Rami Ahmad Shahror
Researcher
Also affiliated: University of Maryland, Baltimore (2020–2023); National Health Research Institutes (2019–2020); Örebro University (2015–2018); University of Arkansas Medical Center (2025); Taipei Medical University (2019–2020)
Faculty Researcher
Pharmacology & Toxicology, College of Medicine
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Rami Ahmad Shahror's research focuses on the role of myeloid cells and innate immune responses in central nervous system and ocular diseases. His work investigates mechanisms underlying neuroinflammation and its contribution to conditions such as traumatic brain injury and ischemic retinopathies. Shahror's publications explore the modulation of inflammatory pathways, including the arginase 1/ornithine decarboxylase pathway and the role of HDAC3 in myeloid cell function, to ameliorate injury in retinal ischemic models. He also examines the potential of nanosystems for ocular drug delivery and employs techniques like multi-color flow cytometry to characterize myeloid cell populations in mouse retina research. Shahror has 25 publications with 424 citations and an h-index of 10. He collaborates with researchers at the University of Arkansas for Medical Sciences, including Abdelrahman Y. Fouda, Carol Morris, Esraa Shosha, and Melissa Wild.
Metrics
- h-index: 10
- Publications: 25
- Citations: 456
Selected Publications
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CD5L promotes efferocytosis and resolution of retinal ischemic injury (2026)
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Myeloid HDAC3 deletion protects against traumatic optic injury (2026)
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Recent advances and future challenges in nanosystems for ocular drug delivery (2025)
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Myeloid HDAC3 Deletion Protects Against Traumatic Optic Injury (2025)
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Disrupting the CD47/SIRPα Axis Protects Against Traumatic Optic Neuropathy (2025)
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Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology (2024)
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Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury (2024)
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Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions (2024)
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The arginase 1/ornithine decarboxylase pathway suppresses HDAC3 to ameliorate the myeloid cell inflammatory response: implications for retinal ischemic injury (2023)
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Multi-color Flow Cytometry Protocol to Characterize Myeloid Cells in Mouse Retina Research (2023)
Collaboration Network
Top Collaborators
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- The arginase 1/ornithine decarboxylase pathway suppresses HDAC3 to ameliorate the myeloid cell inflammatory response: implications for retinal ischemic injury
- Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury
- Multi-color Flow Cytometry Protocol to Characterize Myeloid Cells in Mouse Retina Research
- Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology
Showing 5 of 9 shared publications
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- The arginase 1/ornithine decarboxylase pathway suppresses HDAC3 to ameliorate the myeloid cell inflammatory response: implications for retinal ischemic injury
- Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury
- Multi-color Flow Cytometry Protocol to Characterize Myeloid Cells in Mouse Retina Research
- Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology
Showing 5 of 6 shared publications
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- The arginase 1/ornithine decarboxylase pathway suppresses HDAC3 to ameliorate the myeloid cell inflammatory response: implications for retinal ischemic injury
- Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury
- Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury
- Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology
- Disrupting the CD47/SIRPα Axis Protects Against Traumatic Optic Neuropathy
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- The arginase 1/ornithine decarboxylase pathway suppresses HDAC3 to ameliorate the myeloid cell inflammatory response: implications for retinal ischemic injury
- Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
- The arginase 1/ornithine decarboxylase pathway suppresses HDAC3 to ameliorate the myeloid cell inflammatory response: implications for retinal ischemic injury
- Multi-color Flow Cytometry Protocol to Characterize Myeloid Cells in Mouse Retina Research
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology
- Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
- Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
- Multi-color Flow Cytometry Protocol to Characterize Myeloid Cells in Mouse Retina Research
- The arginase 1/ornithine decarboxylase pathway suppresses HDAC3 to ameliorate the myeloid cell inflammatory response: implications for retinal ischemic injury
- The arginase 1/ornithine decarboxylase pathway suppresses HDAC3 to ameliorate the myeloid cell inflammatory response: implications for retinal ischemic injury
- The arginase 1/ornithine decarboxylase pathway suppresses HDAC3 to ameliorate the myeloid cell inflammatory response: implications for retinal ischemic injury
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