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Presence Current · Arkansas
Last published 2026
Sources OpenAlex · ORCID
Refreshed 2026-10-09

Melissa Wild

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Also affiliated: University of Arkansas Medical Center (2024)

3 h-index 6 pubs 69 cited

  • Retina
  • Histone Deacetylases
  • Mice, Inbred C57BL
  • Diabetic Retinopathy
  • Humans
  • Retinal Diseases
  • Animals
  • Reperfusion Injury
  • Mice, Knockout
  • Mice
  • Macrophages
  • Efferocytosis
  • Histone Deacetylase 3
  • Phagocytosis
  • Aqueous Humor

Biography and Research Information

OverviewAI-generated summary

Melissa Wild's research focuses on the molecular mechanisms underlying retinal diseases, particularly diabetic retinopathy and ischemic retinopathies. Her work investigates the role of histone deacetylases (HDACs) and myeloid cells in the progression and resolution of these conditions. Wild has published on how the deletion of myeloid HDAC3 can promote efferocytosis, a cellular process that helps clear cellular debris, thereby ameliorating retinal ischemic injury. She also studies the involvement of CD5L in efferocytosis and the resolution of retinal ischemic injury, as well as how disrupting the CD47/SIRPα axis offers protection against traumatic optic neuropathy. Her research utilizes mouse models, including knockout mice, and involves proteomic analysis of aqueous humor to understand disease pathophysiology. Wild collaborates with researchers at the University of Arkansas for Medical Sciences, including Abdelrahman Y. Fouda and Carol Morris, with whom she shares multiple publications.

Metrics

  • h-index: 3
  • Publications: 6
  • Citations: 69

Selected Publications

  • CD5L promotes efferocytosis and resolution of retinal ischemic injury (2026)
    Cell Death and Disease 1 citation DOI OpenAlex
  • HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis (2025)
    Acta Pharmacologica Sinica 2 citations DOI OpenAlex
  • Disrupting the CD47/SIRPα Axis Protects Against Traumatic Optic Neuropathy (2025)
    Research Square DOI OpenAlex
  • Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology (2024)
    Translational Vision Science & Technology 3 citations DOI OpenAlex
  • Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury (2024)
    Journal of Neuroinflammation 18 citations DOI OpenAlex
  • Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions (2024)
    Journal of Neuroinflammation 45 citations DOI OpenAlex

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Collaboration Network

19 Collaborators 5 Institutions 2 Countries

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