Melissa Wild

Researcher

Last publication 2026 Last refreshed 2026-05-16

unknown

2 h-index 6 pubs 59 cited

Biography and Research Information

OverviewAI-generated summary

Melissa Wild's research focuses on the role of myeloid cells and specific molecular pathways, such as HDAC3 and the CD47/SIRPα axis, in retinal injury and disease. Her work investigates how these elements influence processes like efferocytosis, metabolic reprogramming, and angiogenesis in conditions such as ischemic retinopathies, central retinal artery occlusion, and traumatic optic neuropathy. Wild has published on these topics, with her recent work exploring proteomic analysis of aqueous humor to understand disease pathophysiology and the impact of myeloid HDAC3 deletion on retinal injury. She has collaborated with researchers at the University of Arkansas for Medical Sciences, including Abdelrahman Y. Fouda, Carol Morris, Rami Ahmad Shahror, and Esraa Shosha, on multiple shared publications. Wild's scholarship metrics include an h-index of 2, with 5 total publications and 58 total citations.

Metrics

  • h-index: 2
  • Publications: 6
  • Citations: 59

Selected Publications

  • HDAC3 mediates retinal endothelial cell metabolic reprogramming and angiogenesis (2025)
  • Disrupting the CD47/SIRPα Axis Protects Against Traumatic Optic Neuropathy (2025)
  • Proteomic Analysis of Aqueous Humor in Central Retinal Artery Occlusion: Unveiling Novel Insights Into Disease Pathophysiology (2024)
    1 citation DOI OpenAlex
  • Deletion of myeloid HDAC3 promotes efferocytosis to ameliorate retinal ischemic injury (2024)
    14 citations DOI OpenAlex
  • Role of myeloid cells in ischemic retinopathies: recent advances and unanswered questions (2024)
    43 citations DOI OpenAlex

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Collaboration Network

19 Collaborators 4 Institutions 1 Country

Top Collaborators

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