 of 6 shared publications
Saleh Mohammad
University of Virginia (US)
5 shared publications
- Targeting Bcl6 in the TREX1 D18N murine model ameliorates autoimmunity by modulating T‐follicular helper cells and germinal center B cells
- ST2+ T-Regulatory Cells in Renal Inflammation and Fibrosis after Ischemic Kidney Injury
- Hybrid cytokine IL233 renders protection in murine acute graft vs host disease (aGVHD)
- Defective Clearance of Nucleic Acids Exacerbates AKI
- Novel Role of ST2+ Tubular Cells in Shaping Immune Microenvironment
Gabrielle Costlow
University of Virginia (US)
4 shared publications
- ST2+ T-Regulatory Cells in Renal Inflammation and Fibrosis after Ischemic Kidney Injury
- IL-33/ST2 Alarmin Signaling Axis in Myeloid Cells Regulates Kidney Injury
- IL-33 (Interleukin 33)/ST2 (Interleukin 1 Receptor-Like 1) “Alarmin” Signaling Axis Regulates Innate Immune Response in Kidney Injury
- Novel Role of ST2+ Tubular Cells in Shaping Immune Microenvironment
Didier Portilla
University of Virginia (US)
3 shared publications
- Spatial Transcriptomics Enables Generation of a Complement Atlas of Mouse AKI
- Spatial transcriptomics maps complement-driven cell-to-cell interactions during acute kidney injury
- Regulation of Mitochondrial Metabolism in T-Regulatory Cells by Programmed Cell Death Protein 1 in AKI
Ajeeth K. Pingili
University of Tennessee Health Science Center (US)
2 shared publications
- Immune checkpoint blockade reprograms systemic immune landscape and tumor microenvironment in obesity-associated breast cancer
- Abstract PS17-04: Immune checkpoint blockade reprograms tumor microenvironment and systemic immune landscape in obesity associated triple negative breast cancer
Mehdi Chaib
University of Tennessee Health Science Center (US)
2 shared publications
- Immune checkpoint blockade reprograms systemic immune landscape and tumor microenvironment in obesity-associated breast cancer
- Abstract PS17-04: Immune checkpoint blockade reprograms tumor microenvironment and systemic immune landscape in obesity associated triple negative breast cancer
Laura M. Sipe
University of Tennessee Health Science Center (US)
2 shared publications
- Immune checkpoint blockade reprograms systemic immune landscape and tumor microenvironment in obesity-associated breast cancer
- Abstract PS17-04: Immune checkpoint blockade reprograms tumor microenvironment and systemic immune landscape in obesity associated triple negative breast cancer
Emily J. Miller
University of Tennessee Health Science Center (US)
2 shared publications
- Immune checkpoint blockade reprograms systemic immune landscape and tumor microenvironment in obesity-associated breast cancer
- Abstract PS17-04: Immune checkpoint blockade reprograms tumor microenvironment and systemic immune landscape in obesity associated triple negative breast cancer
Bin Teng
University of Tennessee Health Science Center (US)
2 shared publications
- Immune checkpoint blockade reprograms systemic immune landscape and tumor microenvironment in obesity-associated breast cancer
- Abstract PS17-04: Immune checkpoint blockade reprograms tumor microenvironment and systemic immune landscape in obesity associated triple negative breast cancer
Radhika Sekhri
University of Tennessee Health Science Center (US)
2 shared publications
- Immune checkpoint blockade reprograms systemic immune landscape and tumor microenvironment in obesity-associated breast cancer
- Abstract PS17-04: Immune checkpoint blockade reprograms tumor microenvironment and systemic immune landscape in obesity associated triple negative breast cancer
Heejoon Jo
University of Tennessee Health Science Center (US)
2 shared publications
- Immune checkpoint blockade reprograms systemic immune landscape and tumor microenvironment in obesity-associated breast cancer
- Abstract PS17-04: Immune checkpoint blockade reprograms tumor microenvironment and systemic immune landscape in obesity associated triple negative breast cancer
D. Neil Hayes
University of Tennessee Health Science Center (US)
2 shared publications
- Immune checkpoint blockade reprograms systemic immune landscape and tumor microenvironment in obesity-associated breast cancer
- Abstract PS17-04: Immune checkpoint blockade reprograms tumor microenvironment and systemic immune landscape in obesity associated triple negative breast cancer
Joseph F. Pierre
University of Tennessee Health Science Center (US)
2 shared publications
- Immune checkpoint blockade reprograms systemic immune landscape and tumor microenvironment in obesity-associated breast cancer
- Abstract PS17-04: Immune checkpoint blockade reprograms tumor microenvironment and systemic immune landscape in obesity associated triple negative breast cancer
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