Melissa Wild
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Also affiliated: University of Arkansas Medical Center (2024)
Research Areas
Biomedical Subjects
Biography and Research Information
OverviewAI-generated summary
Melissa Wild's research focuses on the molecular mechanisms underlying retinal diseases, particularly diabetic retinopathy and ischemic retinopathies. Her work investigates the role of histone deacetylases (HDACs) and myeloid cells in the progression and resolution of these conditions. Wild has published on how the deletion of myeloid HDAC3 can promote efferocytosis, a cellular process that helps clear cellular debris, thereby ameliorating retinal ischemic injury. She also studies the involvement of CD5L in efferocytosis and the resolution of retinal ischemic injury, as well as how disrupting the CD47/SIRPα axis offers protection against traumatic optic neuropathy. Her research utilizes mouse models, including knockout mice, and involves proteomic analysis of aqueous humor to understand disease pathophysiology. Wild collaborates with researchers at the University of Arkansas for Medical Sciences, including Abdelrahman Y. Fouda and Carol Morris, with whom she shares multiple publications.
Metrics
- h-index: 3
- Publications: 6
- Citations: 69
Selected Publications
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CD5L promotes efferocytosis and resolution of retinal ischemic injury (2026)
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HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis (2025)
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Disrupting the CD47/SIRPα Axis Protects Against Traumatic Optic Neuropathy (2025)
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Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology (2024)
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Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury (2024)
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Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions (2024)
Collaboration Network
Top Collaborators
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury
- Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology
- HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
Showing 5 of 6 shared publications
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury
- Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
- Disrupting the CD47/SIRPα Axis Protects Against Traumatic Optic Neuropathy
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury
- Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology
- HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis
- Disrupting the CD47/SIRPα Axis Protects Against Traumatic Optic Neuropathy
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury
- Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology
- HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury
- HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology
- HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis
- Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions
- Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology
- Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
- Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
- HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis
- CD5L promotes efferocytosis and resolution of retinal ischemic injury
- Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury
- Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury
- Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology
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